Biopsychology Flashcards

1
Q

3 types of neuron and their function

A

Sensory- convert info from sensory receptors in various locations in body into nerve impulses and sends to brain and spinal cord.
Relay- connect sensory neurons to motor or other relay neurons.
Motor- connect CNS to effectors such as muscles and glands.

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2
Q

Dendrite

A

Branch like structures that receive signals and carry them towards cell body.

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3
Q

Myelin sheath

A

Protects axon and causes nerve impulses to travel quicker.

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4
Q

Axon

A

Extension of neurones and carries impulse away from cell body towards axon terminal.

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5
Q

Inhibitory neurotransmitter

A

Act as nervous system “off switches” calming the mind and body and inducing sleep. Filter out unnecessary excitatory signals.

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6
Q

Excitatory neurotransmitters

A

“on switch” of nervous system (e.g. adrenaline)

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7
Q

Synaptic transmission process

A

Synaptic vesicles are the sacs at the end of an axon.
As action potential arrives at the end of axon sacs are pushed to release neurotransmitters.
This diffuses across the synapse.
It’s then taken up by receptors.
This is converted back into an electrical impulse and process repeats itself.

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8
Q

Fight or flight response SAM pathway

A

1) Amygdala appraises situation
2) Sends stress situation to hypothalamus which activates sympathomedullary.
3) Adrenal medulla stimulates SNS.
4) Adrenal medulla secretes hormones adrenaline and noradrenaline into bloodstream.
5) Adrenaline causes many psychological changes to prepare for ‘fight of flight’

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9
Q

Changes seen during fight or flight response (Sympathetic state)

A

-saliva production inhibited
-HR increase
-rectum contracts
-Pupils dilate
-Inhibits digestion
-breathing rate increases
-Sweat to regulate body

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10
Q

What are hormones?

A

Chemical substances that circulate in the blood stream and affect target cells. They are very powerful and produced in large quantities but they disappear quickly.

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11
Q

Target cells

A

Cells hormones have an effect on. Cells are capable of responding to hormones because they display receptors which circulatory hormones can bind to.

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12
Q

What does the thyroid gland produce and what is it’s function?

A

Releases Thyroxine in the neck having affects of increasing HR, metabolic rates and affects growth rates.

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13
Q

What is the Pituitary gland?

A

Known as the ‘master gland’ as it controls the release of hormones from other glands. Located in the brain (below hypothalamus).

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14
Q

What is Plasticity?

A

Life experiences lead to changes in brain structure, (easier in infancy as still rapidly forming new neural connections)

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15
Q

Localisation

A

Idea that specific brain areas perform specific functions

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16
Q

Frontal lobe

A
  • Both hemispheres
  • Makes sense of info about environment, memories, emotions and uses this info to make decisions
  • Functions personality, decision making, motor control
  • If damaged personality change is seen, impulsivity and difficulty concentrating/planning
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17
Q

Motor cortex

A
  • Both hemispheres
  • responsible for functioning voluntary movements sending signals to muscles
  • Motor deficits- e.g. weakness, paralysis,loss of fine motor control
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18
Q

Somatosensory cortex

A
  • Both hemispheres
  • Recieves incoming sensory info from skin to produce sensations
  • If damaged then struggle to differentiate sensations e.g. hot/cold
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19
Q

Visual cortex

A
  • Both hemispheres
  • Recieves and processes visual info- e.g.colour, shape, movement in diff parts
  • If damaged visual awarness is abolished, leads to chronic blindness
20
Q

Auditory cortex

A
  • Both hemispheres
  • Responsiblefor analysing and processing acoustic info including volume, tempo and pitch
  • If damaged damaged deficits to detect changes in pitch,understand speech, localise sound in space- cortical deafness could occur.
21
Q

Broca’s area

A
  • Left hemisphere
  • production and coordination of speech
  • If damaged Broca’s aphesia occurs- slow, laboured speech and lacks fluency
22
Q

Wernicke’s area

A
  • Left hemisphere
  • Allows us to comprehend language (written and spoken)
  • If damaged Wernike’s aphesia occurs- speech is robbed of meaning but production is left fluent.
23
Q

Evaluation of the localisation of brain functions

A

-May be that only basic functions are localised and not higher ones
+Support E.g. Stroke patients- Tulving episodic and semantic in different areas. Case Studies- Phinneus Gage PFC
-Gender diffs- women larger Broca’s area
-Conflicting evidence- 2 of Broca’s patients had brains preserved and scanned and showed damage in other areas too.

