Cardiac tamponade Flashcards

1
Q

A patient is post-operative Day 1 status post-coronary artery bypass graft. They complain of new-onset shortness of breath at rest. Your assessment reveals muffled heart sounds, jugular venous distention, decreased pulses with inspiration. What is your suspected diagnosis?
A. Cardiac tamponade
B. Pulmonary hypertension
C. Respiratory failure
D. Fluid volume overload

A

Correct Answer: A. Cardiac tamponade

Rationale:
The presentation—new-onset shortness of breath, muffled heart sounds, jugular venous distention, and decreased pulses with inspiration (suggesting pulsus paradoxus)—is highly indicative of cardiac tamponade. This is a life-threatening condition that can occur post-cardiac surgery and requires immediate intervention.

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2
Q
  1. (Tamponade / Will Kill)
    A post-cardiac surgery patient presents with jugular venous distension, muffled heart sounds, hypotension, and pulsus paradoxus. What is the emergent intervention?
    A. Urgent pericardiocentesis
    B. Delayed follow-up in 24 hours
    C. IV beta-blocker infusion
    D. Oral diuretics
A

Correct Answer: A. Urgent pericardiocentesis

Rationale:
The patient presents with classic signs of cardiac tamponade—jugular venous distension, muffled heart sounds, hypotension, and pulsus paradoxus—which is a life-threatening emergency. Urgent pericardiocentesis is required to promptly remove the pericardial fluid and relieve the pressure on the heart, thereby restoring adequate cardiac output. Delaying treatment or using medications that do not address the underlying issue would likely result in rapid deterioration.

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3
Q

Which delayed intervention in a patient showing signs of tamponade physiology could severely worsen outcomes?
A. Early imaging to confirm effusion
B. Reversal of anticoagulation if applicable
C. Reassurance and outpatient management
D. Pericardial window procedure

A

C. Reassurance and outpatient management

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4
Q
  1. For cardiac tamponade, which intervention is critical to restore hemodynamics?
    A. Observation and outpatient follow-up
    B. Urgent pericardiocentesis
    C. Administration of beta blockers
    D. High-dose aspirin
    o
A

Answer: B
o Rationale: Urgent pericardiocentesis relieves pressure on the heart, restoring cardiac output in tamponade.

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5
Q
  1. Which of the following best describes cardiac tamponade?
    A. Inflammation of the pericardium without effusion
    B. Accumulation of fluid in the pericardial space causing impaired ventricular filling
    C. Thickening of the pericardial sac due to fibrosis
    D. Enlargement of the heart chambers due to volume overload
A

o Answer: B
o Rationale: Cardiac tamponade is defined by the accumulation of fluid in the pericardial sac that compresses the heart and impairs ventricular filling, leading to reduced cardiac output.

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6
Q
  1. What is the primary life‐threatening consequence of cardiac tamponade?
    A. Progressive pericardial thickening
    B. Decreased cardiac output resulting in circulatory collapse
    C. Increased heart rate leading to tachyarrhythmia
    D. Chronic inflammation leading to constrictive pericarditis
A

o Answer: B
o Rationale: When intrapericardial pressure exceeds intracardiac pressure, ventricular filling is compromised, causing a critical drop in cardiac output that can result in shock and cardiac arrest.

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7
Q
  1. Beck’s triad in cardiac tamponade consists of all EXCEPT:
    A. Hypotension
    B. Jugular venous distension
    C. Muffled heart sounds
    D. Hypertension
A

o Answer: D
o Rationale: Beck’s triad comprises hypotension, jugular venous distension, and muffled heart sounds. Hypertension is not a feature of tamponade.

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8
Q
  1. Pulsus paradoxus is defined as a decrease in systolic blood pressure of greater than what value during inspiration?
    A. 5 mm Hg
    B. 10 mm Hg
    C. 15 mm Hg
    D. 20 mm Hg
A

o Answer: B
o Rationale: Pulsus paradoxus is classically defined as a drop in systolic blood pressure greater than 10 mm Hg during inspiration, indicating significant pericardial pressure.

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9
Q
  1. Which imaging modality is considered the gold standard for diagnosing cardiac tamponade?
    A. Chest X-ray
    B. Computed Tomography (CT)
    C. Echocardiography
    D. Magnetic Resonance Imaging (MRI)
A

o Answer: C
o Rationale: Echocardiography is the gold standard because it can directly visualize the effusion, assess chamber collapse, and detect hemodynamic compromise.

