Cardio 4 Flashcards

1
Q

What should be done in a patient with symptomatic (incl. haemodynamically unstable) bradycardia which is not responding to atropine

A

insert temporary pacemaker

NOTE: this is also management of choice if type 2/3 heart block post anterior MI or prior to surgery for trifasicular block

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2
Q

Bradycardia

  1. a) when is management indicated?
    b) how is it managed?
  2. When is there and increased risk of asystole (and therefore need specialist help)
A
  1. a)
    - shock: hypotension (sys. <90), pallor, sweating, cold, confusion
    - syncope
    - heart ischaemia / failure

b)
500mcg atropine IV

if insufficient response:

  • atropine up to 3mg
  • transcutaneous pacing
  • adrenaline
    • complete heart block with wide QRS or type 2 heart block
    • ventricular pause >3s
    • recent asystole
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3
Q

Tachycardia

  1. a) when is the patient seen as unstable?
    b) what is done if this is the case?
  2. Broad-complex tachycardia (VT) - what should be done if
    a) regular
    b) irregular
  3. Narrow-complex tachycardia (SVT) - what should be done if
    a) regular
    b) irregular
A

1.

a)
- shock: hypotension (sys. <90), pallor, sweating, cold, confusion
- syncope
- heart ischaemia / failure

b) 3 DC shocks

  1. a) loading dose of amiodarone followed by 24hr infusion (alternatively lidocaine or procainamide)

NOTE: verapamil contraindicated in VT

b) seek expert help

  1. a)
  2. vagal manœuvres
  3. IV adenosine
    (verapamil if asthmatic)
  4. electrical cardioversion

b) likely AF (follow guidance of that)
- <48hrs can cardiovert
- rate control (beta blockers)

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4
Q

Pulmonary Artery Occlusion Pressure

  1. What is this an indirect measure of?
  2. What should you suspect if
    a) low
    b) low + pulmonary oedema
    c) high
A
  1. left atrial pressure
  2. a) hypovolaemia
    b) ARDS
    c) fluid overload
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5
Q

What is the most common cause of SVT?

A

AV nodal re-entry tachycardia
-> 2 separate conduction pathways within the AV node
(will see 2 different QRS complexes one wider than the other)
-> P wave will follow QRS as conduction originates in AV node

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6
Q

Rheumatic Fever

  1. What is it?
  2. How is it diagnosed?
  3. What is the management?
A
  1. an immune reaction (2-6 weeks after) to recent strep progenies infection
  2. evidence of recent strep infection (strep antibodies, antigens or positive throat swab)
    AND
    2 major / 1 major+2 minor criteria
Major 
sydenham's chorea 
erythema marginatum 
endocarditis (pancarditis) 
poly arthritis 
subcutaneous nodules 

mnemonic: myosin SEEPS into range immune attack against protein M of strep pyogenes

minor:
- raised ESR / CRP
- fever
- prolonged PR interval

    • penicillin V
    • NSAIDs
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7
Q

Prosthetic heart valves

  1. Biological valves (from cow or pig)
    a) What is the disadvantage?
    b) What drugs are required post op?
  2. Mechanical Valves
    a) What is the disadvantage?
    b) Lifelong warfarin indicated. What is the target INR in
    i) mitral
    ii) aortic
A
  1. a) calcification / degeneration of valve

b)
- life-long aspirin
+/- 3 months warfarin depending on risk

2.

a) risk of thrombosis
b)
i) 3.5
ii) 3.0

THINK: higher INR for mitral as blood more likely to pool in atrium than ventricle

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8
Q

State the vein which

  1. originates at the first digit and passes up the medial aspect of the leg draining into the femoral vein at the femoral triangle
  2. originates at the fifth digit and passes up the lateral aspect of the foot and posterior aspect of leg to drain into popliteal vein at knee
A
  1. great / long saphenous vein

2. short saphenous vein

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9
Q

Takayasu’s arteritis

  1. Who is it more common in?
  2. What clinical features are seen?
  3. What is it associated with?
A

1.

  • 10-40
  • females
  • asian people
2. 
typically causes occlusion of aorta 
- unequal BP in upper limb
- limb claudication 
- absent / weak lower limb pulses 
- carotid bruit / tenderness 
  1. renal artery stenosis
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10
Q

Additionally to VT management, what should be given in torsades de pointes?

A

IV magnesium sulphate

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11
Q

Wolff-Parkinson White

  1. What is it?
  2. What clinical features can be seen on ECG?
  3. How would you differentiate between types A and B?
  4. How is it managed?
A
  1. congenital accessory pathway between the atria and ventricles causing an atrioventricular re-entry tachycardia
    - > accessory pathway cannot slow conduction in AF and therefore AF can rapidly progress to VF
    • wide QRS complex with slurred upstroke ‘delta wave’
    • short PR interval
  2. type A - left accessory pathway: RAD and prominent R wave in V1

type B - right accessory pathway: LAD and NO prominent R wave

  1. radio frequency ablation of accessory pathway
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12
Q

What NSAID is contraindicated in CV disease?

A

diclofenac

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