AW assessment - Sem 1 Flashcards

1
Q

What does LEMON stand for ?

A
L = Look
E = Evaluate
M = Mallampati
O = Obstruction / Obesity
N = Neck Mobility
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2
Q

What do you assess in L of LEMON? How do you score it ?

A

L = Look = Trauma, Large Teeth, Large Tongue, Beard.

Score= 1 pt each, max score for this category is 4

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3
Q

What does E from LEMON stand for ? How do you score it ?

A

E = Evaluate, 3-3-2refers to :
a) Mouth Opening - 3 Fingers (patient’s fingers), any less than 3 = 1 point
b) Hyoid-Mental Distance (from hyoid to bottom tip of chin), any less than 3 = 1 point
c) Hyoid-Thyroid Distance (space between the hyoid and thyroid, any less than 2 = 1 point
Max score is 3

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4
Q

What does the M in LEMON stand for? How do you score it ?

A

M = Mallampati Score.

If Grade I or II, that’s good - score 0. If grade III or IV, not good, add 1 point

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5
Q

What does O in LEMON stand for ? How do you score it ?

A

O = Obstruction (any obstruction, ie vomit, swelling, tumors) add 1 point.

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6
Q

What does N in LEMON stand for ? How do you score it ?

A

N = Neck Mobility (anything that decreases Neck Mobility add 1 pt)

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7
Q

What is the max LEMON score possible ?

A

10

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8
Q

Above what LEMON score do we classify it as a “difficult airway” ?

A

above 3

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9
Q

How does an unconscious patient affect our Airway Assessment ?

A

Unable to properly do a Mallampati score, as we need the patient to be sitting up and saying “Ahhhhhhh”

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10
Q

If LEMON assesses for ease / difficulty of ETT insertion, what does MOANS assess for ?

A

Ease / Difficulty of using BVM / Mask Seal

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11
Q

What does MOANS stand for ?

A

M - mask seal (beard, trauma, facial structure)
O - Obesity / Obstruction
A - Age (>55 yrs)
N - No teeth
S - Stiff Lungs (compliance) - sometime add “C-Spine”

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12
Q

Can you differentiate between compliance and resistance using a BVM ? If so, how? If not, why ?

A

Unless it’s a fully obstructed Airway, and nothing goes in - - - in which case it’s an obstruction / resistance…..you can’t tell the difference.

Compliance refers to the ease of which the lungs inflate.

All you are going to feel on the BVM is need to apply more or less pressure to ventilate. Unable to tell is that is due to Resistance or Compliance.

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13
Q

How does a BVM PEEP valve work ?

A

A positive end expiratory pressure (PEEP) valve may be used during BVM to improve oxygenation. PEEP can increase alveolar recruitment and thus oxygenation if oxygenation is compromised even with 100% oxygen due to atelectasis. PEEP has also been shown to prevent lung injury.

PEEP is the pressure above atmospheric pressure measured in the alveoli at end expiration. It is one of the first ventilator parameters set and would typically be “dialed in” to between 3 and 5 cmH2O, sometimes referred to as, “physiologic PEEP.”4 This positive, end expiratory, pressure serves to prevent collapse of the alveoli at end expiration as well as to prevent repeated opening and closing of the alveoli, which is thought to cause ventilator-induced lung injuries. PEEP may also serve to recruit already collapsed alveoli, reopening them so they can participate in gas exchange.5 The primary use of PEEP has typically been to improve oxygenation. PEEP can also be applied when using a bag-valve mask by attaching a PEEP valve to the bag and selecting the level of PEEP desired (see illustration). Some bag-valve-masks are manufactured with integral PE

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14
Q

What is the pneumonic for assessment of airway for insertion difficulty of a SGA?

A

R - restricted mouth opening
O - Obstruction / Obesity
D - Disrupted / Distorted Airway
S - Stiff Lungs / C-Spine

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15
Q

A 23 yr old healthy patient, with no trauma with regular teeth.
She can fit 3 fingers in her mouth, 2 fingers between her hyoid and chin tip, 2 fingers between hyoid and thyroid
Score the LEMON

A

L - no trauma, regular teeth, no beard on pic, tongue looks normal - - - so 0
E - Mouth 3+, H-C 2, H-T 2 —– so 1
M - according to photo looks to be class I - – so zero
O - no obstruction or obesity noted - - - so zero
N - no trauma and healthy - expect good mobility, so 0

Total Score : 1
We do not expect this to be a difficult intubation

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16
Q

A patient can fit 2 fingers in mouth, has large teeth and a beard. They have 2 fingers for H-C, 1 finger for H-T. You can only see the hard palet of the mouth on Ahhhh. No obstructions noted. Good neck mobility.

Score LEMON

A
L = large teeth , beard (no trauma or large tongue noted) - -- score 2
E = 3,2,1 (instead of 3,3,2) - - score 2
M = sounds like a class IV - - -score 1
O = no obstruction / obesity noted - - score 0
N = good neck mobility - - score 0

Total LEMON score = 5

We expect this to be a difficult intubation

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17
Q

What are Ornge’s “Go To” Medications for RSI?

A

Ketamine for Induction

Rocuronium for NeuroMuscularBlocker (NMB / paralytic)

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18
Q

If not using Ketamine as an induction agent what else might you use, typically ? What is one key risk of this medication ?

A

Propofol

Drop in BP

19
Q

Name two common Paralytics used in RSI, with the most usual one listed first.

A

Rocuronium

Succinylcholine

20
Q

Why might we use Succinylcholine ?

