Biopsychology of Motivation Flashcards

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1
Q

Richtor (1992)

A

hypothesized that animals have an internal mechanism that spontaneously generates this rhythm (biological clock)

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2
Q

Edogenous Circadian Rhythms

A

internally controlled cycles that last about a day

e.g. birds that migrate

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3
Q

how do we know that the rhythm we have is internally generated?

A
  • If you stay up all night, you feel sleepier as it gets later, but then perk up a bit in the morning
  • Animals kept in total darkness still keep to a 24 hour cycle (approx.) DeCoursey 1960 – rhythm of activity stays the same
  • Humans kept in an environment with a 28hr cycle cannot synchronise, they slip back into a 24 hour cycle
  • Blind and deaf animals generate nearly normal circadian rhythms
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4
Q

DeCoursey (1960)

A

Animals kept in total darkness still keep to a 24 hour cycle (approx.)

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5
Q

Richter (1967)

A
  • theorized that we all have a biological clock = a mechanism in or brain that generates this cycle – physical part of our brain that keeps the time
  • seems to be robust - maintained despite brain damage
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6
Q

Where is the biological clock?

A

Supachiasmatic nucleus above the optic chiasm

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7
Q

What happens to the SNL when damaged?

A

Damage to circadian rhythms

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8
Q

How do we know that the biologic al clock generates this rhythm automatically?

A
  • Earnest et al 1979 - Remove the SCN and keep it is a tissue sulture it continues to procedure a 24 hour rhythm of action potentials
  • Hamsters with a mutant gene coding a 20hr rhythm, SCN removed and implanted in normal hamsters, they start to live a 20hr cycle – good evidence that this part of the brain does all of the work in terms of our biological clock
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9
Q

How does the biological clock work?

A
  • Regulates waking and sleeping through activity in the brain
  • Controls the pineal gland = an endocrine (hormone) gland posterior (behind) the thalamus
  • Releases melatonin – hormone that makes us sleepy
  • SCN tells the pineal gland to secrete this hormone in the 24 hours so you are asleep in the night and awake in the day
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10
Q

Can the biological clock respond to the environment?

A

YES

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11
Q

Zeitgeber

A

‘time giver’
- anything that has an influence on the biological clock e.g. light, excersie
- most dominant Zeitmesser for land mammals is light
- But we also respond to exercise, noise, temperature and meals
- some marine animals respond to tides
- those in Scandinavian countries are insomniac because of 2 hours of daylight
- Hamsters living in constant light, SCN in one hemisphere out of phase with right
• No coordination!
• Two sleep and two wake cycles!

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12
Q

Jet lag

A
  • Disrupting rhythms by crossing times zones
  • Mismatch between biological clock and external stimuli
  • not used to adapting to things so fast
  • can be stressful and raise cortisol levels
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13
Q

Phase Delay

A
  • travelling east to west (UK to USA)

- easiest to adapt to as you just

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14
Q

Phase Advance

A
  • Travelling west to east (USA to UK)

- harder to adapt to as your nights are shortened

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15
Q

Those who work night shifts..

A
  • Feel fatigued
  • Cant sleep well during the day
  • Don’t adjust when working at night, as the light levels aren’t high enough in artificial conditions
  • Need to have a complete reversal to fully change the biological clock
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16
Q

Optic chiasm

A
  • sits nect to the supachiasmatic nucleus
  • where information is transferred from our retina, links and nerves to the eye
  • important in light
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17
Q

Melanopsis

A
  • special photopigment that knows light from dark
  • responds directly to light
  • repsonds slowly and turns off slowly
  • So the SCN gets an idea of general light intensity – perfect to gauge time of day
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18
Q

Kalat - blind mole rat

A

blind mole rat can respond to light through melanopsin (can tell light from dark)
- other reptiles have third eye on their head where melanopsin is kept

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19
Q

EEG

A

can be used to detect electrical signals of spontaneous brain activity OR in response to a stimulus (ERP – Event Related Potential)

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20
Q

Stage 1 and 2 of sleep

A

irregular activity, high but declining, bursts of activity in stage 2, cortex still receiving sensory input e.g. can still wake up when someone touches you

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21
Q

Stages 3 and 4 of sleep

A

slow wave sleep (SWS) = neuronal activity is highly synchronized, sensory input reduced – harder to wake up

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22
Q

REM

A
  • Also called paradoxical sleep because it doesn’t catagorise into the stages
  • Neither nor deep sleep
  • Can be categorized as Light because lots of brain activity
  • But can also be catagorized as Deep because muscles are relaxed
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23
Q

What does the brain do during REM?

