Capnography Flashcards

1
Q

PACO2

A

concentration of CO2 in the alveoli

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2
Q

PaCO2

A

concentration of CO2 in the arteries

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3
Q

EtCO2

A

concentration of CO2 in expired gas

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4
Q

how much higher is PaCO2 than EtCO2 normally?

A

3-5mmHg

dilutes as it travels to the capnograph

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5
Q

Do PaCO2 and EtCO2 normally correlate?

A

yes if PaCO2 increases so does EtCO2

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6
Q

When is the exception to PaCO2 and EtCO2 correlation?

A

pulmonary embolism
PaCO2 increase
EtCO2 decrease

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7
Q

what are the effects of hypercarbia?

A
  1. repiratory acidosis
  2. central (pulmonary) vasoconstriction
  3. peripheral and cerebral vasodilation
  4. sympathetic response/catecholamine release
  5. CO2 narcosis
  6. possible death
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8
Q

respiratory acidosis

A

pH decrease b/c H increases

catecholamines (vaspressors) don’t work as well

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9
Q

central (pulmonary) vasoconstriction

A

increased PVR

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10
Q

when should hypercarbia be avoided specifically?

A

neurosurgery

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11
Q

CO2 narcosis

A

CO2 acts as a sedative if it reaches 70mmHg

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12
Q

What PaCO2 is equal to 1 MAC?

A

200mmHg

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13
Q

What PaCO2 could cause death?

A

> 120mmHg

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14
Q

What are the effects of hypocarbia

A
  1. respiratory alkalosis
  2. central (pulmonary) vasodilation
  3. peripheral and cerebral vasoconstriction
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15
Q

what is the gold standard for confirmation of ETT placement?

A

etCO2

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16
Q

how fast does PaCO2 rise during apnea?

A

first min= 6mmHg

each min after 3-4mmHg

17
Q

Phase 0

A

inspiration

CO2 = 0

18
Q

Phase I

A

beginning expiration
CO2 = 0
represents anatomic deadspace

19
Q

Phase II

A

portion of expiration where CO2 is first seen

20
Q

Phase III

A

CO2 plateau

21
Q

Phase IV

A

end of plateau

22
Q

curare cleft

A

when there is a dip in the plateau

pt is trying to breath over the ventilator

23
Q

what else can cause a curare cleft?

A

surgeon is pushing on the chest

24
Q

what are the two options to treat a curare cleft

A
  1. suppress their drive to breath

2. turn off the vent and let the patient breathe

25
how do you suppress a patients drive to breathe?
propofol narcotic/ paralytic increase minute ventilation (lower etCO2)
26
what is the first thing you do when the patient is "bucking" on the vent
TURN THE VENT OFF
27
COPD/Emphysema etCO2 waveform
upsloping waveform | prolonged exhalation times
28
esophageal intubation etCO2 waveform
small waves then nothing after a few breaths
29
hypocapnea waveform causes
hyperventilation hypotension/low Q loose circuit connection
30
cardiogenic oscillations
heart contractions that displace air from alveoli
31
when do you normally see cardiogenic oscillations?
low RR | end of expiration
32
what are the three things the capnograph measures?
etCO2 etvolatile agent RR
33
what if the EtCO2 wave never reaches 0?
the CO2 absorbent is exhausted
34
at what flow is the CO2 absorber not necessary?
5L/min
35
when does the etCO2 wave look like a chair?
loose connection or circuit leak
36
At what stage to patients usually go through irregular breathing?
Stage II
37
when could exhaling during inspiration happen?
mapleson circuit or faulty expiratory valve
38
When would you see decreased CO2 at the end of the plateau phase?
emergence
39
What is the value of capnography during sedation?
``` RR detect apnea (before hypoxic) ```