Book1 Flashcards
perceptual organization
depth, form, motion, constancy
Binocular cues
Two eyes allow them to recieve visual cues from the environment.
binocular cues
depth, retinal disparity, covergence
retinal disparity
eyes are 2.5 inches apart
convergence
Gives humans a sense of depth based on how much the eyeball has turned
monocular cues
See things with one eye
Relative size
closer object appear bigger
Interposition (overlap)
Perception that one object is in front of another. An object that is in front is closer.
relative height
things higher in our field of vision look further away
shading and contours
using light and shadows to percieve form depth/contours-crater/moutain
motion parallax
relative motion, things farther move slower, things closer move faster
constancy
perception of object doesnt change despite image on retina changing, we are smarter, size/shape/color constancy
size constancy
if object is closer we dont perceive it as bigger just because its close, we know its the same size
shape constancy
a changing shape still maintains the same shape perception.
Color constancy
the ability to recognize colors despite changes in lighting
sensory adaptation
Ability of our senses to adapt and change their sensitivity stimuli
Weber’s law
JND= just noticeable difference
Linear relationship of jnd
Linear relationship between threshold and background intensity
Weber’s Law (math)
ΔI (JND)/I (initial intensity) = k (constant)
absolute threshold of sensation
differences based on the individual
Absolute threshold of sensation (graph)
May not hear all of the sounds a 100% of the times.
Absolute threshold is NO T equal to jnd?
Difference threshold- difference between the when the light got brighter
Factors that affect absolute threshold
(1) Expectations- eg. expecting a text
subliminal stimuli
stimuli below the absolute threshold of sensation
somatosensation
Receive info on intensity, timing, location
Somatosenation types
(1) Temperature = thermoreception
intensity
how quickly the neurons fire’
timing
Neurons encode 3 ways of timing:-
vestibular system
balance and orientation
inner ear=
semicircular canals (posterior, lateral, anterior)
endolymph
inner ear filled with endolymph to detect motion of the fluid with rotation
otolithic organs=
utricle and saccule,
Otolithic organs function
- detect linear acceleration
Calcium carbonate crystals
When the head moves:
What happens when the balance goes wrong?
(1) Dizziness
Dizziness
Endolymph doesn’t stop spinning the same time as we do, so it continues
Signal Detection Theory
how we make decision under conditions of uncertainty, discern between importnat stimuli and unimportant “noise”
SDT in psychology
ex. if given 2 lists in psych experiment and then person asks which words came from first list, they would be uncertain
SDT example
It’s foggy day & you have to decide when to start driving. How strong does a signal have to be for you to drive? Signal is present or absent (red). or traffic light in the fog and can’t tell
signal detection theory
-Hit, the subject responded affirmative when a signal was present,
type I error
false positive
type II error
false negative
d’
*dick is strong= strength
c’
C= strategy
Types of C’ strategy
(1) Conservative (2) Liberal
Conservative
always say no unless you are 100% sure the signal is present
Liberal
liberal strategy= always say yes
signal detection theory pt 2 graph
noise distribution and signal distribution
bottom-up processing
Stimulus influences –> what we perceive (our
top-down processing
use background knowledge –> influence perception, ex. where’s waldo
Gestalt Principles
tries to explain how we perceive things the way we do
Law of similarity
items similar to one another grouped together by brain.
Law of Pragnanz
reality is organized or reduced to the simplest form possible
law of proximity
objects near each other tend to be grouped together
law of continuity
lines are seen as following the smoothest path
Law of closure
objects grouped together are seen as a whole
Law of Symmetry (Gestalt)
the mind perceives objects as being symmetrical and forming around a
law of common fate
if half dots move up and half dots move down we perceive the dots as 2 distinct units
law of past experiences
visual stimuli are categorized according to past experience
context effects
context of how stimuli are presented influences how people perceive the stimuli
vision
conjunctiva= thin layer inside eyelid
Trasnmission
Electrical activation of one neuron by another
Processing
Nueral signal –> perception
Transduction
Energy transform from one form to anohter
Light pathways
Light enters –> pupil –> rods/ cones –> bipolar cell –> retinal ganglion cell –> optic nerve –> brain
Cones
Red + Green + Blue
Fovea
center of macular
phototransduction cascade
ROD ON —> ROD OFF
Bipolar cells
Found in the retina
Ganglion cells
Found on retina
Optic nerve
visual signals go from ganglion cells to optic nerve (They are just axons of the ganglion cells)
trichromatic theory of color
3 cones in eye, red, green, blue
opponent process theory
cones perceive 4 colors: red, green, blue, YELLOW
Retina
made of rods + cones
Phototransduction cascade
turn off retina
Light hits rods
light –> neural impulse
rods
Optic disks stacked on top of each other
Retinal
Protien inside rhodopsin
mechanism of vision
rhodopsin in the rods, has a cis-retinal molecule
Transducin
3 different parts - attached to the rhodopsin
What happens when rhodopsin changes shape?