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10
Q
  1. On echocardiography, the “swinging heart” sign is indicative of:
    A. Severe left ventricular hypertrophy
    B. Cardiac tamponade
    C. Aortic dissection
    D. Constrictive pericarditis
A

Answer: B
o Rationale: The “swinging heart” sign occurs when the heart moves within a large, fluid-filled pericardial sac, which is a classic finding in cardiac tamponade

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11
Q
  1. Which right heart chamber is most vulnerable to collapse in cardiac tamponade due to its thin wall?
    A. Right atrium
    B. Right ventricle
    C. Left atrium
    D. Left ventricle
A

o Answer: A
o Rationale: The right atrium is typically the first chamber to collapse during tamponade because of its lower pressure and thinner wall.

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12
Q
  1. Which of the following EKG findings is commonly seen in patients with cardiac tamponade?
    A. High voltage QRS complexes
    B. Low voltage QRS complexes with diffuse ST and T wave changes
    C. Pathologic Q waves in the precordial leads
    D. Delta waves
A

o Answer: B
o Rationale: Low voltage QRS complexes with diffuse ST and T wave changes are common EKG findings due to the insulating effect of the pericardial fluid.

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13
Q
  1. A patient in cardiac tamponade is tachycardic. What is the physiological reason for this tachycardia?
    A. To compensate for reduced stroke volume
    B. Due to direct myocardial irritation by the effusion
    C. As a side effect of pericardiocentesis
    D. Because of high sympathetic drive from pain alone
A

o Answer: A
o Rationale: Tachycardia is a compensatory mechanism to maintain cardiac output when stroke volume is reduced by impaired ventricular filling

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14
Q
  1. In the management of cardiac tamponade, what is the immediate definitive treatment?
    A. Administration of beta-blockers
    B. Pericardiocentesis
    C. High-dose diuretics
    D. Intravenous thrombolytics
A

o Answer: B
o Rationale: Pericardiocentesis is the definitive procedure to remove pericardial fluid and relieve the pressure on the heart.

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15
Q
  1. The term “blind pericardiocentesis” refers to which of the following?
    A. Using only clinical signs without imaging guidance
    B. Utilizing echocardiography during the procedure
    C. Performing the procedure under fluoroscopic guidance
    D. Draining fluid only from the left ventricular side
A

o Answer: A
o Rationale: A blind pericardiocentesis is performed based solely on anatomical landmarks and clinical findings without real-time imaging guidance, although it carries higher risk.

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16
Q
  1. When performing a pericardiocentesis, which approach is most commonly used?
    A. Transradial approach
    B. Subxiphoid approach
    C. Transaortic approach
    D. Transesophageal approach
A

o Answer: B
o Rationale: The subxiphoid approach is the most common method for pericardiocentesis, providing access to the pericardial space with a relatively low risk of complications.

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17
Q
  1. What is the rationale for using echocardiography-guided pericardiocentesis over a blind approach?
    A. It is faster to perform
    B. It reduces the risk of injury to adjacent structures
    C. It increases the amount of fluid removed
    D. It eliminates the need for sedation
A

o Answer: B
o Rationale: Echocardiography guidance allows for precise needle placement, minimizing complications such as injury to the heart or coronary vessels.

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18
Q
  1. Which patient symptom is most concerning and suggests an imminent circulatory collapse in cardiac tamponade?
    A. Severe anxiety with a sense of impending doom
    B. Mild chest discomfort
    C. Occasional palpitations
    D. Intermittent headaches
A

o Answer: A
o Rationale: An extreme sense of anxiety or impending doom is often reported by patients in critical states, reflecting the significant hemodynamic compromise of tamponade.

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19
Q
  1. In the initial management of low cardiac output in cardiac tamponade, which of the following is most appropriate?
    A. Administration of fluids and inotropes
    B. Immediate use of high-dose vasopressors
    C. Initiation of beta-blocker therapy
    D. Immediate pericardiectomy
A

o Answer: A
o Rationale: Fluid resuscitation and inotropic support help maintain cardiac output temporarily until definitive drainage can be performed.

