Bell's Palsy Flashcards
Define Bell’s Palsy
Acute unilateral lower motor neurone facial nerve palsy
Aetiology of Bell’s Palsy
Idiopathic
60% are preceded by an URTI (suggests viral or post-viral aetiology)
Suggested: reactivation of HSV-1 within the geniculate ganglion -> facial nerve conduction blockade
Risk factors for Bell’s Palsy
Intranasal influenza vaccination Pregnancy Upper respiratory tract infection Black/hispanic Arid/cold climate Hypertension Family history Diabetes
Symptoms of Bell’s Palsy
Preceding URTI + pain (post-auricular + otalgia)
Single episode of unilateral facial weakness and droop
May have facial, neck or ear pain/numbness (50%)
Hyperacusis (sensitivity to sound due to stapedius reflex involvement)
Dysgeusia (taste disturbance)
Signs of Bell’s Palsy
Unilateral LMN weakness of facial muscles with NO forehead sparing
Bell’s phenomenon: eyeballs roll up but the eye remains open when trying to close the eyes
Clinical sensation is normal despite reported numbness
Keratoconjunctivitis sicca -> epiphora + hyperlacrimation
Synkinesis
Investigations for Bell’s Palsy
Clinical diagnosis
Electroneuronography (ENoG): >90% in amplitude of compound muscle action potential
EMG: absence of voluntary motor unit potentials
Serology for Borrelia burgdorferi: negative (Lyme disease)
Management for Bell’s Palsy
Acute
- Corticosteroid e.g. prednisolone
- Eye protection (glasses + artificial tears)
Severe palsy/paralysis
1. Concurrent antiviral therapy e.g. aciclovir
2. Surgical decompression
3, Lateral tarsorrhaphy (suture the lateral parts of the yield together) if corneal damage
Complications of Bell’s Palsy
Keratoconjunctivitis sicca Exposure keratopathy Ulcerative keratitis Ectropion (sagging eyelid) Contracture and synkinesis Gustatory hyperlacrimation Corneal ulcers
Prognosis for Bell’s Palsy
Incomplete paralysis on clinical examination - 94% will fully recover
Complete paralysis - 61% will fully recover
Pregnancy-associated Bell’s may be associated with worse long-term outcomes