Cardiac Flashcards

1
Q

What is the 5 finger method for cardiac exam?

A
history
physical
ecg
x-ray
lab tests
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2
Q

What are the aspects of the cardiac exam?

A
• Inspection
• Jugular Venous Pressure (JVP)
• Precordial Palpation
• Percussion
• Auscultation-Heart Sounds
• Grading System of Murmurs
• ECG • Echocardiogram • X-rays • Lab tests – Pressures –
Hemodynamic measurement
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3
Q

What are some cardiac Non specific history elements you should look out for?

A

Fatigue, dyspnea, chest pain, palpations, syncope –

non specific

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4
Q

What aspects of history MUST you consider during a cardiac diagnosis?

A
  1. Underlying etiology – hypertensive, ischemic,
    congenital, infections
  2. Anatomic abnormalities
    - which chamber involved - which valve is affected - is pericardium involved - has there been a MI
  3. The physiologic disturbance – is arrhythmia present – is
    CHF present
  4. FAMILY HISTORY: hypertrophic
    cardiomyopathy, Marfan ’s syndrome, prolonged QT
    syndrome
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5
Q

What is the proper sequence of a cardiac function exam? What should the Pt be wearing ;) ? How should they be positioned?

A
  1. Inspection 2. Palpation 3. Percussion 4. Auscultation

Quiet room
Gown patient (do not listen through clothing)
Sitting, supine, left lateral decubitus, leaning forward, standing

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6
Q

What physical exam aspects are important for cardiac exam that are not on the chest? (face, nails, body, etc)

A
Face – acromegalic, cushnoid, Down’s syndrome, hyperthyroid, myxedema 
Jaundice – yellow 
Cyanosis – blue 
Pallor – pale, anemia, shock 
Nails – clubbing, hemorrhages 
Body habitus – tall, short 
Hydration – Blood pressure, weight 
Temperature
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7
Q
What is:
Barrel Chest
Pectus Carinatum
Pectus Excavatum
and what are they indicative of
A

Barrel Chest: COPD, increased A-P diameter
Pectus Carinatum: central protrusion
Pectus Excavatum: central depression

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8
Q

What are the S1 and S2 sounds? When my S2 split?

A
S1 • MV closure 1st component
• TV closure 2nd component
• Beginning of systole
• Loudest at apex 
S2 • 
Aortic valve closure 1st component
Pulmonic valve closure  2nd component • Loudest at the base • End of systole
S2 could split during inspiration due to increased venous return
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9
Q

Where do you feel the PMI ( Point of Maximal Impulse) or apical impulse?

A

– Supine or left lateral decubitus
– Normal: 4th-5th intercostal space at the Mid-clavicular line
-Feeling left ventricle, tapered inferior tip = cardiac apex

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10
Q

What do Jugular Vein visibility given you an indication about? What is JVP? What is a normal JVP?

A

Jugular Veins reflect the activity of the right side of the
heart
Level of JVP visibility gives an indication of the RAP
Internal jugular (IJ) is better than external jugular (EJ)

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11
Q

What is JVP? What is a normal JVP? What does an elevated JVP symbolize?

A

Jugular Venous Pulse (JVP
Normal: 0-9

Increase JVP in:
– SVC obstruction
– Severe heart failure
– Constrictive pericarditis, cardiac tamponade, RV infarction
– Restrictive cardiomyopathy
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12
Q

When do you see a giant A wave?

A
  1. Obstruction between RA and RV (i.e. T.S., Right atrial
    myxoma)
  2. Increased pressure in RV (i.e. P.S.)
  3. Pulmonary hypertension
  4. Recurrent pulmonary emboli 5. A-V dissociation (complete heart block, V.T.) (cannon ‘a’
    waves) . RA contracts against the closed TV.
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13
Q

What is the C wave indicative of?

A

Backward push by closure of TV during isovolumetric

systole and by impact of carotid artery adjacent to the JV.

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14
Q

What is the ‘X wave’? What is a steep X descent indicative of?