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20
Q
  1. Which of the following complications may occur if pericardiocentesis is not performed promptly in a patient with tamponade?
    A. Ventricular septal defect
    B. Progression to cardiac arrest
    C. Atrial fibrillation
    D. Myocardial infarction
A

o Answer: B
o Rationale: Untreated cardiac tamponade can lead to a complete collapse of cardiac output and subsequent cardiac arrest.

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20
Q
  1. What is the “water bottle sign” on chest X-ray indicative of?
    A. Aortic dissection
    B. Cardiac tamponade
    C. Pulmonary edema
    D. Enlarged cardiac silhouette due to pericardial effusion
A

o Answer: D
o Rationale: The “water bottle sign” is a radiographic appearance of an enlarged, globular cardiac silhouette, which is typically seen with a large pericardial effusion.

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21
Q
  1. Which of the following is an essential nonpharmacologic management step for a patient with suspected cardiac tamponade?
    A. Aggressive physical therapy
    B. Continuous telemetry monitoring
    C. High-intensity exercise
    D. Early ambulation
A

o Answer: B
o Rationale: Continuous telemetry monitoring is crucial in detecting rapid changes in hemodynamics and arrhythmias in a patient with tamponade.

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22
Q
  1. In a case of traumatic cardiac tamponade due to penetrating injury, what additional management step is often required?
    A. Immediate pericardiocentesis alone
    B. Surgical intervention to repair the injury
    C. High-dose NSAIDs
    D. Observation in the intensive care unit
A

o Answer: B
o Rationale: Traumatic tamponade often requires surgical intervention to address the source of bleeding and repair cardiac or vascular injuries.

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23
Q
  1. How does the rate of fluid accumulation affect the development of cardiac tamponade?
    A. Rapid accumulation requires a larger volume to cause tamponade
    B. Rapid accumulation requires a smaller volume to cause tamponade
    C. Slow accumulation is always more dangerous
    D. The rate of accumulation does not affect tamponade development
A

o Answer: B
o Rationale: Rapid fluid accumulation does not allow the pericardium time to stretch, so even a small volume can cause significant hemodynamic compromise.

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24
Q
  1. Which underlying conditions can lead to cardiac tamponade besides pericarditis?
    A. Post-surgical bleeding
    B. Traumatic injury
    C. Aortic dissection
    D. All of the above
A

o Answer: D
o Rationale: Cardiac tamponade can occur from a variety of etiologies including post-surgical bleeding, trauma, and aortic dissection, among others.

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25
Q
  1. In the setting of aortic dissection, how can cardiac tamponade develop?
    A. Dissection-induced inflammation causing fluid accumulation
    B. Rupture of the dissection into the pericardial sac
    C. Increased intrathoracic pressure from dissection
    D. Direct compression of the heart by the dissected aorta
A

o Answer: B
o Rationale: Aortic dissection may lead to rupture into the pericardial space, rapidly accumulating blood and causing tamponade.

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26
Q
  1. What is the effect of intrapericardial pressure exceeding intracardiac pressure?
    A. Enhanced coronary perfusion
    B. Impaired venous return and decreased ventricular filling
    C. Increased stroke volume
    D. Improved cardiac output
A

o Answer: B
o Rationale: When intrapericardial pressure exceeds intracardiac pressure, it restricts ventricular filling, reducing cardiac output and venous return.

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27
Q
  1. Which clinical sign is considered a hallmark of decreased cardiac output in tamponade?
    A. Hypertension
    B. Altered mental status
    C. Warm, well-perfused extremities
    D. Bradycardia
A

o Answer: B
o Rationale: Reduced cardiac output can lead to poor cerebral perfusion, resulting in altered mental status among other signs of shock.

28
Q
  1. What is the role of inotropic support in the management of cardiac tamponade?
    A. To decrease myocardial oxygen demand
    B. To improve contractility and temporarily augment cardiac output
    C. To directly remove the pericardial fluid
    D. To decrease heart rate
A

o Answer: B
o Rationale: Inotropes enhance myocardial contractility, which can temporarily help maintain cardiac output until definitive treatment is available.

29
Q
  1. Which feature on an echocardiogram is most specific for cardiac tamponade?
    A. Left ventricular hypertrophy
    B. Right ventricular diastolic collapse
    C. Atrial septal defect
    D. Mitral valve prolapse
A

o Answer: B
o Rationale: Right ventricular diastolic collapse is a highly specific echocardiographic sign indicating elevated pericardial pressures consistent with tamponade.

