Cardio Flashcards
Mx of VT/VF
defib!
if awake, anaesthetist GA/midazolam then defib
if cant have GA, amiodarone IV +/- BB
symptoms of VF
syncope/ LOC
symptoms of VT
palpitations
SOB
syncope/pre-syncope
chest pain
causes of VT/VF
MI drugs LV impairment electrolytes channelopathies [long QT/Brugada] HCM
if Pt went into VT/VF due to MI, recurrence not v likely unless another MI.
If cause is still there e.g. HCM, how would you Mx?
amiodarone/ BB
ICD [internal cardiac defib]
[maybe ablation]
VT ECG findings
broad complex
regular
VF ECG findings
broad complex
irregular
Atrial flutter ECG finding
saw tooth
regular
SVT ECG findings
narrow complex
SVT sx
palpitations
Mx of SVT
vagal manoeuvres [syringe + carotid massage]
adenosine 6mg, then try 12mg
verapamil
if compromised, dc cardioversion
long-term - BB, flecainide, CCB, (ablation)
causes of AF
age big LA HF mitral disease hyperthyroid HtN MI > LV damage
tool to decide whether to anticoagulate someone with AF & score to anticoag
CHADS2VASC
>1 male, >2 female
chronic AF Mx
warfarin/NOAC
metoprolol [/diltiazem/verapamil/amiodarone]
digoxin in sedentary
cardioversion +/- amiodarone, or flecainide
should the AVR lead on an ECG have a positive or negative tracing
-ve
how can you identify a patient is in sinus rhythm from an ECG
every QRS must be preceded by a P wave [impulse originates from sinus node]
regular
rate 60-100
how do you work out the axis from an ECG
lead 1 and AVF should both be positive
what is sinus arrhythmia
slight shortening and lengthening with respiration, common in young
what causes a prolonged PR interval
heart block
what causes a short PR interval
accessory pathway e.g. WPW
how long should the PR interval be
3-5 small squares
describe the degrees of heart block
1st = constant prolonged PR 2nd = mobitz 1 lengthening, then drops 1. mobitz 2 constant prolonged then drops 1. 3rd = no relationship between P + QRS
mx of heart block
pacemaker
define heart block
disrupted passage of impulse through AVN
causes of 1st and 2nd degree heartblock
IHD/MI myocarditis athletes sick sinus syndrome drugs - digoxin, B blocker
what is sick sinus syndrome
dysfunctional sinus node (fibrosis)
can cause brady/tachycardia, AF, sinus pause
usually in the elderly
what causes a deep / pathological Q wave on ECG
MI
what causes a tall/big QRS on ECG
LV hypertrophy
what causes a long/ wide QRS
BBB [ventricle conduction problem]
sign of hyperkalaemia on ECG
tall tented T waves
what does T wave inversion on ECG indicate
infarct/ ischaemia [MI/IHD]
leads II, III, and AVF affected. Likely site of infarct + vessel
inferior. RCA
leads I, aVL, V4-6 affected. Likely site of infarct + vessel
lateral, circumflex
leads V1-3 affected. Likely site of infarct + vessel
anterioseptal, LAD
causes of long-QT on ECG
genetic predisposition [long-QT syndrome]
drugs: antipsychotics, macrolides
hypocalc/hypokal
what hormone abnormality might cause someone to go in and out of AF
hyperthyroidism
If the SAN gives HR of 100bpm, what slows the heart rate?
