Bleeds in the brain - EDH, SDH, SAH, ICH, DAI Flashcards

1
Q

what are the 5 main types of bleeding into the brain

A
extradural
subdural
subarachnoid
intracerebral
(diffuse axonal injury)
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2
Q

what does each type of bleed look like on a CT scan

A

ED - lens shape

SD - ellipse shape (banana)

SA - best seen around circle of willis, dense grey outline

IC - lump of blood somewhere in the brain (common in basal ganglia/thalamus area

diffuse axonal injury - brain looks “fuzzy”, loss of distinction between grey/white matter

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3
Q

how does blood show up on a CT

A

acute blood - white
old blood - black
actively bleed site - dusk sign, grey

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4
Q

what type of bleed is an EDH and how does it occur

A

high pressure arterial bleed

high force mechanism of injury

  • dura firmly attached to the bone
  • small arteries run through this space
  • when force shears the dura away from the bone these arteries bleed
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5
Q

what area/vessel is most at risk of an EDH

A

temple area - pterion is weak

middle meningeal artery at risk

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6
Q

what is the general presentation/progression of a patient with an EDH

A

concussed
then appears well
bleed continues (high pressure arterial)
patient worsens when space expands between bone and dura - starts pushing into brain

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7
Q

what is the management of an EDH

A

emergency - burr hole or small craniotomy to remove blood/release pressure
early detection = better recovery

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8
Q

what type of bleed is a SDH and how does it occur

A

low pressure venous bleed

small trauma mechanism

  • veins go between dura and arachnoid layers (subdural space)
  • stretching/increased pressure across these veins lead to tears
  • bleeds
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9
Q

what is the general presentation/progression of a patient with a SDH

A

small trauma (patient might not even notice it) causes bleed - takes a long time to present clinically
might have multiple bleeds before getting noticeable signs/symptoms
on CT can show “acute on chronic SDH” from multiple small traumas over time

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10
Q

who is at risk of a SDH

A

people with atrophy of the brain
eg elderly, dementia, alcoholics

atrophy of the brain leads to stretching of the veins within subdural space - increases likelihood of ruptures

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11
Q

what is the treatment of a SDH

A

if big can drain with a burr hole

if large and old can insert two small holes and do a wash out of broken down blood

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12
Q

what type of bleed is a SAH and how does it occur

A

high pressure arterial bleed

can occur from head injury or spontaneous aneurysm

  • bleeding between the arachnoid and pia
  • blood accumulates in the SA space around the curvatures of the brain

one type of haemorrhagic stroke

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13
Q

what is the general presentation/progression of a patient with a SAH

A

*sudden onset thunderclap headache
N/V
meningism (irritation of the meninges) - headache, neck stiffness, photosensitivity

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14
Q

what are some causes and risk factors of a SAH

A

trauma
aneurysm (more common in younger patients)

HTN
polycystic kidney disease
smoking
alcohol
cocaine use
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15
Q

where is the most common place for an aneurysm

A

where the arteries bifurcate eg anterior communicating artery in the circle of willis

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16
Q

what are the investigations and management of a SAH

A

CT head within 6 hours of onset
CT angiogram
MRI

investigations guide management

  • lower BP eg labetalol
  • prevent vasospasm eg CaCB (nimodipine) for 21 days
  • coiling (intravascular) to prevent bleeding

reduce risk factors

17
Q

what are some complications of a SAH

A

hydrocephalus - needs a shunt to drain

vasospasm - can lead to ischaemic stroke

18
Q

what type of bleed is an ICH

A

high pressure arterial bleed

one type of haemorrhagic stroke
- ie bleeding into the brain

19
Q

where is it most common for an ICH to occur

A

thalamus and basal ganglia

20
Q

what are risk factors for an ICH

A

HTN
anticoagulants
blood thinners

21
Q

what is a complication of an ICH

A

can spread to the ventricles causing hydrocephalus

22
Q

what is the management of an ICH

A

if spread to ventricles - drain to outside
craniotomy to relieve pressure in extreme cases
antihypertensives

secondary prevention to control BP

23
Q

what is a diffuse axonal injury

A

shearing of tiny vessel between the white an grey matter - leads to petechial haemorrhage

24
Q

how does a DAI occur

A

high velocity impact
shaken head (paeds - “shaken baby”)
white matter and grey matter have different densities
- one moves faster than the other
- axons crossing between the two break
- very tine blood vessels break
- widespread petechial haemorrhage on top of multiple white and grey matter lesions

causes LOTS of swelling - can be very difficult to treat

25
Q

what complications can a DAI lead to

A

increased pressure on the brain due to extensive swelling throughout

  • can lead to brainstem herniation as its pushed down through the foramen magnum
  • can cause ischaemia and pressure damage
  • common cause of persistent vegetative state
26
Q

what is a normal value for ICP and when should you start to worry

A

~10 normal

>20 worry

27
Q

what regulates the ICP

A

systolic BP - cerebro autoregulation can maintain a normal ICP between systole 50-150
outside of this auto regulation fails = symptoms

28
Q

what can cause raised ICP and therefore autoregulation to be put off

A

anything that raises pressure

  • bleeding
  • mass/tumours/cysts
  • oedema
29
Q

DAI management

A

very difficult - try reduce raised ICP from swelling, palliative