30
Q
  1. A patient with suspected tamponade presents with marked tachycardia and hypotension. What should be the immediate next step?
    A. Start intravenous beta-blockers
    B. Urgent pericardiocentesis
    C. Schedule an elective pericardial window
    D. Increase the patient’s fluid restriction
A

o Answer: B
o Rationale: In the face of hemodynamic instability, urgent pericardiocentesis is necessary to relieve the pressure on the heart.

31
Q
  1. What laboratory findings might you expect in a patient with cardiac tamponade secondary to malignant pericardial effusion?
    A. Elevated inflammatory markers only
    B. Cytology positive for malignant cells in pericardial fluid
    C. Normal pericardial fluid analysis
    D. Low protein levels in the fluid
A

o Answer: B
o Rationale: In malignant effusions, pericardial fluid cytology may reveal malignant cells, which aids in determining the underlying etiology.

31
Q
  1. What potential risk is associated with blind pericardiocentesis?
    A. Increased risk of infection
    B. Injury to the coronary arteries or myocardium
    C. Excessive fluid removal
    D. Over-reliance on echocardiography
A

o Answer: B
o Rationale: Without imaging guidance, there is a higher risk of inadvertently injuring nearby cardiac structures during needle insertion.

32
Q

injuring nearby cardiac structures during needle insertion.
29. In the context of tamponade, why is the pericardial window procedure sometimes performed?
A. To permanently remove the pericardium
B. To create a drainage route for recurrent effusions
C. To prevent arrhythmias
D. To reduce the risk of myocardial infarction

A

o Answer: B
o Rationale: A pericardial window creates a continuous drainage route, helping prevent re-accumulation of fluid in recurrent effusions.

33
Q
  1. Which of the following is a common iatrogenic risk during management of cardiac tamponade?
    A. Exacerbation of pericardial effusion from excessive fluid administration
    B. Arrhythmias induced during needle insertion
    C. Infection from the drainage catheter
    D. Over-sedation during the procedure
A

o Answer: B
o Rationale: During pericardiocentesis, mechanical irritation of the myocardium can precipitate arrhythmias, representing an important iatrogenic risk.

34
Q
  1. In patients with cardiac tamponade, why is it critical to monitor oxygenation continuously?
    A. Because pericardiocentesis often causes hypoxemia
    B. To detect early signs of respiratory failure secondary to low cardiac output
    C. To guide fluid therapy
    D. To determine if mechanical ventilation is needed for sedation
A

Answer: B
o Rationale: Low cardiac output can lead to poor tissue oxygenation, making continuous oxygen monitoring essential to detect respiratory compromise.

35
Q
  1. Which scenario best illustrates a rapid development of cardiac tamponade?
    A. A slow accumulation of 2,000 mL over several weeks
    B. An acute hemorrhage of 200 mL within minutes
    C. A gradually increasing effusion from chronic pericarditis
    D. Effusion secondary to long-standing renal failure
A

Answer: B
o Rationale: A rapid hemorrhage can quickly overwhelm the pericardial stretch capacity, leading to acute tamponade with even a relatively small volume of blood.

36
Q
  1. What is the main mechanism by which pericardial fluid accumulation leads to hypotension in tamponade?
    A. Direct myocardial ischemia
    B. Compression of the heart that limits ventricular filling
    C. Reflex vasodilation
    D. Increased afterload due to effusion
A

o Answer: B
o Rationale: The accumulated fluid compresses the heart, especially during diastole, reducing ventricular filling and ultimately decreasing cardiac output and blood pressure.

37
Q
  1. In the setting of post-cardiac surgery, what is a common cause of cardiac tamponade?
    A. Aortic dissection
    B. Postoperative bleeding into the pericardial space
    C. Viral pericarditis
    D. Drug-induced effusion
A

o Answer: B
o Rationale: Bleeding following cardiac surgery can accumulate in the pericardial space, resulting in tamponade if not promptly recognized and treated.