24
Q

Lateralisation

A

Idea that each hemisphere has functional specialisms

25
Lateralisation in the left hemisphere
* Receives info from right visual field * Controls right side of the body * Performs tasks like logic, language and reasoning
26
Lateralisation in the right hemisphere
* Recieves info from left visual field * Controls left side of the body * Performs spatial tasks, art, music
27
Describe Sperry's split brain research procedure
11 patients, quasi experiment, screen, flashing image Task- name what you see (language test) or draw what you see (visuo-spatial test)
28
Sperry's split brain research findings
Left hemisphere- analytic and verbal tasks Right hemisphere- Spatial tasks and music However the fact all ptts had severe epilepsy may suggest they became more lateralised than the general population
29
Lateralisation evaluation
-Age: we become less lateralised as we age (Szaflarski et al 2006)- Lang becomes more lateralised to LH w increasing age when we are children & adolescents but when we reach 25 yrs lateralisation decreases w every decade of life. Raises Qs about whether everyone has a hem that's more dominant. +/- Sperry's work: Well controlled standardised procedures, impressive findings, however he used a small sample size of 11 patients who all had severe epilepsy so may make difficult to generalise. -Case studies such as JW (who learned to speak out of his right hemisphere) shows that language can be processed in right hem which contradicts the idea that language is processed in left hem. -Duality theory (idea of 2 seperate brains) is oversimplified: in reality the differences between right and left hem are more intricate than lateralisation suggests.
30
What are the 3 biological rhythms?
Circadian rhythm- 24 hrs (e.g. sleep/wake cycle) Infradian rhythm- period longer than 24hrs (e.g. menstrual cycle, SAD) Ultradian rhythm- occurs more than once in 24hrs (e.g. sleep cycle)
31
What are the stages of the sleep cycle? (ultradian rhythm)
Stage 1- theta waves appear,feel as if falling breathing and HR and temp fall slows. Stage 2- sleep spindles (sudden bursts activity) and K-Complexes (suppressing outside stimuli). Stage 3 & 4- deep sleep, delta waves, gradually become unresponsive,confused if woken, sleep walk & talk, bedwetting, no eye movement, the lowest point for HR, BP and temp. Stage 5- REM (rapid eye mov), sleep paralysis,beta waves resembeling awake state, HR & BP fluctuate, dreaming occurs.
32
Describe menstrual cycle (Infradian rhythm)
* Typically 28 days * Controlled by Pituitary gland receiving info from hypothalamus * Ovulation occurs as oestrogen rises * Progesterone helps womb lining grow thicker * If pregnancy doesn't occur egg is absorbed and womb lining leaves body
33
Describe SAD (Infradian rhythm)
* Seasonal affective disorder 'winter blues' * Pineal gland secretes melatonin until dawn (there is more light) * Lack of morning light in Winter means melatonin secretion lasts longer making us more tired. * Has knock on effect of melatonin production in the brain
34
Endogenous pacemakers (circadian rhythm)
* Time makers * Synchronised by the master clock: Suprachiasmatic nuclei (SCN) found in hypothalamus. * SCN constantly reset by light levels and tells pineal gland to produce melatonin to induce sleep.
35
Exogenous Zaitgebers (circadian rhythm)
* External factors (time givers) * Help rest endogenous pacemakers * Light primary input into system- setting body clock to correct time * Entrainment can occur when we move across zones * Other EZs (e.g. regular meal/bath times
36
What was Siffre's cave study?
* He spent extended periods underground to study effects on his own bio rhythm * He had no exposure to natural light and was sound deprived * He had access to adequate food and drink * Did 2 months underground in a cave in the alps and 6 months in a Texan cave * He discovered that his biological clock settled down to a **25hr clock**
37
Aschoff & Wever study
* Convinced group of ptts to spend 4 weeks in WW2 bunker * All ptts kept a circadian rhythm between 24-25 hrs apart from 1 ptt (29hrs) * Suggests natural sleep/wake cycle is slightly longer than 24hrs but is entrained by exogonous zeitgebers.
38
Folkard study
* Ptts lived in a cave and apparant 24hrs turned into 22hrs * Only 1 ptt was able to adjust * Represents we have internal mechanisms that can't be easily overriden
39
# Menstrual cycle Stern & McClintock study (1980)
* Collected sweat from one woman (donor) * Wiped under noses of female ptts * In 68% of cases changes seen in womens menstural cycle as they became closer to donor * This occurs as pheromone molecules dissolve inside the nose, stimulate neurones, affecting hypothalamus which alters pattern of hormone production.
40
# Ultradian rhythm Dement & Kleitman (1957)
* looked at EEG readings * Made sure exp controlled for effects of caffine and alchol * Found REM sleep to be highly correlated with dreaming * the more vivid the dream, the more active the brain * If woken during REM stage ppl could easily recall their dreams
41
Evaluation of circadian rhythms
+RWA: shift work (lose concentration at 6am), jet lag (Burgess=exposure to light). +Research support: Sifffre's cave study that our sleep/wake cycle settles down between 24-25 hours and that exogenous zeitgebers help keed it to a 24hr rhythm. -Individual differences:
42
What is functional recovery?
Following injury/trauma (e.g.stroke) unaffected areas of brain compensate and adapt.
43
# SCS How does plactisity occur?
*** Synaptic reweighting**- If activity increases more neurotransmitters released and stronger signal. *** Creation of new synapses**- If axon of one nerve cell passes close to dendrite of another then a new synapse is formed. *** Synaptic pruning**- deleting synapses that are not used, 'use it or lose it'.
44
How does functional recovery happen?
*** Neuronal unmasking**- Dormant neurones unmasked that are close to area of damage. *** Axonal sprouting**- Undamaged axons physically grow new nerve endings and hook onto dendrites of other neurones and form new synapses. *** Recruitment of homologous areas**- Mirror neurones on opposite side of brain gradually take over lost functions.
45
Studies supporting plasticity
**Maguire research on taxi drivers:** Changes in brain detected as a result of extensive experience of spatial navigation. MRI discovered had greater posterior hippocampi. **Boyke et al study on 60yr olds learning to juggle:** Increase in grey matter (neurones) in visual cortex, however changes reversed when practise stopped.
46
Studies supporting functional recovery
**Danielli et al, case study of EB**- 2 and half yr old Italian boy. Virtually all of LH removed meaning his linguistic abilities disappeared. At 5 after rehab there were no major problems in his linguistic ability, by 17 his RH had fully compensated. **Takatsuru et al**- If undamaged area stimulated soon after stroke then recovery can be improved.