Bell's Palsy Flashcards

1
Q

What is a Bell’s Palsy?

A

Acute neurological condition that presents with a rapid onset of unilateral facial paralysis (usually less than 72 hours)

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2
Q

What causes Bell’s Palsy?

A

Unknown origin

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3
Q

What type of lesion is Bell’s palsy?

A

Lower motor neuron lesion

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4
Q

What nerve does Bell’s palsy effect?

A

7th cranial nerve (facial nerve)

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5
Q

What do the motor fibres, carried by the facial nerve, innervate?

A

Motor fibres innervate the muscles of facial expression

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6
Q

What do the sensory fibres that the facial nerve carries do?

A

The sensory fibres provide taste to the anterior tongue

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7
Q

What do the parasympathetic fibres that are carried by the facial nerve do?

A

The parasympathetic fibres go to the lacrimal and salivary glands

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8
Q

What are the three key components that are affected in Bell’s Palsy?

A

Motor component = unilateral facial weakness
Sensory component = altered taste
Parasympathetic component = dry mouth

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9
Q

What is the prevalence of Bell’s palsy per year?

A

20-30 per 100,000

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10
Q

What are the risk factors for developing a Bell’s palsy?

A
Age 15-45 years
Pregnancy 
Diabetes mellitus
Recent upper respiratory tract infection
Intranasal influenza vaccination (no longer in use)
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11
Q

What are the symptoms of a Bell’s palsy?

A
Unilateral fcaial weakness
Altered taste
Hyperacusis (noise sensitivity)
 - Due to paralysis of stepedius muscle (which is innovated by VII)
Dry mouth + dry eyes
Ear pain 
 - seen in upto 50% of patients
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12
Q

What is the key primary differential between a stroke and Bell’s palsy?

A

Bell’s patients cannot raise eyebrows whereas patients with a stroke can move their forehead (fore-head sparing) as it receives innovation from both sides of the brain

N.B. Absence of other weakness/sensory disturbance to upper and lower limbs in Bell’s Palsy

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13
Q

What are the signs of a Bell’s palsy?

A

Facial paralysis

  • Inability to move facial expressions on affected side
  • Forehead is affected
  • Inability to raise eyebrows

Change in taste to the anterior tongue

Unable to fully close eyes

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14
Q

What is the predicted pathophysiology of Bell’s palsy?

A

Herpes simplex 1 within geniculate ganglion

Reactivation results in destruction of ganglion cells and infection of Schwann cells

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15
Q

How is Bell’s palsy diagnosed?

A

Diagnostic investigations are not required = Bell’s palsy is clinical diagnosis + ‘diagnosis of exclusion’

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16
Q

What investigations should be considered if an alternative cause of the facial palsy is suspected?

A

CT/MRI - view facial nerve and surrounding structures

Infectious disease screen - HIV, syphilis, Lyme disease, glandular fever

Angiotensin-converting enzyme (ACE) - sarcoidosis can present with bilateral facial nerve palsy
Serum ACE used as screening tool

17
Q

What is the management for Bell’s palsy?

A

Oral prednisolone - if patient presents within 72 hours of onset of symptoms

Anti-virals e.g. aciclovir - not recommended alone but considered in combination with corticosteriod

Eye protection - eye care discussed with patients + prescription of artificial tears + eye lubricants

18
Q

What are the complications of a Bell’s palsy?

A

Eye injury
Facial pain
Psychological disturbance - increased risk of depression

19
Q

How long will it take for patients to recover from a Bell’s palsy?

A

70% of patients recover their motor function completely within six months without any treatment

30% of patient have incomplete recovery or residual effects

20
Q

What factors indicate a poor prognosis for a Bell’s palsy?

A

old age, hypertension, diabetes mellitus and taste impairment