38
Q
  1. Why might a patient with cardiac tamponade experience altered mental status?
    A. Due to direct central nervous system infection
    B. From decreased cerebral perfusion secondary to low cardiac output
    C. As a side effect of sedative medications
    D. Because of hypercapnia from respiratory failure
A

o Answer: B
o Rationale: Low cardiac output leads to diminished cerebral perfusion, which can manifest as confusion or altered mental status.

39
Q
  1. Which clinical intervention may inadvertently worsen hemodynamics if performed before fluid resuscitation in tamponade?
    A. Urgent pericardiocentesis
    B. Administration of positive pressure ventilation
    C. Intravenous fluid administration
    D. Use of inotropic agents
A

o Answer: B
o Rationale: Positive pressure ventilation can further decrease venous return and worsen cardiac output in a patient with tamponade, making cautious management essential.

40
Q
  1. Which hemodynamic measurement is most likely to be elevated in cardiac tamponade?
    A. Left ventricular end-diastolic pressure
    B. Central venous pressure
    C. Cardiac output
    D. Systemic vascular resistance
A

o Answer: B
o Rationale: Elevated central venous pressure is a classic hemodynamic finding in tamponade due to impaired right heart filling.

41
Q
  1. What role does the pericardium normally play that becomes detrimental when fluid accumulates rapidly?
    A. It normally facilitates rapid cardiac filling
    B. It normally prevents overdistension of the heart but becomes a constrictive shell when fluid accumulates
    C. It normally regulates coronary blood flow
    D. It normally produces pericardial fluid
A

o Answer: B
o Rationale: The pericardium restricts sudden cardiac dilation; however, in tamponade, this restraint becomes detrimental as even small volumes of fluid can markedly impede cardiac filling.

42
Q
  1. In a patient with cardiac tamponade, what is the expected effect on pulmonary pressures?
    A. Markedly decreased pulmonary pressures
    B. Normal pulmonary pressures
    C. Elevated pulmonary pressures due to backup from impaired left heart filling
    D. Fluctuating pulmonary pressures with respiration
A

o Answer: C
o Rationale: Impaired filling and reduced cardiac output can lead to elevated pulmonary pressures as blood backs up into the pulmonary circulation.

43
Q
  1. Which of the following is an important monitoring tool after pericardiocentesis to detect re-accumulation of fluid?
    A. Serial chest X-rays
    B. Continuous echocardiographic evaluation
    C. Daily EKGs
    D. Routine measurement of serum electrolytes
A

o Answer: B
o Rationale: Continuous or serial echocardiography is crucial after pericardiocentesis to monitor for re-accumulation of pericardial fluid.

44
Q
  1. Which patient demographic is LEAST likely to develop cardiac tamponade from idiopathic pericarditis?
    A. Young adults
    B. Elderly patients
    C. Patients with autoimmune conditions
    D. Patients with malignancy
A

o Answer: A
o Rationale: Idiopathic pericarditis is more common in young adults, but they are less likely to develop tamponade compared to patients with comorbid conditions like malignancy or autoimmune diseases, where effusions can be larger and more rapid.

45
Q
  1. In the management of cardiac tamponade, why is rapid recognition essential?
    A. Because the effusion is always malignant
    B. To prevent progression to irreversible myocardial injury and cardiac arrest
    C. So that glucocorticoids can be started immediately
    D. To ensure the patient is discharged from the hospital quickly
A

o Answer: B
o Rationale: Early recognition and treatment of tamponade are critical to preventing irreversible hemodynamic collapse and potential cardiac arrest

46
Q
  1. A patient with a history of cancer presents with signs of tamponade. Which additional diagnostic test may help determine the etiology of the effusion?
    A. Coronary angiography
    B. Pericardial fluid cytology
    C. Cardiac stress testing
    D. Holter monitoring
A

o Answer: B
o Rationale: In patients with malignancy, analysis of pericardial fluid for malignant cells (cytology) helps identify the underlying cause of the effusion.

47
Q
  1. Which statement best explains why a slow accumulation of fluid in the pericardium may be tolerated better than a rapid accumulation?
    A. The pericardium is unable to stretch over time
    B. The pericardium can gradually stretch, accommodating a larger volume before causing hemodynamic compromise
    C. Slow accumulation is usually sterile
    D. Slow accumulation always results in a transudative effusion
A

o Answer: B
o Rationale: A slowly accumulating effusion allows the pericardium time to stretch, meaning that a larger volume may be present before tamponade physiology develops.