A
  • Lateral geniculate neucleus in the thalamus (limbic system) → responsible for emotion
  • Pons (brainstem) = ‘bridge’ axons from cortex cross here to spinal cord → movement (inhibits) – any signals going through movement or muscles go through this brain stem - it is not activated in a way that increases movement it actually inhibits, so your pons are activated and your muscles are relaxed as a result of this
  • PGO waves (pons-geniculate-occipital) – work in a cycle – activity of pons so the muscles are relaxed, which leads to activity of the geniculate so you get lots of emotional activity and then lastly there’s the activity of the occipital cortex which is where a lot of visual processing occurs which is why we appear to see our dreams, feels like we are there, this is why we get rapid eye movement – controls our wakefulness
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24
Q

What can happen when you are deprived of sleep?

A

Hallucinations - start to dream when you are awake

25
Q

Morrison et al 1995

A

healthy cat, damage pons, can has normal REM sleep but muscles aren’t relaxed so cat chases prey (acts out dreams)
- the pons is thus inhibited when we sleep so that we don’t act out our dreams

26
Q

Insomnia

A
  • stress, anxiety, depression
  • shifting circadian rhythms - out of sync with the environment
  • dependence on sleeping pills
27
Q

Sleep Apnea

A
  • inability to breathe while sleeping

- due to obesity or old age?

28
Q

Narcolepsy

A
  • attacks of sleepiness during the day

- fall on the floor because they jump into REM so their pons is inhibited and all of their muscles are relaxed

29
Q

Periodic Limb Movement Disorder

A
  • Involuntary movement of the legs/arms
  • Maybe something to do with pons?
  • Brainstem is not inhibited properly and the muscles are not relaxed
30
Q

Sleep ad a form of Hibernation

A
  • Allows the Conserving of energy when you cant get much done – when food is scarce, light is too high/too low etc. – anscestors sleep during the night because that’s when it is most convenient e.g. when its cold and they don’t have light – instead of wasting energy during this time they sleep
  • Lyman et al 1981 - hibernating hamsters live longer
31
Q

Lyman et al (1981)

A

Hibernating hamsters live longer than other hamsters

32
Q

Animals vary in sleep depending on:

A
  • safety from predators
  • how much time they need to find food
  • whether they need to surface for air or not (e.g. dolphins)
  • food - grazing animals eat a lot of unnutricious food (don’t spend a lot of time sleeping)
  • giant sloths have a lot of rich, nutritious food so don’t have to eat all the time so sleep more
33
Q

What are the other functions of sleep other than conservation?

A
  • sleep deprivation causes dizziness, hallucinations etc.
  • immune system will fail eventually
  • sleep enhances memory
34
Q

Who has more REM sleep?

A

babies

- some birds do but not all animals - shows it must be a biological process

35
Q

Why do we need REM?

A

1 - To strengthen memories and weed out pointless connections
- however sleep as a whole improves memories not just REM
- anti-depressant drugs reduce REM but people have no problems with memory
2 - To moisten eyeballs
- but those who are deprived of REM through the use of anti-depressant drugs don’t have any worse eyes

36
Q

Why do we dream?

