Brachial Plexus Flashcards

1
Q

Most common traumatic brachial plexus injuries are:

A

supraclavicular injuries involving complete involvement of all roots (80%)

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2
Q

Speed of nerve regeneration is:

A

1mm per day.

Plexus recovery can take up to 3 years

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3
Q

Worst prognosis is with these injuries:

A
root avulsions (pre-ganglionic)
- not reparable
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4
Q

Best prognosis with these injuries

A

Infraclavicular plexus injuries

Upper plexus injuries (preserves hand function)

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5
Q

Signs of Pre-ganglionic Injury

A
  • Horner’s syndrome
  • Medial scapular winging (Long thoracic n)
  • flail arm
  • normal histamine test
  • elevated hemidiaphragm (phrenic)
  • Rhomboid paralysis (dorsal scapular n)
  • Cuff weakness (suprascapular n)
  • Latissimus weakness
  • EMG = no innervation to cervical paraspinals
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6
Q

Signs of post-ganglionic injury:

A

EMG with maintained innervation to cervical paraspinals

- abnormal histamine test… redness and wheal positive, NO FLARE

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7
Q

Most common brachial plexus obstetrical palsy:

A

Erb’s

  • C5, C6
  • has best prognosis
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8
Q

Causes of a Klumpke Palsy

A

C7-8 root involvement

  • hyperabduction injury
  • cervical rib
  • lung mets in lower deep cervical lymph nodes
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9
Q

Clinical features of a Klumpke:

A
  • claw hand
  • unopposed wrist extensors
  • loss of hand intrinsics therefore IP flexion and MP hyperextension
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10
Q

Essential muscles to check in brachial plexopathy:

A

serratus anterior

- rhomboids (if functioning, then likely a post-ganglioinc)

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11
Q

Define: SNAPs

A

sensory nerve action potentials

  • measured in NCS
  • preserved in lesions proximal to the DRG. Therefore distinguishes between pre and post-ganglionic
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12
Q

Best clinical sign of nerve regeneration?

A

advancing Tinel sign

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13
Q

Indication for immediate surgical exploration?

A

sharp, penetrating trauma

  • iatrogenic injuries
  • open injuries
  • progressive defects
  • expanding hematoma or vascular injury
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14
Q

IS direct nerve repair possible for plexus injuries?

A

typically not as these are avulsion injuries

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15
Q

Nerve grafting plexus injuries:

A
  • useful in traction injuries

- preferable to graft the upper and middle trunk

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16
Q

Neurotization for plexus injuries:

A
  • transfer a working but non-essential motor nerve

- use extra-plexal source of axons such as CN XI, intercostal nerves, contra-lateral C7, CN XII

17
Q

Oberlin Transfer:

A

ulnar nerve is used for upper trunk injury to gain biceps function

18
Q

Horner’s Syndrome

A

disruption of the sympathetic chain at C8 or T1 root level

- seen concomitantly with pre-ganglionic injuries at this level

19
Q

Phrenic Nerve

A

C3,4,5

20
Q

Neurotization to restore elbow flexion - what procedure?

A

ulnar nerve fascicle to biceps
AND
median nerve fascicle to the brachialis

21
Q

Best surgical management of late-presenting plexopathies? (liek 2 years out…)

A

free muscle transfers

- nerve transfers unlikely to be sufficient due to loss of neuromuscular endplates at that time frame

22
Q

Quadrilateral space syndrome

A

dominant shoulder in overhead throwing athlete

  • axillary nerve compression
  • atrophy of deltoid and teres minor
23
Q

Risks for obstetrical brachial plexopathy

A
  • LGA
  • multiparous
  • dystocia
  • forceps
  • breech
  • prolonged labor
24
Q

Upper extremity issues from obstetrical brachial plexopathy

A
  • glenoid dysplasia, retroversion, humeral head flattening, posterior humeral head subluxation
  • Elbow flexion contracture
25
Q

Poor prognosis for recovery of obstetical plexopathy?

A
  • lack of biceps function by 3 months

- preganglionic injuries

26
Q

Narakas Classification

A

I: Erb’s
II: Intermediate (C5-7)
III: total palsy without Horner’s
IV: total palsy with Horner’s

27
Q

Elbow contracture of <40 degrees, treatment

A

serial extension splinting
casting if >40 deg
if recalcitrant to treatment, use surgery

28
Q

How do you influence the natural history of glenoid dysplasia in obstetrical brachial plexopathy?

A
  • address GH internal rotation contracture wth latissimus and teres major transfer
  • ifdone at young age, significant remodeling potential exists
  • relative contraindications include severe dysplasia such as glenoid convexity or absence, humeral head flattening
29
Q

Waters Classification - significance

A

if Waters GH dysplasia types II or III, do latissimus/teres major transfer

if Waters IV or V, do humeral derotational osteotomy

30
Q

When do you do reconstruction for post-ganglionic injuries?

A

at 3-9 months of life, using neuroma excision and sural nerve grafting

31
Q

When do you reconstruct pre-ganglionic injuries?

A

at 3 months of life, using nerve transfers

32
Q

What’s the prognostic value of a Horner’s syndrome in context of brachial plexopathy?

A

poor prognosis.

<10% will regain function (al-Qattan 2000, Waters 1999)