Cardiology Flashcards

1
Q

HOCM Clinical signs

A
  1. Pulse: Jerky
  2. Double carotid impulsations
  3. Prominent a waves
  4. Apex: Undisplaced, double impulse, pressure loaded
  5. Systolic thrill
  6. S2- Reverse split
  7. S4
  8. ESM at LLSE- Louder with Valsalva, Softer with isometric hand grip
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2
Q

HOCM Complications

A
Biventricular failure
Arrhythmia; AF, VT
SCD
Angina
Endocarditis
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3
Q

HOCM Poor prognosis

A
1. Clinical
History of syncope
Family history of SCD
2. Investigations
Holter evidence of Ventricular arrhythmias
BP drop with exercise
3. Echo
Asymmetrical septal wall thickening >30mm
Systolic anterior motion
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4
Q

HOCM Investigations

A
ECG: LVH, LAD, Q waves anterolaterally, ST changes
CXR: Cardiomegaly
ECHO: LVH, septal hypertrophy, LVOT
Exercise testing/Holter monitor
Angiogram- right and left
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5
Q

HOCM treatment

A

Non-pharmacological
Pharmacological if symptomatic: BB
Alcohol septal ablation

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6
Q

Eisenmenger’s syndrome

A

Pulmonary hypertension with shunt reversal

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7
Q

Eisenmenger Complications

A
Haemoptysis
RVF
Paradoxical emboli
IE
SCD
Polycythemia
Thrombosis
Bleeding
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8
Q

Tetralogy of fallot

A

VSD
RVOT
RVH
Overriding aorta

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9
Q

Fallot’s Tetralogy Complications

A
Cyanosis
IE
RHF
Polycythaemia
Thrombosis
Paradoxical emboli
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10
Q

Fallot’s Tetralogy Investigations

A

ECG: RVH, RAD
CXR: Boot shaped heart
ECHO: AR, Pul Htn

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11
Q

Fallot’s Tetralogy signs

A
Central cyanosis
Clubbing
Diminished Left radial pulse
BP lower in Left arm
Absent a waves
Thoracotomy scar
Normal apex beat/Volume loaded
Parasternal heave
Palpable A2
Murmur: ESM (RVOT), AR (prolapse of coronary cusp
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12
Q

PDA clinical signs

A
Clubbing of toes
Pulse: large volume, collapsing
Wide pulse pressure
Parasternal heave
Left subclavicular thrill
Apex displaced, thrusting
Continuous machine murmur
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13
Q

Cause Collapsing pulse

A
AR
PDA
Fever
Thyrotoxicosis
Anaemia
Pregnancy
AVF
Severe bradycardia
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14
Q

Continuous murmurs

A

PDA
Mixed mitral and aortic disease
VSD
AVF

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15
Q

PDA complications

A

IE
LVF
Pulmonary hypertension
Eisenmenger’s syndrome

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16
Q

Coarctation of aorta Clinical signs

A
Upper torso better developed
Radial-radial delay
Vigorous carotid pulsations
Radio-femoral delay
Apex pressure loaded
Ejection click
ESM at LSE and posteriorly
17
Q

Coarctation associated with

A

Bicuspid aortic valve
PDA
VSD
Transposition of great vessels

18
Q

ASD clinical signs

A

Fixed spilt of S2

ESM at upper LSE

19
Q

Wide split S2

A

ASD
VSD
MR
RBBB

20
Q

VSD clinical signs

A

Parasternal thrill

PSM at LLSE

21
Q

Pulmonary hypertension signs

A
JVP: prominent a and v waves
Parasternal heave
Palpable P2
Loud P2
PSM at LLSE, Inspiration
RVF
22
Q

PHTN Types

A
Primary
LV disease
Lung
CTE
Other
23
Q

PHTN Inx

A
ECG: RAD, RVH, biphid P waves, RBBB
CXR: Prominent pulmonary vasculature, ?Cause: cardiomegaly, hyperinflation, reticular nodular opacifications
ABG
ECHO
CT/V/Q
PFT
HRCT
RHC- PAP >25 mmHg, wedge >15mmHg
24
Q

JVP

A
A- atrial contraction
x: emptying
C: Tricuspid closure
v: filling RA
y: open Tricuspid
25
Q

AR clinical signs

A
Large volume, Collapsing pulse
Wide pulse pressure
Visible carotid pulsations
Apex: displaced volume loaded
Early diastolic murmur, expiration
PH/CCF
26
Q

Causes AR

A

Aortic: dissection, aortitis, marfans
Valvular: biscupid, RHD, CTD, IE

27
Q

Eponymous signs

A

Corrigans: visible carotid pulsations
Quinke’s: Nailbed pulsations
De Musset: Head nodding
Muller: Uvula pulsations

28
Q

AR surgical indications

A

Symptomatic: Serve AR
Asymptomatic: LVF (EF <50%), LVESD >55 mm, Aortic Root > 50 mm