rhodopsin changes shape –> transducin breaks from rhodopsin –> alpha subunit binds to another disk protein called phosphodiesterase (PDE)
PDE
Takes cGMP –> converts to GMP
Na+ channels on rods
Allow Na+ ions to ocme in
Na+ unbound of cGMP
less Na+ enters
What happens to bipolar cells when light enters?
When light hits ROD –> turned off–> on center bipolar cells active –> off center bipolar cells inactive
On center bipolar cells
ON CENTER = TURN ON WITH LIGHT
Rod turne don
ROD on –> on center bipolar inactive –> off center active –> activate off center retinal ganglion cell –> send signal to optic nerve
Photopsin
Rhodopsin in cones
Photoreceptor
Light –> neural impulse
Light hits rhodopsin
trigger phototransduction cascade
Diff b/w rods and cones
(1) More rods
visual field
L field of vision hits the R side of both eyes (so the nasal side of L eye and the temporal side of R eye)
feature detection
color= trichromatic theory
audition (sound)
pressure wave stimuli and hair cell
place theory
perception of sound depends on where each freq produces vibrations along the basilar membrane
hearing mechanism
hair bundle in upper membrane of organ of corti, has kinocilium (stereocilia) connected by tip links which have K+ channels
basilar tuning
varying hair cells in cochlea
primary auditory cortex
in the Temporal lobe
cochlear implants
restores hearing from “nerve deafness”= sensorineural hearing loss
sensory adaptation
change over time of receptor to a constant stimulus
sensory homunculus
different areas of body have signals that go to different parts of the strip
proprioception
spindle receptor/sensor located in muscles
pain/temp
pain= nociception
gate control theory
NAME?
pheromones
chemical signal to indicate an innate response to member of same species or another species
olfaction
sends info to olfactory bulb
mechanism of olfaction
olfactory molecule binds to GPCR receptor on olfactory receptor cell then G protein dissociates causeing binding to ion channel which allows outside ions to come in triggering AP which goes to cribiform plate to glomerulus to mitral cell which then synapses on the brain
labeled-line theory of olfaction
each receptor responds to specific stimuli and is directly linked to the brain in that way
vibrational theory of olfaction
vibrational frequency of a molecule gives that molecule its specific odor profile
steric theory/shape theory of olfaction
odors fit into receptors similar to lock-and-key
anosmia
inability to perceive odor
taste
bitter, salty, sweet, sour, umami
labeled lines model of taste
each taste bud receptor has 5 axons which all send each dif taste to different parts of the gustatory cortex and they remain separate in the brain
sweet/umami/bitter
GPCR receptors
sour/salty
ion channels
what if we put salty receptor inside a sweet cell?