A

X: Passive atrial filling and atrial relaxation. Blood flows
into the RA from the cava and closure of TV
Steep X descent in cardiac tamponade and constrictive
pericarditis

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15
Q

When is the V wave? When is it prominent/what causes this?

A

Atrial Filling
Cause: – Increasing volume and pressure in RA when TV closed

Prominent V wave in TR and pulmonary hypertension

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16
Q

What is the Y slope and what does it mean when it is deep? or slow?

A

– Open TV and rapid RV filling in RV diastole
– Deep Y descent in severe TR
– A slow Y descent suggests obstruction to RV filling (i.e. TS or RA myxoma)

17
Q

What is an abnormal S3 sound due to?

A

Due to high pressures and abrupt deceleration of inflow across the mitral valve at the end of the rapid filling phase. Physiologic in children/young adults, pathologic >40 y/o

18
Q

What is an abnormal S4 sound due to?

A

Atrial gallop from forceful contraction of atria against a stiffened (low compliant) ventricle. Can be normal in trained athletes.

19
Q

Describe auscultation listening posts (Where do you here what valves?)

A

Aortic – Right 2nd intercostal space at sternal border
Pulmonary – Left 2nd intercostal space at sternal border
Tricuspid – Left 4th intercostal space at sternal border
Mitral – Left 5th intercostal space at mid-clavicular line

20
Q

What is the murmurs grading system? (1-6)

A

1 barely audible
2 soft, but easily heard
3 loud without thrill
4 loud with thrill
5loud with minimal contact btwn steth and chest with thrill
6 LOUD can be heard w/o stethoscope - thrill

21
Q

How to grade pulses? 1-4? What does R/R/A stand for

A
O = absent 
1 = barely palpable 
2 = average intensity 
3 = strong 4 = bounding

R/R/A =
Rate /Thythm/Amplitude

22
Q

Describe capillary refill

A

Assesses digital perfusion • Pt’s hands at heart level,
fingers pointing up. Doc
presses on nail bed till it
turns pale, then lets go.
Normal refill time <2sec. • Also check skin color and
turgor if possible

23
Q

How do you grade edema? (1-4) Where do you grade it?

A

Dorsum of foot, behind m. malleolus, anterior tibia
0 Absent
1+ Barely detectable, nonpitting (2mm)
2+ Slight indentation (4mm); 10-15 sec
3+ Deeper indentation (6mm); can be >1 min
4+ Very marked indentation (8mm); 2-5 min

24
Q

What is a PMI of >2.5 cm indicative of?

A

left ventricular hypertrophy

(LVH) from hypertension or aortic stenosis.

25
Q

Where might a PMI be palpable for COPD patients?

A

Chronic obstructive pulmonary disease (COPD), the most prominent palpable impulse or PMI may be in the xiphoid or epigastric area due to right
ventricular hypertrophy.

26
Q

What does displacement of the PMI lateral to midclavicular line happen?

A

LVH
Ventricular dilation from MI
Heart failure

27
Q

Split S2 when does it happen?

A

Wide physiologic splitting: during inspiration, normal caused by delayed closure of pulmonic valve
FIXED: does not vary with respiration, prolonged RV systole, ASD or RV failure
PARADOXICAL: Appears on expiration, disappears on inspiration, caused by L bundle branch block

28
Q

What are the Diastolic murmers?

A

P-MAT

• Aortic Insufficiency • Pulmonic Regurgitation • Mitral Stenosis • Tricuspid Stenosis

29
Q

What are the systolic murmurs?

A

• Aortic Stenosis • Aortic Sclerosis • Innocent (benign) murmur • Hypertrophic Cardiomyopathy • Ventral Septal Defect • Tricuspid Regurgitation • Mitral Valve Prolapse • Mitral Insufficiency

30
Q

When do you use the diaphragm vs bell?

A

Diaphragm: High pitched sounds (i.e. – S1, S2, AR, MR,
Friction Rubs)
Bell: Low pitched sounds (i.e. – S3, S4, MS, carotid
bruit)

31
Q

What does the Allen test evaluate? What does a negative allen test indicate?

A

Fxn of radial and ulnar arteries

Indication: Lack of dual blood supply to the hand (a negative indication for radial catheterization)