cardiac anamotmy Flashcards
Cardiac Ischemia is caused by coronary atherosclerosis.
O2 Supply is lower than O2 Demand = Ischemia
Typical symptoms of Cardiac Ischemia:
Chest pain (angina);
Dyspnea;
Diaphoresis.
Which is the hallmark of ischemia?
Imbalance between oxygen supply and demand.
Stable angina - When the Symptons start to occur?
Must occlude ~75% of lumen to cause symptoms;
Exertion
Stable angina occur with…
Exertion and go away with rest.
Acute Coronary Syndromes are defined by rupture of an atherosclerotic plaque and formation of a thrombus.
True
Unstable angina and Non ST elevation myocardial infarction are subtotal occlusion of the artery.
True
Total occlusion of the vessel is noun as…
ST- elevation myocardial infarction
STEMI
Sudden Death is a common complication of CAD.
CAD is most common cause of sudden death adults.
The most common cause of sudden deaths in youger patients is…
Hypertrophic cardiomyopathy (HCM)
Plaque rupture can cause which type of arrhythmias?
Ventricular tachycardia;
Ventricular fibrillation;
Major risk for Coronary acute disease:
Prior coronary disease;
Coronary risk equivalents:
Diabetes;
Peripheralartery disease;
Chronic kidney disease;
Risk factors for coronary disease:
Hypertension;
Hyperlipidemia;
Family History;
Smoking;
Obesity;
Sedentary lifestyle;
Transmural ischemia occurs when the patient have…
COMPLETE occlusion of the vessel.
Subendocardial Ischemia occurs when the patien haven…
Subtotal occlusion
Limited distal flow
ST depressions are present in…
Subendocardial Ischemia
T wave inversions have many causes other than CAD.
Raised intracerebral pressure;
Resolving pericarditis;
Bundle branch blocks;
Ventricular hypertrophy;
ST elevations are seen only with transmural ischemia.
True
Hyperacute T Waves can be seen in transmural ischemia.
THEY ARE A EARLY SIGN OF ischemia.
Seen before ST elevations.
Poor R wave progression is a sign of…
Acute anterior wall infarction
Prior anterior wall infarction
The first 24 to 72 hours after ST-elevation myocardial infarction is the highest risk period for arrhythmias including ventricular fibrillation.
TRUE
The myocardium goes through a progression of gross and microscopic changes following infarction due to coagulative necrosis and inflammation. These changes can be used by pathologists to determine the cause of death in cases like the one described. By about 5 to 10 days, the myocardium becomes tan or yellow from infiltration of infarcted tissue with inflammatory cells. Microscopy will show necrosis of myocytes and macrophages performing phagocytosis. Granulation tissue appears at the margins of the infarcted area around days 7 to 10.
ok
The myocardium goes through a progression of gross and microscopic changes following infarction due to coagulative necrosis and inflammation. These changes can be used by pathologists to determine the cause of death in cases like the one described. By about 5 to 10 days, the myocardium becomes tan or yellow from infiltration of infarcted tissue with inflammatory cells. Microscopy will show necrosis of myocytes and macrophages performing phagocytosis. Granulation tissue appears at the margins of the infarcted area around days 7 to 10.