Body

Question 1

Sign of malignant transformation of IPMN

Answer 1

Solid enhancing components, rapid interval growth, ductal dilation

Question 2

marginal ulcers post RYGBP

Answer 2

occur as GJ anastamosis, 3-13% of pts. usually solitary, variable size

Question 3

cortical nephrocalcinosis

Answer 3

chronic GMN
acute cortical necrosis

Question 4

medullary calcinosis

Answer 4

hyperPTH, RTA type 1, medullary sponge kidney,

Question 5

Protocol for staging pacreatic CA workup?

Answer 5

arterial and PV

Question 6

seminal vesicle cyst association

Answer 6

ipsilateral renal agenesis, renal dysplasia (67 % of cases due to common embryonic origin)

Question 7

what is malacoplakia

Answer 7

uncommon granulomatous reaction to infection, most often occurs in bladder

Question 8

complicatoins of cross-fused renal ectopia

Answer 8

VUR is most common
nephrolithiasis, pyelo, hydro also common

Question 9

pancreatic embryologic origins (buds and ducts?)

Answer 9

ventral and dorsal buds, ventral rotates posteriorly to fuse.
ventral duct (of Wirsung)
Dorsal duct (Santorini)

Question 10

pancreatic divisum

Answer 10

most common anatomical variant/anomaly.
usually Asx. can have recurrent pain/ pancreatitis (because the main duct drains through minor papilla, CBD drains major papilla)
different variants of duct fusion/drainage

Question 11

Santorinicle?

Answer 11

outpouching near the end of the dorsal duct in pancreas divisum