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bactéries Flashcards

1
Q

Salmonella sp micro bio

A

fermenteur, gram -, oxydase -, catalse +

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2
Q

salmonella sp clinique

A

gastroenterite (fièvre, nausées, vomissement, diarhée sans sang, crampes abdo)

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3
Q

salmonella sp facteurs virulence

A

tolerance aux acides phagocytiques (survie dans macrophages, se propagent)

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4
Q

salmonella sp réservoir

A

animal et humain

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5
Q

salmonella typhi réservoir

A

juste humain

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6
Q

salmonella typhi facteur virulence

A

multipli a temperature piece

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7
Q

salmonella typhi clinique

A

fièvre typhoide ou enterique (colonisation asymptomatique)

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8
Q

Shigella sp microbio

A

gram-. oxydase -, catalase +, fermenteur lent

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9
Q

shigella sp reservoir

A

juste humain

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10
Q

shigella sp clinique

A

insuffisance rénale, shigellose (gastroenterite), ténesme, diarhée liquide, crampes

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11
Q

shigella sp facteurs virulence

A

toxine shiga: cause dommage glomerules: insuffisance renale (systeme secretion type 3)
réarrange actine: induit apoptose

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12
Q

klebsiella sp clinique

A

cause destruction necrotique des alveoles pulmonaire, mene a cavité), granulome inguinae (ITS: nodule, brisure, lesion granulomateuses)

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13
Q

klebsiella sp microbio

A

gram-, oxydase-, catalse +, fermenteur

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14
Q

klebsiella sp reservoir

A

juste humain

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15
Q

protéus mirabilis clinique

A

divise urée en co2 et ammoniac, augmente pH, precipite Ca et Mg, forme pierres aux reins

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16
Q

proteus mirabilis microbio

A

fermenteur lent ou nul, gram-, oxydase-, catalase +

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17
Q

yersina sp microbio

A

fermenteur lent, gram-, oxydase-, catalase +

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18
Q

yersina sp clinique

A

gastroenterite, abces intra-abdominale, hepatite, osteomyelite

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19
Q

yersina sp facteurs virulence

A

adherines, cytotoxines(interruption filaments actine), inhibe migration phagocytes (supprime production cytokines, empeche inflammation), inhibe aggregation plaquettaires

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20
Q

yersina pestis clinique

A 
peste bubonique (bacteremie)
peste pulmonaire: fièvre, malaise
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21
Q

yersina pestis facteurs virulence

A

sequestration facteurs croissance (absorbe fer organique), active plasmogène (degrade C3b:opsonisation et C5a:recrutement macrophages et caillots fibrines:propagation rapide)

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22
Q

yersina pestis reservoir

A

animal et humain accidentel

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23
Q

yersina enterolitica clinique

A

enterocolite (gastroenterite: fièvre, diarhée, douleur abdominale, gonflement ganglions)

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24
Q

yersina enterolitica reservoir

A

animal (aliments ou sang contaminé)

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25
Q

yersina enterolitica facteur virulence

A

croit a basse temperatures

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26
Q

escheria coli microbio

A

fermenteur, gram-, oxydase-, catalase+, bouge par flagelles et peritriches

27
Q

escheria coli facteur virulence

A

adhesine, toxines(hemolysine)

28
Q

escheria coli clinique

A

gastroenterite, meningite (souche exogene)

29
Q

clostriduim perfringens microbio

A

gram+, double hemolyse, anaerobe

30
Q

Clostridium perfringens facteurs virulence

A

toxines:

alpha: lyse erythrocytes, leucocytes, plaquettes, cell endotheliales
beta: stase intestinale: necrose: enterite necrotique
epsilon: augmente permeabilité vasculaire GI
iota: augmente permeabilité vasculaire et activité necrotique

31
Q

clostridium perfringens reservoir

A

sol, eau contaminé par scelles

32
Q

clostridium perfringens clinique

A 
gangrene gaseuse par activité metabolique rapide: necrose musculaire: choc: mort
enterite necrosante (type C): douleur abdo, diarhée+sang, vomissement, ulceration, perforation
empoisonement alimentaire (type A): crampes abdo, diarhée
33
Q

clostridium tetani microbio

A

anaerobe, gram +, non fermenteur

34
Q

clostridium tetani facteur virulence

A

formation spores, production toxines: tetanolysine, tetanoplasmine (inactive protéines relachent neurotransmetteurs inhibiteurs)

