Cardio/vasc Flashcards
Peripheral arterial disease management?
Strongly linked to smoking
Treat comorbitidies - HTN, DM, obesity
- Statin (atorvastatin 80mg)
- exercise training
Severe PAD or critical limb ischaemia may be treated by:
- angioplasty
- stenting
- bypass surgery
Amputation should be reserved for patients with critical limb ischaemia who are not suitable for other interventions such as angioplasty of bypass surgery.
CHA2DS2-VASc score?
Congestive heart failure - 1 Hypertension (or treated hypertension) - 1 Age >= 75 years - 2 Age 65-74 years - 1 Diabetes - 1 Prior Stroke or TIA - 2 Vascular disease (including ischaemic heart disease and peripheral arterial disease) - 1 Sex (female) - 1
Management of pericarditis?
First line - NSAID and colchicine
Pericarditis reminder
- pleuritic chest pain
- widespread ST elevation (saddle shaped)
- ECG changes are often widespread
- PR depression
All suspected pts with pericarditis - TTE
Becks triad? What does it suggest?
Raised JVP
Hypotension
Muffled heart sounds
Suggests cardiac tamponade - urgent pericardiocentesis
May also see - dyspnoea, tachycardia, pulsus paradoxus, absent Y descent, ECG showing electrical alternans. Often Kussmaul sign negative (Kussmaul sign - JVP that doesnt fall with inspiration)
What is WPW syndrome? What can you see on ECG?
congenital accessory conducting pathway between the atria and ventricles leading to a atrioventricular re-entry tachycardia (AVRT).
As the accessory pathway does not slow conduction AF can degenerate rapidly to VF
Possible ECG features:
- short PR interval
- wide QRS complexes with a slurred upstroke ‘delta wave’
- left axis deviation if right-sided accessory pathway
- right axis deviation if left-sided accessory pathway
Differentiating between type A and type B
- type A (left-sided pathway): dominant R wave in V1
- type B (right-sided pathway): no dominant R wave in V1
Angina pectoris management (stable)
All - aspirin and statin (in absence of contraindications)
Sublingual GTN to abort angina attacks
Beta blocker or CCB based on cormorbidities/contraindications
- if CCB monotherapy use a rate-limiting one such as verapamil or diltiazem (and no b-blocker alongside - risk of complete heart block)
If poor response put to max dose
If still symptomatic after monotherapy with a beta-blocker add a calcium channel blocker and vice versa
If patient on monotherapy and cannot tolerate the addition of a CCB/BB then consider one of the following drugs: a long-acting nitrate, ivabradine, nicorandil or ranolazine
Causes of long QT syndrome
Management?
Genetic
Electrolytes
- hypocalcaemia
- hypomagnesaemia
- hypokalaemia
Drugs
- antiarrhythmics
- antibiotics - macrolides, fluoroquinolones
- psychotropic drugs
Other
- myocarditis
- hypothermia
- subarachnoid haemorrhage
MANAGEMENT - IV Mag sulph
MI and their corresponding coronary artery/ECG changes?
Anteroseptal: V1-V4 = LAD
Inferior: II, III, aVF = Right coronary
Anterolateral: V4-6, I, aVL = LAD or L circ
Lateral: I, AVL +/- V5/6 = L circ
Posterior: Tall R waves V1-2 = L circ or right coronary
HOCM management?
Management Amiodarone Beta-blockers or verapamil for symptoms Cardioverter defibrillator Dual chamber pacemaker Endocarditis prophylaxis*
Drugs to avoid
nitrates
ACE-inhibitors
inotropes