Cardiac Flashcards

1
Q

Concentric vs eccentric hypertrophy

A

Con-seen with LVH and HTN from pressure overload

Eccentric-seen with volume overload

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2
Q

Diuretics can cause which electrolyte derangements?

A

Decreased K, Mg, NA.

Increased glucose, Urate, lipid, and possibly increased K

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3
Q

Probs with Calcium channel blockers:

A

Can cause heart block, myocardial depression, potentiation of NMB, peripheral edema and reflex tachycardia (nifedipine)

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4
Q

Pre-op assessment for HTN: Look for signs of CHF when you see HTN? What else to look for-labs, imaging, ECG?

A

Yes look for CHF signs.

BUN/Cr for rental involvement, Na/K for diuretic effects, ECG for LVH, CXR for cardiomegaly and pulmonary edema

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5
Q

Pre-medication in a pt with HTN:

BP neds?

A

Sedation and anxiolytics are often good

Acute control with beta blockers can be helpful

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6
Q

Where from baseline should BP be kept?

A

Within 20% unless it is markedly elevated

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7
Q

Anesthetic significance of HTN
CV, Renal, Neuro. Causes if HTN:
Hemodynamics profile-HTN-early va late and how it relates to CO and SVR

A

LVH, diastolic dysfunction, vasopressin sensitivity, increased risk of MI, CHF, Aortic dissection
Rental-overactivity of RASS system and nephropathy
Neuro-potential increase risk of stroke and right shift of auto regulation curve

Causes: essential, followed by renal, endocrine-hyperaldosteronidm, Cushing’s, costctstion, drug side effects (estrogen)

HTN early vs late-early increased CO with normal SVR. Later-increase in SVR without Co

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8
Q

Cancel the case at which BP? A line for everyone?

A

There is no absolute cutoff. Emergency-would proceed no matter what the BP. It depends on the cause of HTN, chronic ott, symptomatomogy, difficulty with it being controlled, co-existing disease and type of surgery to be performed.

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9
Q

Best induction technique for OT with HTN?

A

No absolute beat technique, but a good one would include avoiding precipitous drops in BP from a rapid induction, achieve a deep level of anesthesia prior to laryngoscopes and intubation, and avoid vasopressors

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10
Q

HTN with a full stomach?

A

HTN with full stomach-need to analyze which risk is greater-aspiration or BP lability on induction. Elective-wait, had to proceed and aspiration risk low-proceed with slow induction.
High aspiration risk-a line prior to induction, RSI with etomidate and sux

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11
Q

Difficult airway, full stomach, HTN

A

Airway first-needs to be spontaneously breathing:
Supplemental O2, sedation!!, a line, topicalize airway with aerosolized 2%lidocaine, topicalize nasal mucosa with lidocaine jelly and phenylephrine. Glossopharyngeal, superior laryngeal, trans trachea blocks

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12
Q

How to do a glossopharyngeal, superior laryngeal, and tramstracheal block:

A

Hug

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13
Q

Automatically use etomidate?

A

No. In the normal, euvokemic, or HTN patient, it is not as reliable in preventing HTN with laryngoscopy and intubation

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14
Q

Ketamine in HTN patients-is it contraindicated?

A

It may cause more tachycardia and HTN, but no contraindicated especially if it currently hypotension or hypovolemic. Catecholamine depletion would unmask the direct myocardial depressant effect of ketamine, but the patient would still be spontaneously breathing

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15
Q

After surgery, your hypertensive patient is HTN in PACU-what to do?

A

Make sure HTN is real

ABCs-hypoxia and hypercarbia can cause sympathetic stimulation

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16
Q

Limb leads:
Criteria of ischemia on EKG:
Oscillometry concept:

A

Limb-1,2,3
1 mm ST depression, or 2 mm ST elevation
Oscillometry-cuff inflates to Supra systolic pressures and deflated in increments. Arterial pulsations are measurable oscillations in pressure

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17
Q

How big should a BP cuff be?
Too small va too large
For every 1 cm the cuff is above the heart? Below?

A

Width of cuff should extend at least 1/2 of extremity’a length, and be 20-50% greater than the extremity’s diameter
Too small-overestimates. Too large-underestimates
Above-subtract 0.7
Below-add 0.7

18
Q

Dampening coefficient:
What is overdamped?
What is underdamped?
How to stop these deviations?

A

Coefficient-tendencybfor fluid only the system to extinguish motion.
Over-flattened waveform-lower DBP. Kink, clot, loose connection, large air bubble.
Under-exaggerated waveform, overestimation of BP.
Stiff tubing, small mass if fluid (short length), fee stopcocks

19
Q

Risks of a line:

A

Hematoma, thrombosis, infection (femoral), nerve injury

20
Q

Central venous catheter-what is the point?
Draw CVP wAveform
Explain each

A

Monitor right heart filling pressures and waveforms
A-atrial contract in, c-ventricular systole (tricuspid calve bulges into atrium), v-venous return, x descent-downward displacement ofnventricle during systole. Y descent-atrial emptying with opening of tricuspid valve.