still interprets it as a sweet signal because the cell will be depolarized by the salty ion channel opening but then it fires to the sweet glomerulus
consciousness
awareness of self and environment, states range from alertness to sleep
alertness
you’re awake, focus attention
daydreaming
feel more relaxed adn less focused, light-meditation
drowsiness
just before falling asleep, self-induced deep meditation
sleep
not aware of self of world around you
sleep stages
4 main stages
N1
first non-rapid eye movement sleep stage
N2
deeper stage of sleep, harder to awaken
N3
slow wave sleep, very difficult to awaken
REM
rapid eye movement stage
sleep
cycle through the stages 4-5 times per night, each one 90 minutes
circadian rhythm
regular body rhythms acorss 24 hour period
dreaming
when eyes are moving rapidly under eyelids
freud theory of dreams
unconscious thoughts and desires that need to be interpreted, little scientific support
evolutionary biology theory of dreams
threat simulation to prepare for real world, problem solving, no purpose
other dream theories
maintain brain flexibility
memory consolidation dream theory
to conolidate memories in deep sleep
activation synthesis hypothesis of dreams
brain gets neural iimpulses in brainstem
sleep deprivation
irritable and worse memory
how much sleep is needed
7-8 hours adults
insomnia
persistent trouble falling or staying asleep
narcolepsy
can’t help themselves from falling asleep
sleep apnea
1/20 people
sleep walking/talking
genetic
induced states of consciousness
hypnosis and medications
hypnotism
relax and focus on breathing, more susceptible to suggestion in this state
dissociation theory of hypnosis
extreme form of divided consciousness
social influence theory of hypnosis
people do and report what’s expected of them, like actors caught in their roles
meditation
training to regulate attention and awareness
psychoactive drugs
alter consciousness and perceptions
depressants
lower body fx and neural activity, dec hr, dec bp
stimulants
enhance CNS, inc bp/hr, inc alertness
hallucinogen/psychedelics
seeing/hearing things
opiates
opiate= natural
cannabis
marijuana, hallucinogen and depressant or stimulant
MDMA
stimulant or hallucinogen
drug classification
baed on legal status and how likely to be abused
homeostasis
how you maintain temp, hr, metabolism
routes of drug entry
oral= slow, GI tract, 30 mins
reward pathway in brain
ventral tegmental area
animal model of reward pathway
if you give non-addicted rat regular food it likes w/ substance that makes it sick, the rat learns to avoid the food
addiction
inc genetic risk
tolerance
get used to drug so need more to achieve same effect
cross tolerance
reducion in efficacy or responsiveness to novel drug due to common CNS target
withdrawal symptoms
when you dont have the drug
drug processes
intoxication and withdrawl
withdrawal stages
acute= few weeks, physical withdrawal, for alcohol seen 2 days after and then gets better
substance-induced disorders
mood/sleep disorders
substance-use disorders
drug causes serious degree of impairment of life functioning
methadone
used to treat heroine addiction because activates opiate receptors but more slowly so dampens the high and reduces the craving
CBT
cognitive behavioral therapy
motivational interviewing
find intrinsic motivation to change, goal oriented therapy, few sessions can be doorway to another treatment
group meeting
AA, 12-step program
relapse
patient slips and goes back to dependence
divided attention
switching attn between 2 tasks rather than doing them simultaneously, when performing 2 tasks which require attn simultaneously
joint attention
focus on object by 2 separate individuals
direct attention
attn focused on single sustainable task
selective attention
selecting one thing at a time (studying vs. TV)
exogenous/external cues
pop in the corner
endogenous/internal cues
requires internal knowledge
cocktail party effect
concentrate on one voice in a crowd
inattentional blindness
aka perceptual blindness
change blindness
fail to notice changes from previous to current state in environment
distal stimuli
events out in world around you
proximal stimuli
patterns of stimuli from objects and events that actually reach your senses
covert orienting
act of brining attnetion to something without body/eye movement
overt orienting
person turns body to maximize sensory impact
neglect syndrome
damage to brain causes loss in capacity of spatial dimension or divided attention
vigilance attention
detect signal of interest, allows for priming
alerting attention
affected by aging, norepinephrine modulates it in locus coerculus
orienting attention
capacity to change focus of attention from one stimulus to another, modulated by acetylcholine from basal forebrain
basal forebrain
striatum, nucleus accumbens, nucleus basalis, septal nuclei
executive attention
goal-directed behavior
shadowing task
theory of selective attention
broadbent’s early selection theory
info goes into sensory register which stores everything
Deutch and Deutch’s late selection theory
selective filter after perceptual processes
treisman’s attenuation theory
we have an attenuator that weakens but doesn’t eliminate input from the unattended ear
Johnson and Heinz propositin
location of info attenuator was able to be varied by listener depending on demand needed for a particular task
spotlight model of attention
take info from 5 senses but dont pay attention to everything
resource model of attention
limited resources ni attention, inability to multitask
tast similarity
harder to multitask with similar tasks
task difficulty
harder tasks require more focsu
information processing model
brains are similar ot computers
sensory memory
iconic= memory for what you see, lasts .5 seconds