Cardio Overall Flashcards

1
Q

What is RAAS?

A

Renin-Angiotensin Aldosterone system, increases salt & water retention, increasing ECFV

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2
Q

What is ADH?

A

Antidiuretic hormone, increases water reabsorption, increasing ECFV

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3
Q

What are NPs?

A

Natriruetic peptides, causes water and salt excretion, opposes RAAS, decreasing ECFV

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4
Q

How do you treat unstable Angina/NSTEMI?

A

Aspirin, Fondaparinux, Ticagrelor

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5
Q

How do you treat IHD?

A

Lipid lowering drugs (statins eg fluvastatin, fibrates eg fenofibrate and Ezetimibe)

Beta-blockers eg Propranolol and metoprolol

Ca channel blockers eg amlodipine (can cause ankle oedema)

Antiplatelets eg aspirin and clopidogrel

Coronary revascularisation – Angioplasty and CABG (Coronary Artery Bypass Graft)

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6
Q

How do you treat Chronic Heart failure?

A

ACEI if cough, ARB.
Spironolactone
Beta blocker

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7
Q

How do you teat Hypercholesterolaemia?

A

Statins (eg fluvastatin)
Fibrates,
Ezetimibe

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8
Q

How do you treat Hypertension?

A

<55 and not black use ACEI. 55+/black = CCB

Not black: + CCB. Black: + ARB

+ Thiazide Diuretic

+ Extra Diuretic depending on serum K levels

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9
Q

How do you treat acute MI?

A
MONA+C:
Morphine + metoclopramide
O2
Nitroglycerine
Aspirin 300mg
Clopidogrel 600mg
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10
Q

How do you treat Acute heart failure?

A
LMNOP:
Lasix (Furosemide)
Morphine
Nitrate
O2 if <90%
Position - upright
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11
Q

How can Acute heart failure present?

A

Oedema
Tachypnea
Tachycardia
3rd Heart sound

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12
Q

What are the 3 main coronary arteries?

A

LAD, Right marginal and Circumflex

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13
Q

What is the first branch of the aorta?

A

The coronary arteries

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14
Q

What is the route of Heart conduction?

A

SAN (made of pacemaker cells)-> atria and AVN -> Purkinje fibres -> right and left fibres

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15
Q

What is S1?

A

1st Heart sound, Tricuspid and Mitral valves closing

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16
Q

What is S2?

A

2nd Heart sound, Aortic and Pulmonary valves closing

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17
Q

What is S3?

A

Early passive filling against taut LV. Abnormal in elder cardio patients, due to left ventricular systolic dysfunction, refer for echocardiography.

18
Q

What is S4?

A

4th heart sound, late active filling against taut LV. Indicates hypertension and AS, refer for ECG

19
Q

What is the name for a high pitched ejection systolic murmur that can radiate to the carotids?

A

Aortic stenosis

20
Q

What is the name of a murmur that is high pitched and pan-systolic?

A

Mitral regurgitation

21
Q

What is the name of a murmur that is early diastolic soft rumbling?

A

Aortic regurgitation

22
Q

What is the name of a murmur that is mid diastolic and low pitched?

A

Mitral stenosis

23
Q

What’s the ECG Axis if Lead I and aVF are positive?

24
Q

What’s the ECG Axis if Lead I is positive and Lead aVF is negative?

25
What's the ECG Axis if Lead I is negative and Lead aVF is positive?
RAD
26
What's the ECG Axis if Lead I and Lead aVF are both negative?
Extreme Axis Deviation
27
What's the normal PR interval?
0.12-0.2 secs
28
What's the normal QRS complex duration?
≤ 0.12
29
What does a prolonged PR interval mean?
1st degree Heart block
30
What does it mean if the PR intervals get increasingly longer and eventually drop?
2nd degree heart block, mobitz I
31
What heart block involves a skipped beat?
2nd degree heart block, mobitz II
32
What heart block involves no relationship between the P and Q waves?
Third degree/complete heart block
33
What view of the heart is represented by leads V2-5?
Anterior
34
What view of the heart is represented by leads V1-3?
Anteroseptal
35
What view of the heart is represented by leads V4-6, I & aVL?
Anterolateral
36
What view of the heart is represented by leads II, III, aVF?
inferior
37
What view of the heart is represented by leads V1 & V2?
Posterior
38
Is ventricular tachycardia shockable?
Yes
39
Is Ventricular Fibrilllation shockable?
Yes
40
What's an example and mechanism of ACEI?
Ramirpil, Blocks angiotensin
41
What view of the heart is represented by leads I, V5 & V6?
Lateral
42
If Leads V3 and V4 have ST elevation, what coronary artery is most likely affected?
LAD