35
Q

clostridium tetani clinique

A

paralysie spastique

generalysé: touche SNC, sudation, deshydratation, sourire sardonique

36
Q

clostridium tetani reservoir

A

humain et animal et omnipresent sols fertiles

37
Q

clostridium botulinum microbio

A

gram+, anaerobe, spores

38
Q

clostridium botulinum clinique

A 
paralysie flaccide (inactive proteines qui relachent acetylcholine)
alimentaire(vision floue, bouche seche, 0 fievre, douleur abdo), infantile, lesionnel
39
Q

clostridium botulinum reservoir

A

sols et echanitillons d’eau

40
Q

clostridium botulinum facteurs virulence

A

a une protéine qui facilite passage dans GI

41
Q

clostridium difficile microbio

A

gram+, anaerobe

42
Q

clostridium difficile facteurs virulence

A

spores, toxine: enterotoxine(effet chimotaxique et interuption jonctions cell) et cytotoxique(depolymerisation actine)

43
Q

clostridium difficile clinique

A

diarhée, colite pseudomembranaires(crampes abdo, diarhée profuse, fièvre)

44
Q

bacteroide fragilis microbio

A

gram-, anaerobe, flore normale, catalse +

45
Q

bacteroide fragilis facteurs virulence

A

adhesine ou fimbriae, production toxine

46
Q

bacteroide fragilis clinique

A

infection tractus respiratoire, abces cerebraux, infetion intra-abdominale, gynecologiqie, tissus mous, peau, gastroenterite

47
Q

pseudomomas aeruginosa identification

A

vert, sent raisin, gram-, aerobie stricte, beta hemolyse, oxydase+

48
Q

pseudomomas aeruginosa reservoir

A

humide

49
Q

pseudomomas aeruginosa facteur virulence

A

exotoxine s et t rearange actine(invasion tissulaire), adhesine, exotoxine A(interompt synthese protéines), pyocianine(forme peroxyde d’hydrogene), pyoverdine(lie le fer), serine protease(degrade elastines+systeme complement),phospholipase C(hemolysine)

50
Q

pseudomomas aeruginosa clinique

A

infection pulmonaire, peau, tissus mous, tractus urinaire, oreilles, yeux

51
Q

staphylocoque microbio

A

en amas, coagulase+ catalase+

52
Q

streptococcus classés selon

A

beta-hemolitique: groupes de lancefields (ABCFG)

alpha et gamma hemolytique: groupe viridians:propriété biochimiques/physiologique

53
Q

streptococcus beta-hemolytique

A

B:zone etroite hemolyse, antigene B, pneumonie
CFG: antigene C/F/G ou non specifique, cause abces

54
Q

streptococcus groupe viridians

A

anginosus: abces
mitis: endocartite
salivarius: endocartite
mutans: carrie dentaire
bovis: meningite

55
Q

enterococcus

A

antigene du groupe D, colonise tube GI, infection conduit urinaire, peritonite, endocartite

56
Q

staphylococcus aureus microbio

A

gram+, catalase+, coagulase+, jaune

57
Q

staphylococcus aureus pathogenese

A

proteine a se lie immunoglobuline
AGR est quorum sensing et produit surface adherente(slime layer) ou enzymes hydrolitiques
coagulase libre active thrombine et lié active coagulase reacting factor convertit fibrinogene en fibrine (localise et protege)

58
Q

staphylococcus aureus toxines

A

cytotoxines: lyse membrane
exfoliante: detruit adhesion entre cells (0inflammation)
enterotoxine:(associé bouffe) superantigene, inflammation, vomissements
TSST: superantigene:relache cytokines: perce barriere mucosal: effet systemique, choc hypovolemique: MOF
enzymes: hyaluronidase, fibrinolysine, lipase, nuclease

59
Q

streptococcus pyogene facteur virulence (sans toxines)

A

protéine M bloque liason C3b (facteur H le degrade)
semblable a M se lie a anticorps
C5a peptidase: inactive C5a
adhesion aux cellules hotes

60
Q

streptococcus pyogene toxines

A

streptococcus pyogene exotoxine: superantigene responsable fascite necrosante
streptolysine S et O: lyse leucocytes, plaquettes et erythrocytes
streptokinase: clive fibrine et fibrinogene

61
Q

streptococcus pyogene clinique

A

fascite necrosante: cellulite:bulles:gangrene:symptomes systemiques: multiorgan failure: mort
(infection tissus sous cutanés profond qui se propage)
choc toxique: commence par inflammation et infection des tissus, symptomes vagues (fievre, malaise), MOF

62
Q

streptococcus pneumoniae microbio

A

vert si alpha-hemolyse (aerobe) si betahemolyse (anaerobe)

63
Q

streptococcus pneumoniae facteur virulence

A

adhesine (peut migrer aux poumons), produit pnemolysine(crée pore cell epitheliales, active systeme complement classique) et protéase secretrice IgA(empeche emprisonnement dans mucus), produit peroxyde d’hydrogene et phosphorylcholine, se lie a plaqueete activating factor et entre dans cell, ce qui facilite propagation

bactéries (1 decks)

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