21
Q

Cannon a wave-

A

Tricuspid stenosis, RVH, AV block, LV noncompliance.

22
Q

Giant CV waves-

A

tricuspid regurgitation or right papillary muscle ischemia.

23
Q

6 indications for CVP. Does pressure equal volume?
When is CVP unreliable to measure left sided filling volumes?
CVP is properly placed where-

A
  1. Monitoring CVP
  2. Fluid resuscitation
  3. Infusion of drugs and hyper alimentation
  4. Insertion of pacing leads
  5. Venous access in patients with poor peripheral veins
  6. Aspiration of air with Air emboli

Pressure doesn’t always equal volume because the 2 are related by compliance. Compliance= volume/pressure

Unreliable with pulmonary hypertension, not just because if pulmonary arterial hypertrophy, but also RV non compliance

Properly placed at junction of SVC and RA
No in left due to thoracic duct and higher apex of lung

24
Q

PAC-directions and indications

A

Goes from RA-RV-PA-pulmonary arterial where it wedges
Indications-when it is important to monitor any or All of 4 variables:
1. Filling pressures-pulmonary edema, volume status (large fluid shifts), conditions such as tamponade and primary pulmonary HTN
2. CO-thermodikution
3. Calculated hemodynamics indices-SV, CI, SVR, PVR
4. MVO2

Volume status-things May elevate CVP without elevating PCWP (right sided heart stuff) 
Distinguishing cardiac (high PCWP) be non cardiac pulmonary edema
25
Q

PCWP measures what-

Elevations in CVP, PAD, and PCWP worh equalization of filling pressures is consistent with-

A

left atrial pressure

Consistent with cardiac tamponade (not diagnostic though)

26
Q

What are some conditions that elevate both the PCWP and CVP?

A

Conditions to elevate both the PCWP and CVP include mitral valve disease and left ventricular noncompliance. This makes the PCWP less of an accurate indicator of left ventricular filling pressures

27
Q

What is the normal cardiac index?

A

2.8-4.2

CO/BSA

28
Q

What is the formula for SVR? What are the normal values for SVR?

A

80 (MAP-CVP) /CO

1200-1500 dyne/sex/cm

29
Q

What is the formula for PVR? What are the normal values?

A

80 (PA-PCWP) / CO)

100-300

30
Q

What is the formula for stroke volume? What is the normal value?

A

CO x 1000/ HR

Normal is 60-90 mL/beat

31
Q

What is the formula for stroke index? What are the normal values?

A

SV / BSA

30-65 mL/beat/m^2

32
Q

What is the formula for LV stroke work index?

A

0.0136 (MAP - PCWP) x SI

Normal 45-60

33
Q

What is the formula for RV stroke work index? What are the normal values

A

0.0136 (PA - CVP) x SI

34
Q

Drawl the pulmonary artery catheter waveform

A

Okay. Look at pic

35
Q

What is a normal CVP pressure? What is a normal PCWP pressure?

A

CVP 0-8

PCWP 5-15

36
Q

What are two things that can falsely elevate thermodilution CO readings?

A

Low injectate volumes and warm injectate temperatures falsely Elevate thermodilution CO readings.

37
Q

What are some limitations of the pulmonary artery catheter?

A
  1. Over inflation of the balloon can cause it to herniate over the catheter tip or driving into the vessel wall artifactual increasing pressure.
  2. Structural physiologic conditions that can impair the ability of proximal pressure to reflect downstream pressures for example tricuspid stenosis, pulmonary HTN, tumors.
  3. Incorrect calculations of cardiac output due to incorrect thermodilution
  4. Elevations in mixed venous O2. From other situations. The most common cause of increased mixed Venus 02 is in advertent wedge pulmonary artery catheter causing sampling of pulmonary capillary blood.

Basically the decision to place a pulmonary artery catheter should be used in patients undergoing major surgeries who have instability hemodynamically in with a user who is familiar with its insertion.

38
Q

What are some complications of placing a pulmonary artery catheter?

A

Arterial puncture such as the carotid or subclavian artery. Hematoma, mediastinal/plural effusion, pneumothorax, nerve injury causing Horner syndrome, emboli, cardiac perforation, dysrhythmias,

39
Q

What are some complications of the pulmonary artery catheter just being there?

A

Thrombosis, thromboembolism. Pulmonary artery rupture pulmonary infarction, sepsis, endocarditis, dysrhythmias, hypotension, due to balloon inflation causing occlusion of PA flow.

40
Q

When it comes to placement of a pulmonary artery catheter, what are risks for rupture? What is a hallmark of this rupture?

A

Risks for rupture-Elderly, anticoagulation, overinflation. hallmark is hemoptysis