A

1 - The Activation-Synthesis hypothesis

2- The Clinico-Anatomical Hypothesis

37
Q

The Activation-Synthesis hypothesis

A
  • Effort to make sense of distorted info

- PGO waves from pons activate parts of the cortex, which synthesizes a story

38
Q

The Clinico-Anatomical Hypothesis

A
  • Dreaming is thinking
  • Senses are suppressed. So brain left to its own devices
  • Motor cortex suppressed, so no action
  • Pre-frontal cortex suppressed, so no working memory to link a believable story together
39
Q

Wood frogs temperature regulation

A
  • can survive the freezing cold through psychological mechanisms because they have anti freeze agents in their physiology they can be frozen and still survive
40
Q

Flamingos temperature regulation

A

– stand on one leg when in water so that less of their body is exposed to water - keeps them warm

41
Q

Human temperature regulation

A
  • Shiver – increases the metablolism in our cells -
  • Sweat – cycle of evaporation
  • Blood vessel constriction when we are cold e.g. hands turn blue
  • Blood vessel dilation when we are warm
  • All of these behaviours are a product of the hypothalamus
42
Q

Pre-optic area of the brain

A
  • near the hypothalamus

- responsible for temperature regulation

43
Q

Gerbils Thirst Mechanisms

A
  • may never drink
  • eat lots of fatty and dry foods like seeds and nuts
  • have a mechanism and metabolism to turn this fat into water and thus they can survive on metabolism alone
  • dry faeces so they don’t excrete and water that they produce
44
Q

Beavers thirst mechanisms

A

drink lots and urinate lots

45
Q

Human Thirst mechanisms

A
  • If water is scarce, the pituitary gland secretes vasopression (a hormone)→ blood vessels constrict → raises blood pressure and compensates for the low fluid volume
  • vassoprosin also an antidiuretic hormone (ADH) - makes urine more concentrated by causing the kidneys to reabsorb fluid form urine
46
Q

why do hangovers make us thirsty?

A
  • Drinking alcohol blocks the production of vasopression by the pituitary gland
  • As this is blocked, it Prevents the kidneys from absorbing water
  • Makes urine more diluted
  • Morning after we experience massive widespread dehydration – hense very thirsty
47
Q

How do we know what to eat?

A
  • Combination of learned and unlearned strategies
    o Learned from peers, culture
    o But innate atstes are essential
    o Likeness of sweet food, disgust for bitter/sour
48
Q

How do we know when to eat?

A
  • Centres around the hypothalamus (forebrain area associated with regulating behaviours) hypothalamus has neurons that are sensitive to hunger and it can communicate when then person is hungry
  • Hypothalamus has neurons sensitive to hunger and feeling full
49
Q

Obesity

A
  • cultural influence
  • Castor 2000 - social factor of eating
  • rooted in our biology, propensity to like fatty foods
  • native population adapted to a specific diet, now exposed to new foods
  • some genetic disorders
50
Q

Anorexia Nervosa

A
  • unwillingness to eat
  • 0.3% of women
  • perception of fatness
  • body can deteriorate, muscle wasting, can even cause death
  • unlikely to have a specific genetic basis
  • serious condition, but not as widespread or culturally problematic as obesity
51
Q

Bulimia Nervosa

A
  • extreme dieting mixed with binge eating
  • vomit after mealtimes (not all)
  • imbalance of hormones associated with feeding
  • but may be a result of erratic eating rather than cause
52
Q

How is bulimia related to drug addiction?

A
  • rats deprived of food for 12h
  • drank lots of sugar solution which released dopamine and opiate like compounds in the brain
  • when deprived of sugar solution, withdrawal symptoms occurred
53
Q

How high men and women differ?

A
  • sexual strategies - reproductive behaviour(mate choice, attitudes to sexual behaviour)
  • cognition (reproductive and survival strategies)
54
Q

Sexual strategies of men

A
  • numerous, mobile sperm

- More promiscuous

55
Q

Sexual strategies of women

A
  • few, immobile eggs

- the choosy sex

56
Q

Waynforth and Dunbar (1995)

A

Evolutionary battle of the sexes

57
Q

Men

A
  • Androgens (testosterone) produced in the testes and the adrenal gland (in the brain)
  • Sensitizes regions of the brain underlying sexual motivation
  • Removal of the testes (i.e. cancer) results in decreased sex drive, BUT still some production in the brain
58
Q

Women

A
  • Oestrogens produced by the ovaries
  • AND androgens (testosterone) produced by the adrenal gland
  • As in men, androgens (testosterone) associated with sex drive
  • Postmenopausal women can still have high sex drive due to androgen production (even when oestrogens have dropped) – because they are still producing testosterone