Cardiac Flashcards
optimal way to obtain hemodynamics/ for vessels
angio
causes of AS
65: degeneration and sclerosis of valve
lab findings of AS
ECG: LVH, Increased QRS or just R amplitute
CXR: LVH, Ca deposit
Echo: immobile valves LVH
Findings of AS
- hyperdynamic LV
- soft/normal S1
- absent A2
- paradoxical splitting of the S2
- Ejection click
- prominent S4
- systolic murmur
st segment depression
myocardial hypoxia
indications for intervention of AS
sx: angina, syncope, dyspenia, chf
EF<50%
CAD with moderate AS
sever/critical AS
HCM
hypertrophic cardiomyopathy :subvalvular as
HCM
no calcification of av, murmur louder standing or valsalvaing , ejection sound uncommon, similiar sx to AS
tx of HCM
ca chan blocker, beta blocker
acute AR etiologies
aortic dissection, iatrogenic-cath/surgical, endocarditis
chronic AR etiologies
- rheumatic valve dz (~30%), bicuspid aortic valve, dilated aortic root, bacterial endocarditis, senile degeneration, connective tissue dz
RA, Marfans, ehlers-danlos may cause
AR
narrow PP
AS
wide PP (low diastolic)
AR
chronic AR results in
LV overload, dilation, hypertrophy, HF
acute AR results in
pulmonary edema
PE findings of AR
high pitched diastolic decescendo murmur at aortic are and left sternal border
austin flint murmur
low pitch diastolic murmur at the apex sounds like a diastolic mitral stenosis murmur
wide PP
increased systolic and decreased diastolic –>water hammer or corrigan pulse
Acute MR
papillary m. necrosis and rupture, endocarditis
marfans, graves dz, muscular dyst. a/w
barlows syndrome –>MR
Acute MR
papillary m. necrosis and rupture, endocarditis
marfans, graves dz, muscular dyst. a/w
barlows syndrome –>MR
MR RV failure signs
JVD, Hepatomegaly, edema
MR pulmonary HTN
s4 gallops, loud p2, rv heave and lift
most common cause of MS
rheumatic heart dz
emboli to the brain a/w
MS
most common cause of MS
rheumatic heart dz
anticoagulant
bleeding and thromboembolism
most common cause of hf
CAD
used to differentiate pulmonary from cardiac dz in pt w dyspnea and/or an inconclusive PE
BNP level
most common cause of hf
CAD
used to differentiate pulmonary from cardiac dz in pt w dyspnea and/or an inconclusive PE
BNP level
recommended initial therapy for ALL pt with HF
ACE inh., digoxin, diuretics
diastolic dysfunction is more common in
elderly female with HTN and DM
side effect of ACE
cough and renal dysfunction (okay)
ARBs (angiotensin-receptor blockers)
use if ACE not tolerated , assess bp, renal fxn, electrolytes regularly
digoxin
pt w a fib
indication of sporonolactone
pt with rest dyspnea within past 6 months, post MI with systolic dysfxn
digoxin
pt w a fib
PE sx
dyspnea, productive cough, diaphoresis
management of PE
O2, morphine, diuretics, nitrates
most common cause of HF is
left ventricular systolic dysfxn
side effect of statins
myopathy (check CK level), elevated LFTs