Bradyarrhythmias & life threatening causes of syncope Flashcards
Serious signs and symptoms of bradydysrhythmias
CHAPS
Chest pain, SOB
AMS
Giddiness
Hypotension (SBP < 90)
Syncope
*Stokes-Adams attack
What is bradycardia
Heart rate < 60 bpm
Approach to bradycardia (<60bpm)
Bradycardia identified
Ask yourself: where does the problem lie?
- Absent P waves: think SA node
- P waves present: think sinus brady or AV nodal block
LOOK AT LEAD II
Causes of bradycardia:
Where does the problem lie?
- SA node
- AV node
- Slow conduction due to drugs and electrolytes
Types of AV blocks
1st degree AV block
2nd degree AV block (Mobitz 1: Wenkebach)
2nd degree AV block (Mobitz 2)
2nd degree high grade AV block
3rd degree AV block (complete heart block)
ECG features: 1st degree AV block
Prolonged PR interval > 0.2s (1 big square) and constant
Every P wave is associated with a QRS complex
P-P interval: regular
P-R interval: regular
R-R interval: regular
ECG features:
2nd degree AV block, Mobitz 1 (Wenckebach)
Progressive lengthening of PR interval followed by a dropped QRS (by right it’s p wave not conducted)
P-P interval: regular
P-R interval: irregular
*P-R longest before the dropped beat
R-R interval: irregular
ECG features:
2nd degree AV block, Mobitz 2
Dropped QRS every 3 or more P
- P QRS P QRS P DROP
P-P interval: regular
P-R interval: regular
R-R interval: depends on ratio
ECG features:
High grade block (2:1, 3:1)
Conducted beats every 2 or more beats
eg.
2:1 = 1 dropped QRS (aka 1 solo P wave) followed by 1 conducted P+QRS
3:1 = 2 dropped beats (aka 2 solo P waves) followed by 1 conducted P+QRS
P-P interval: regular
P-R interval: regular
R-R interval: depends on ratio
ECG features:
3rd degree AV block
Every beat is NOT conducted
AV dissociation (P and QRS. are independent rhythms)
P-P interval: regular
P-R interval: irregular
R-R interval: regular (escape rhythm)
Causes of AV block
- Ischemia: ACS
- Electrolytes:
- Hyper or hypoK+
- Hypomagnesium
- Hypo or hyperCal - Drugs:
- BB
- CCB
- Digoxin - Infection: Myocarditis
- Others
Management of bradycardia
- Identify rhythm
- Determine possible cause of heart block and treat reversible cause
- Look for serious signs and symptoms (CHAPS)
- CP, SOB
- SBP < 90
- AMS
- HF (pul edema)
- Shock - If present -> Unstable
Initiate treatment
1st line: IV atropine 0.6mg every 3-5mins
2nd line: IV dopamine or adrenaline
IV fluid resus
Transcutaneous pacing, then transvenous pacing
Permanent pacemaker
If stable
- BP > 90/60 AND NO symptoms
Monitor - no need intervention
If Mobitz 2 or 3rd degree block, close monitoring + prepare for pacing
ECG features: Sinus bradycardia
Any HR < 60bpm
Every P wave is associated with QRS complex
ECG features: Sinus node dysfunction aka sick sinus syndrome
- Absence of P waves
- Prolonged sinus pause / sinus arrest
- Tachy-brady syndrome (sinus arrest interspersed with A FIB)
What syndrome can be seen in SSS
Tachy-brady syndrome
- sinus arrest interspersed with A FIB
What is the definitive treatment for SSS
Permanent pacemaker for sinus pause
Anticoagulation
ECG features:
Hyperkalemia
A: Tall peaked T waves (Eiffel Tower)
B: Prolonged P-R
C: Flattened P wave
D: Widened QRS
E: Sine wave (serious)
HyperK+ management
Stop any K+ infusion if any
- IV calcium gluconate 10% 10mls over 10 mins (repeat multiple doses if sine waves)
- IV 50% dextrose 40mls + 10units of insulin
- Oral resonium 15g
- +/- Neb Salbutamol
- Urgent hemodialysis if got sine waves
How to tell QT prolongation?
If QT is > than 1/2 of RR interval, QT is likely to be prolonged
Implication of prolong QT syndrome
Sudden cardiac death from ventricular arrhythmia
Definitive treatment in familial QT syndrome
Automatic Implantable Cardioverter Defibrillator
ECG feature: Brugada syndrome
Cove shaped ST elevation in V1-2
Risk of Brugada syndrome
Sudden cardiac death from ventricular arrhythmia
Criteria for brugada syndrome
ECG pattern pattern +ve
AND
Syncope OR
Fam hx of SCD OR
Fam hx of PPM/AICD
Genetic testing
Definitive treatment in Brugada syndrome
Automatic Implantable Cardioverter Defibrillator
Massive PE management
Reperfusion
1. 1st line: IV thrombolytic rTPA 100mg over 2h
2. Percutaenous embolectomy
3. Open embolectomy
Bridging therapy
- ECMO
Syncope causes
Hyperkalemia
Brugada
QT prolongation
VFIB