vagal tone (activity of the vagus nerve)
signs of ischaemia/ infarct on ECG
ST elevation/ depression
T wave inversion
Q waves
blood results/ biomarkers that might be seen in alcoholism
^GGT
low urea
^MCV
[^AST + ALT]
brugada on ecg
high J point
coved ST elevation
“saddleback” between ST + T wave
what causes brugada
autosomal dominant
causing Na+ chanelopathy
brugada Sx
syncope
sudden death
brugada Mx
internal defib
what causes a bifid P wave
1 atria hypertrophy e.g. mitral stenosis
in terms of the level of damage to myocardium, what do STEMI and NSTEMI represent
STEMI = transmural infarct NSTEMI = ischaemia/ not fully occluded vessel
likely troponin findings in STEMI, NSTEMI and unstable angina
stemi + nstemi = raised troponin
unstable angina not
immediate Tx of suspected MI
300mg aspirin morphine 5-10mg anti-emetic e.g. metoclopramide 10mg anticoag: bivalirudin/enoxaparin/fondaparinux [O2] [GTN] cath lab for PCI
consequences of MI
cardiac arrest
HF
VF
AF
long term Mx of ACS
aspirin + clopidogrel anticoag [fondaparinux] til discharge atorvastatin BB ACEi STOP SMOKING antiHTN, DM Mx
usual pathology in ACS
+ rarer causes
plaque rupture
thrombosis
inflammation
vasculitis
emboli
coronary spasm
causes of secondary hyperTN
cushings conn's disease phaeochromocytoma renal disease coarctation of the aorta drugs: NSAIDs, COCP, steroids
secondary causes of hyperlipidaemia
renal failure liver disease hypothyroidism diabetes excess alcohol biliary obstruction drugs: steroids, oestrogens
what is the side effect of statins, how would someone present and how would you investigate this?
myositis
muscle tenderness
CK
53 yr old hypertensive male Pt presents w/ sudden onset severe central CP, radiating to scapular.
BP low, sweaty, pale, early diastolic murmur, ECG shows LVH only. CXR widened mediastinum. Main differential?
aortic dissection
how do you confirm a suspected aortic dissection?
CT
how do you manage BP 80/40 in Pt with aortic disection/
conservatively /permissive hypoTN
until aorta repaired
provided Pt awake [cerebral perfusion] + bilateral radial pulses
68 yr old w/ dizzy spells increasing in frequency + w/ exertion.
Hx of angina, neuro exam normal, BP 110/70, sinus rhythm, systolic murmur which radiates across precordium + to carotids. Likely diagnosis?
aortic stenosis
Ix technique for suspected aortic stenosis
transthoracic echo
68 yr old ^SOB, palpitations, CP. HR 160, BP 88/40, sat 92, RR 22, creps. ECG shows AF. Acute Mx of the tachyarrhythmia?
electrical cardioversion [with sedation or GA]
ECG axis deviation - looking at lead I and lead II, what will you notice in left and right axis deviation
Left Leaving [away from each other]
Right axis deviation - pointing towards each other
on ECG, how many little squares should the PR interval and the QRS complex be
PR - 3-5 small squares
QRS - up to 3 small squares
what is the most common heart valve problem after MI?
mitral regurg
signs of bleeding in your patient
visible bleeding low BP tachycardia weak pulses cold/clammy peripheries prolonged cap refill not talking [=not perfusing brain]
what movement improves pericarditic pain
leaning forward
what would you notice in BP in aortic dissection?
unequal between L and R arms
causes of palpitations
arrhythmia: sinus tachy, ectopics, AF, SVT, VT
Thyrotoxicosis
Anxiety
Phaeochromocytoma
when do you see a J wave on ECG
hypothermia
SAH
hypercalcaemia
causes of sinus bradycardia
fitness vasovagal sick sinus syndrome drugs: BB, digox, amiod hypothyroid hypothermia ^ICP cholestasis
common causes of AF
IHD/MI HF HTN thyrotox alcohol obesity
causes of 3rd degree heart block
IHD/MI idiopathic fibrosis congenital aortic valve calcification cardiac trauma/ surg digoxin toxicity infiltration: abscess, granuloma, tumour, parasite
what is prinzmetals angina?
coronary spasm
causes of T wave inversion
many including ischemia, BBB, hypertrophy, PE
PE ECG changes
sinus tachy
RBBB
RV strain [R axis dev, dominant R wave, T wave inv/ ST dep in V1/V2]
(SIQIIITIII)
hyperkal on ECG
tall tented T waves
wide QRS
absent P
‘sine wave’ appearance
hypokal on ECG
small flattened T waves
prominent U waves
peaked P waves
hypercalc on ECG
short QT
hypocalc on ecg
long QT
small T waves
what are bifascicular and trifascicular block
bi = RBBB + left bundle hemi block
manifests as axis deviation
tri = bi + 1st degree heart block