48
Q
  1. What is the most common physical exam finding in cardiac tamponade?
    A. Loud heart sounds
    B. Clear lung fields
    C. Muffled heart sounds
    D. Peripheral edema
A

o Answer: C
o Rationale: Muffled or distant heart sounds are common due to the insulating effect of the pericardial fluid.

49
Q
  1. Which of the following is an example of a nonpharmacologic measure to support a patient with tamponade prior to definitive treatment?
    A. Initiation of high-dose steroids
    B. Strict bed rest and oxygen supplementation
    C. Aggressive diuresis
    D. Early mobilization
A

o Answer: B
o Rationale: Supportive care with bed rest and oxygen therapy can help stabilize the patient while preparing for definitive intervention.

50
Q
  1. How does cardiac tamponade affect intracardiac filling pressures?
    A. It decreases them uniformly
    B. It increases them due to external compression
    C. It has no effect on intracardiac pressures
    D. It selectively increases left ventricular pressures only
A

o Answer: B
o Rationale: External compression from the effusion elevates intracardiac filling pressures, especially in the right-sided chambers.

51
Q
  1. What is the significance of the rate of blood withdrawal during pericardiocentesis?
    A. It determines the risk of re-accumulation
    B. It ensures the procedure is completed before hypotension worsens
    C. Slow withdrawal minimizes the risk of arrhythmia
    D. It has no clinical significance
A

o Answer: C
o Rationale: A controlled, slow withdrawal of fluid helps reduce the risk of precipitating arrhythmias during the procedure.

52
Q
  1. Which of the following summarizes the critical management principles for cardiac tamponade?
    A. Identify and treat underlying causes, provide supportive care (fluids, inotropes), and perform urgent pericardiocentesis
    B. Immediately initiate diuretic therapy and delay invasive procedures
    C. Start high-dose beta-blockers and plan for elective surgery
    D. Use only noninvasive monitoring until the patient stabilizes
A

o Answer: A
o Rationale: Effective management involves rapidly identifying the cause, supporting hemodynamics with fluids and inotropes as needed, and performing urgent pericardiocentesis to relieve the pressure on the heart

53
Q

Cardiac tamponade is caused

A

by the rapid accumulation of fluid in the pericardial space, which compresses the heart and prevents proper ventricular filling.

54
Q

The severity of hemodynamic compromise depends largely

A

on the rate of fluid accumulation rather than the absolute volume.

55
Q

Rapidly developing tamponade can lead to

A

critically decreased cardiac output, circulatory collapse, and cardiac arrest if not promptly managed

56
Q

(hypotension, jugular venous distension, and muffled heart sounds) and pulsus paradoxus (drop in systolic blood pressure >10 mm Hg on inspiration) are key clinical markers indicating potential circulatory compromise.

A

o Beck’s triad

57
Q

o EKG findings often include low voltage QRS complexes and diffuse ST/T wave changes due to the insulating effect of the effusion.

57
Q

the gold standard for diagnosis, with findings such as the “swinging heart” sign and right ventricular diastolic collapse being highly specific.

A

Echocardiography

58
Q

EKG findings

A

often include low voltage QRS complexes and diffuse ST/T wave changes due to the insulating effect of the effusion.

59
Q

Chest X-ray may reveal

A

a “water bottle” cardiac silhouette, although this is less specific.

60
Q

the definitive treatment to relieve the pressure; delays can be fatal.

A

Urgent pericardiocentesis

61
Q

(e.g., careful fluid resuscitation and inotropic support) is essential while preparing for intervention.

A

Nonpharmacologic support

62
Q

iatrogenic harm, which increases the risk of injuring adjacent structures; thus, imaging guidance (echocardiography) is preferred.

A

Blind pericardiocentesis may cause

63
Q

as they can further compromise venous return and worsen hemodynamics.

A

Caution with interventions like positive pressure ventilation is warranted

64
Q

various etiologies such
as pericarditis (infectious, idiopathic, autoimmune), post-surgical bleeding, trauma, or aortic dissection.

A

Cardiac tamponade can result from

65
Q

critical to detect re-accumulation of fluid, often requiring serial echocardiography.

A

Monitoring after drainage

66
Q

vital to prevent irreversible myocardial injury and death.

A

Rapid recognition and intervention