Bicipital Tendinopathy and SLAP Flashcards

1
Q

general term that refers to chronic tendon pathology

A

Tendinopathy

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2
Q

inflammation of the synovial membrane covering a tendon.

A

Tenosynovitis

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3
Q

inflammation of a tendon; there may

be resulting scarring or calcium deposits.

A

Tendinitis

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4
Q

inflammation with thickening of a

tendon sheath.

A

Tenovaginitis

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5
Q

degeneration of the tendon due to

repetitive microtrauma.

A

Tendinosis

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6
Q

inflammation of the outer layer of the tendon (paratenon) alone.

A

Paratenonitis

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7
Q

paratenonitis associated with intratendinous degeneration.

A

Paratenonitis with tendinosis

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8
Q

what type of lesions show degenerative
fraying with no detachment of the
biceps insertion?

A

Type 1

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9
Q

what type of lesions show detachment of the

biceps insertion

A

Type 2

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10
Q

what type of lesions show a bucket handle
tear of the superior aspect of the
labrum with an intact biceps tendon
insertion to bone?

A

Type 3

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11
Q

what type of lesions show an intrasubstance
tear of the biceps tendon with a bucket
handle tear of the superior aspect of
the labrum?

A

Type 4

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12
Q

a Bankart lesion that extends superiorly to

include a Type II SLAP lesion.

A

Type 5

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13
Q

an unstable flap tear of the labrum in

conjunction with a biceps tendon separation.

A

Type 6

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14
Q

a superior labrum and biceps tendon
separation that extends anteriorly, inferior to the
middle glenohumeral ligament.

A

Type 7

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15
Q

pathological shoulder condition that deems an athlete
unable to throw due to pain and subjective unease in the shoulder – associated SLAP type II lesion as the MC pathology associated with the syndrome

A

Dead-arm syndrome

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16
Q

with the arm in the cocked position of abduction and lateral rotation the peel-back occurred. The change in angle and the twist of the biceps tendon produced a torsional force to the posterosuperior labrum, causing
detachment if the superior labrum was not well anchored to the glenoid

A

Peel-back phenomenon

17
Q

– occurred as the biceps contracted to slow the rapidly extending elbow in the
follow-through phase
– believed that this created a high tensile load in the biceps that acted to pull
the biceps and superior labrum complex from the bone.

A

Deceleration Mechanism

18
Q

– Occurs in late cocking, as the arm began to accelerate forward from an
abducted and laterally rotated position, the long head of the biceps and the
superior labrum were peeled back rather than pulled from the bone

A

Acceleration Mechanism

19
Q

Clinical factors associated to dead-arm

syndrome:

A

– a tight posteroinferior capsule - > causes a GIRD and a shift in the glenohumeral rotation point
– the peel-back mechanism produced by the biceps tendon -> leads to a SLAP lesion,
– hyperexternal (lateral) rotation of the humerus
– scapular protraction.

20
Q

Two cited causes of SLAP lesions

A
  • FOOSH which drives the humeral head up
    onto the labrum and the biceps tendon
  • excessive and forceful contraction of the
    biceps in throwing athletes (e.g., baseball
    and football players)
21
Q

triangular-shaped area between the

supraspinatus and the subscapularis tendons

A

rotator interval

22
Q

2 Classification of Biceps Tendonitis

A

Primary tendonitis & Secondary tendonitis

23
Q

occurs as a result of

pathology of the tendon sheath

A

Primary tendonitis

24
Q

occurs secondary to an
underlying injury that causes subsequent
biceps irritation and injury

A

Secondary tendonitis

25
Q

three-part classification for

biceps lesions

A

Type A lesion
Type B lesion
Type C lesion

26
Q
  • referred to as impingement tendonitis
    because it occurs secondary to an impingement
    syndrome and rotator cuff disease.
  • Most common
A

Type A lesion

27
Q

describe a subluxation of the biceps
tendon. All subluxations and dislocations of the biceps
tendon are included in this category.

A

Type B lesions

28
Q

attrition tendonitis

A

type C lesion

29
Q

Clinical Presentation of Biceps Lesions

A

• pain in the proximal anterior area of the shoulder directly over the biceps
tendon, with occasional radiation of pain down into the muscle belly
• pain can also be described at the deltoid insertion
• pattern of pain can be linked to repetitive types of activities
• Night pain
– compressive load may be present (e.g., lying on the affected shoulder)
or in some instances traction may be applied to the shoulder
– Position of comfort: lying on the contralateral side with the affected
arm in neutral, resting on the pillows
– Many patients with a painful shoulder report that their best rest is
achieved in a semireclined position, such as in a recliner, with the arm
resting on a pillow in the shoulder resting position

30
Q

Labral Injury Mechanism

A

Deceleration mechanism & Acceleration mechanism

31
Q

Arthrography

A

advantages:
shows biceps tendon subluxation, ruptures, dislocations

disadvantages:
invasive
sharp images of the tendon may be lost

32
Q

Bicipital groove view radiography

A

advantages:
shows the width and medial wall of the bicipital groove, spurs in the groove

disadvantages:
does not show possible intra-articular d/o

33
Q

MRI

A

advantages:
excellent evaluation for superior labral complex and biceps tendon

disadvantages:
partial tears of the biceps tendon are more difficult to detect than complete ruptures
expensive

34
Q

Radiography

A

advantages:
R/O SH fx & strains or dislocations of the AC jt

disadvantages:
shows only bony origins of impingement syndrome and not soft tissue

35
Q

Ultrasonography

A

advantages:
relatively inexpensive
dynamic
widely available

disadvantages:
requires experienced operator
high frequency array transducer