Burns Flashcards
introduction to burn injuries
- 1.25-2 million Americans treated for burns annually
- 45,000 require hospitalizations
- 90% of burns can be prevented
- 3-5% considered life threatening
- 2nd leading cause of death for children <12
- half of all tap water burns occur to children <5
greatest risk for burn injuries
- very young -> greater BSA/weight ratio and thinner skin
- very old -> thinner and doesn’t heal as well
- infirm
- firefighters
- metal smelters
- chemical workers
- drugs and alcohol play major role
- uncle rob
reduction in burn injuries
- improved building codes
- safer construction techniques
- sprinkler systems
- use of smoke detectors
- educational campaigns aimed primarily at school children
layers of skin
- epidermis - dead
- dermis - nerves, hair
- subcutaneous
- underlying structures:
- fascia
- nerves
- tendons
- ligaments
- muscles
- organs
functions of the skin
- protection from infection
- sensory organ- temperature, touch, pain
- controls loss and movement of fluids
- temperature regulation
- insulation from trauma
- flexible to accommodate free body movement
emergent phase (stage 1)
- pain response
- catecholamine release:
- tachycardia
- tachypnea
- mild hypertension
- mild anxiety
fluid shift phase (stage 2)
- length 18-24 hours
- begins after emergent phase- reaches peak in 6-8 hours
- damaged cells initiate inflammatory response:
- vasodilation:
- increased capillary permeability
- intravascular hypovolemia
- extravascular edema
- burns over 30% BSA present with system immune response *****
hypermetabolic phase (stage 3)
- lasts for days to weeks
- large increase in the body’s need for nutrients as it repairs itself
- fluid and electrolytes begin to move back into the vasculature
- influx of fluid within vascular space causes the GFR to increase, leading to diuresis
- fluid shifts may lead to hypernatremia and hypokalemia
- cardiac workload and O2 consumption increase
resolution phase (stage 4)
- scar formation
- general rehabilitation and progression to normal function
stages of pathophysiology of burns
- emergent phase
- fluid shift phase
- hypermetabolic phase
- resolution phase
types of burns
- thermal
- electrical
- chemical
- radiation
thermal burns
- heat changes the molecular structure of tissue -> denatures proteins
- extent of burn damage depends on:
- temperature of agent
- concentration of heat
- duration of contact
jacksons theory of thermal wounds: zone of coagulation
- area in a burn nearest the heat source that suffers the most damage as evidenced by clotted blood and thrombosed blood vessels
- center of burn
jacksons theory of thermal wounds: zone of stasis
- area surrounding zone of coagulation characterized by decreased blood flow
- middle portion
jacksons theory of thermal wounds: zone of hyperemia
- peripheral area around burn that has an increased blood flow
- outermost area
burn infection
-people dont come in because they think the skin is intact but really a whole layer is burned off and an infection has already developed
electrical burns
- greatest heat occurs at the points of resistance:
- entrance and exit wounds
- dry skin = greater resistance
- wet skin = less resistance
- longer the contact, the greater the potential of injury -> increased damage inside body
- smaller the point of contact, the more concentrated the energy, the greater the injury
electrical current flow
- tissue of less resistance - blood vessels and nerve
- tissue of greater resistance - muscle and bone
- results in:
- serious vascular and nervous injury
- immobilization of muscles
- flash burns
chemical burns
- destroy tissues
- acid
- alkalis
chemical burns: acids
- form a thick, insoluble mass where they contact tissue
- coagulation necrosis - limits burn damage
chemical burns: alkalis
- destroy cell membrane through liquefaction necrosis
- deeper tissue penetration and deeper burns
radiation
- transmission of energy
- nuclear energy
- ultraviolet light
- visible light
- heat
- sound
- x-rays
radioactive substance
- emits ionizing radiation
- radionuclide or radioisotope
alpha particles
- slow moving
- low energy
- stopped by clothing and paper
- penetrate a few cell layers on skin
- minor external hazard
- HARMFUL if ingested
beta particles
- smaller than alpha
- higher energy than alpha
- stopped by aluminum or similar materials
- less local damage than alpha
- HARMFUL if inhaled or ingested
gamma rays
- highly energized
- penetrate deeper than alpha or beta
- EXTREMELY DANGEROUS
- penetrate thick shielding
- pass entirely through clothing and body- extensive cell damage
- indirect damage- cause internal tissue to emit alpha and beta particles
- LEAD SHIELDING
neutrons
- more penetration than other radiation
- 3-10 times greater penetration than gamma
- less internal hazard when ingested than alpha or beta
- direct tissue damage
- only present in nuclear reactor core