Brain Haemorrhages and Fractures Flashcards

1
Q

what does TBI stand for

A

traumatic brain injury

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2
Q

what is a common cause of brain haemorrhage

A

Trauma - Head Injury - RTA = one of the most severe causes, assault, accident trips and falls, sports-related injuries, industrial accidents

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3
Q

facts and figures involving head injuries

A

each year, around 700,000 people attend A&E with a head injury
more than 80% only have a minor injury
around 3/4 of those with head injuries are male
nearly half the people with head injuries are children

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4
Q

how has risk of head injuries been reduced

A

legislative measures - seatbelt in cars, anti-drink drive campaigns, bicycle/sports helmets
reducing hazards in the home that may cause a fall
childproofing the home
using correct safety equipment for work and DIY

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5
Q

what is a ‘head injury’ defined by the NICE guidelines

A

‘any trauma or the head or superficial injuries to the face’ caused when an external mechanical force causes an insult to the brain

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6
Q

what 2 categories can brain injuries fall into - explain

A

primary - occurring at time of injury - cannot be altered/reversed
secondary - occurring later - often amenable to prevention/reversal

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7
Q

how to decrease a secondary brain injury

A

early diagnosis and rapid appropriate treatment

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8
Q

causes of secondary brain injury - 5 points

A
hypoxia 
hypovolaemia 
intracranial haematoma causing pressure effects
cerebral oedema 
infection
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9
Q

signs and symptoms of head injury - 13 points

A
Headache
Reduced or loss of consciousness
Acute neurological deficit (motor/sensory)
Reduced GCS
Confusion
Agitation
Decreased alertness
Nausea/Vomiting
Seizures
Diplopia
Seizures
Rhinorrhoea
Otorrhoea
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10
Q

describe the Glasgow coma scale

A

a practical method for assessment of impairment of conscious level in response to defined stimuli
1974
easily repeatable measure of conscious state and neurological ability

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11
Q

what is the primary investigation of choice for head injury and what do the guidelines advise

A

CT

advises who gets a CT scan and the time scale

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12
Q

which modality is not the imaging modality of choice and why

A

MRI
accessibility - not for the patient in the acute scenario, contraindicated due to its magnetic field
inability to screen the unconscious patient - requires a number of staff who are unaware of the magnetic field hazard
staff would have to go through safety screening - time consuming
CT provides similar information for most clinical purposes

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13
Q

what is the criteria for an ‘immediate’ scan and a ‘delayed’ scan

A
immediate = < 1hr
delayed = within 8 hrs
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14
Q

when else should a CT scan be carried out within 8 hours

A

patients on warfarin presenting with a head injury in the absence of other indications
or immediately if 8 hrs has already elapsed

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15
Q

what are the 6 types of haemorrhage

A

extradural haemorrhage - trauma
subdural haemorrhage - trauma
contusion - trauma
diffuse axonal injury- trauma, complex (MRI)
subarachnoid haemorrhage - trauma, spontaneous
intracerebral haemorrhage - trauma, spontaneous

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16
Q

describe extradural haemorrhage

A

occurs between dura mater and skull
requires trauma to draw dura from bone, skull in 80% of cases
stripping of dura from bone = laceration of the arteries - usually the middle meningeal artery
arterial bleed = confined by limits of dura where its torn away or by the sutures

17
Q

radiographic appearance of extradural haemorrhage

A

typically bi-convex/lentiform hyperdensity

18
Q

who is mainly affected?

how can it present

A

typically affects young patients involved in a clearly defined blow (assault/fall/sports-injury etc)
newborns - due to abnormal/difficult labour or forceps delivery

severe headache - due to ripping of the dura from skull
?LOC - depends on force of impact
classic lucid interval after concussive force of blow
patient regains consciousness but the EDH continues to expand = mass effect = increased intracranial pressure = decreased level of consciousness and possible coning

19
Q

describe subdural haemorrhage

A

severe or minor head injury causes rapid acceleration/deceleration of the brain tissue
bridging veins tear = low pressure venous bleeding
large haematomas can form before clinical signs appear
associated with cerebral atrophy - common in elderly and alcoholics
bridging veins under greater tension as the brain gradually shrinks from skull
easily ruptured - with only minor trauma

20
Q

radiographic appearance of subdural haemorrhage

A

between dura mater and arachnoid mater
crescent, concave, sickle shape
time of presentation since injury is important because radiographic density of blood alters with age

ACUTE - hyper dense
SUBACUTE - isotense (4-21 days after)
CHRONIC - hypodense (21 days or more after)

21
Q

who does it affect?

A

> 60yrs, more common in men than women
puts with clotting disorders including alcoholics
associated with NAI - shaken baby syndrome

22
Q

how is acute usually presented and chronic usually presented

A

ACUTE
usually younger patients/alcoholics due to the way they present
rates of mortality and morbidity = high
emergency management = critical
CHRONIC
usually seen in elderly
often slow deterioration seen in patient as haematoma expands
gradual onset of confusion, decreased level of consciousness, headache, difficulty with gait or balance, memory loss

23
Q

describe contusion

A

bruise on the brain
often occurs in frontal and temporal regions due to brain colliding with the brain protuberances within the skull
blood vessels are damaged and haemorrhage due to direct trauma

24
Q

radiographic appearance of contusion

A

often multiple small haemorrhages at grey/white matter interface
delayed scan demonstrates oedema and atrophy

25
Q

how is contusion usually presented and who’s affected

A

associated with young males due to mechanism
can be minor with few symptoms and little/no damage to brain
severe contusions = extended period of unconsciousness
patients aware confused, tired, agitated, emotional
other symptoms = memory loss, attention problems, difficulty with motor coordination, loss of ability to understand or express speech
more severe contusions = increased intracranial pressure, coning, possibly coma and death

26
Q

what are the pharmocological options for brain haemorrhage

A

painkillers
corticosteroids/diuretics to reduce swelling
anticonvulsants - control seizures
ACE inhibitors to lower blood pressure

27
Q

what are the surgical intervention for brain haemorrhage

A

craniotomy and evacuation of haemorrhage to alleviate swelling
neurological clipping of aneurysm
endovascular coiling of aneurysm

28
Q

conservative options for brain haemorrhage

A

close clinical observation

follow up CT scans to assess - decline in neurological status, extent of clot evacuation, anyone haematoma formation

29
Q

what is the prognosis for a brain haemorrhage

A

depends on size and location of haemorrhage

initial presentation (GCS) significantly correlates to outcome of patients who survive

30
Q

describe skull #s

A

not always seen but can see fluid in sinuses and pneumocephalus in the skull vault, intracranial air - leaked in via #

31
Q

describe base of skull #s

A

s diagnosed clinically from these signs

may involve the foramen though which cranial nerves pass - dysfunction in these may lead to loss of smell, hearing, imbalance, weakness in the facial muscles

may also tear the dura resulting in a leakage of CSF into the ear (otorrhea) or nose (rhiorrhea)
or cause blood to leak behind the ear (Battle’s sign) or around the eyes (Racoon eyes)

32
Q

treatment for skull #s

A

patient given antibiotic cover to prevent infection/meningitis and surgical intervention may be required