A

Paralytic / NMB - - short acting; so in some cases where you just want to paralyze for easy intubation and you want the patient to use their inherent Resp soon after, OR - - - It you have a difficult intubation with questionable back ups, and need it to reverse in case you don’t get the tube.

21
Q

What is the difference between Facilitated Intubation and Rapid Sequence Intubation ?

A

Facilitated used just sedation, but no neuromuscular blockade (paralytic)

RSI uses both sedation (induction) and a neuromuscular blockade (most often Ketamine and ROC)

22
Q

What does SALAD stand for in terms of airway management ?

A

Suction Assisted Laryngescopic Airway Decontamination (we should use it with EVERY intubation according to Britton)

23
Q

What is Decanto?

A

It’s a large bore suction catheter (used in SALAD) instead of using a Yanker (Yanker useless in resuscitation for anything more than light spit)

24
Q

What does APENIC OXYGENATION mean ?

A

It means using both a 15 LPM NC and an NRB at 15 LPM to pre-oxygenate patient prior to intubation

25
Q

Why do you have to use a regular NC for APNEIC OXYGENATION and not the one that is part of Nasal ETCO2 ?

A

The ETCO2 NC are designed in such a way that anything more than 5 LPM does not go into patient, it goes into room. (Small holes in it so that it can measure ETCO2)

26
Q

When suctioning, say using SALAD method, how do we avoiding causing desaturation if the suction is staying in the mouth ?

A

Park it in the esophagus, to avoid sucking air out of Airway

27
Q

What does “DeNitrogenation” mean in terms of AW management ?

A

Denitrogenation involves using oxygen to wash out the nitrogen contained in lungs after breathing room air, resulting in a larger alveolar oxygen reservoir

28
Q

Case Study: Male mid 50’s , GCS 15, RR 32, Sat 88% with NRB, BP 110/70 , MAP 83, HR 121, Obese

Discuss key management considerations and approach.

A

Concerns: low BP, given size and age - - - and low 02 even on NRB…..so, will need to be “adequately resuscitated” prior to Intubation before we (if we) do that. Pressors ? Inotropes ? Fluids ? - - - - we will also need to increase 02 sat (push up the curb)
Shock Index can help guide how worried to be about BP. (Remember SHOCK INDEX = HR / SBP = 121 / 110 = 1.1
Assume occult shock is SI > 1.0, or if change in SI > 0.1 (normal SI is 0.7) - different values for peads.

Tx - Pressors, Intoropes, Fluids ; start with non-invasive High Flow NC or CPAP, reassess…If going to RSI - - - > 15 LPM x 2 (NC and NRB) then RSI.

If Airway pressures are high / compliance low - – will need PEEP and high FiO2 to maintain Oxygenation (on Non Invasive)….your non-invasive settings will guide your Vent settings.

29
Q

What is DSI and what does that entail ?

A

Delayed Sequence Intubation.

Used typically for say agitated patients that aren’t ventilating well. So you sedate them with something that doesn’t affect their respirations, then you can pre-oxygenate them (push them up curb) , wash out nitrogen (increase O2 reserve) before Intubating.

Then - you paralyze and intubate.

30
Q

Can’t get a good view to intubate, what can you do to trouble shoot ? (5)

A
BURP (patient's right ; intubator guides partner's hand)
Lift Head (30 degrees? ear to sternum ? lift head!)
Lift More on Blade (up and out)
Partner can try a Jaw Thrust
Readjust Blade Depth (too little or too far into the velecula)
31
Q

What does ELM mean when it comes to intubation ?

A

ELM = External Laryngeal Manipulation = BURP

32
Q

Case Study: 48 yr old male, 90 kg, massive hemoptysis, SBP =84, MAP 57, HR 56, RR 22, sat 93% on NC at 5 LPM, GCS 12-13, Dx: Esophageal Varices.

What are your concerns, approach and treatment ?

A
  • AW bad (blood) ; need to secure AW
  • BP and O2 low, SI = 56/84 = 0.666 which is technically normal, but still bleed and AW issue and low BP (why does he not have higher HR?? what else is going on ? on BetaBlockers ?)
  • Needs intubation now before he gets worse, but needs significant resuscitation first
  • LEMONS score based on Obstruction (blood) and unobtainable Mallampati will be >3 so expect difficult intubation
  • SALAD very important here because of blood
  • If you can treat esophageal varices in hosp first, do that - - - Doc does this, if they are able to (pressure / pinch off)
  • Treat with Vasopressin (decreased portal venous blood flow) and Beta Blockers (both are mainstays for this condition)….although watch the HR if HR is already 56.
  • balance BP increase vs Bleed, Oxygenate +++, then RSI
  • might want to hold off on NMB, cause if you can’t get tube - because messy airway.,…..might be a problem….
  • SGA might be an option, and can use NG port to suction out
  • only low dose induction with ketamine, don’t want to blunt SNS based on vitals,
33
Q

Does Ketamine potentiate Hypertension or Hypotension?

A

MDSO - says HYPERtension (no mention of Hypotension) and this is somewhat accurate at analgesic doses

However, when SNS drive is playing a significant role in maintaining hemodynamics, the sedation effect may blunt their SNS drive, so keep that in mind….it could also cause HYPOtension (but this is not in MDSO)

34
Q

A patient has low BP, say from a large hemorrhage. What must you be mindful of when patient is put on a Vent ?

A

Once you start BVM or VENT, you’ve moved from a negative pressure system to a positive pressure system. As such intra-thoracic pressures will increase - - - which will drop Preload - - - even less blood in R ventricle - - - so just mechanical ventilation can affect hemodynamics. Sick, non compliant lungs that need higher pressures also have a similar concern over increased intra-thoracic pressures decreasing Preload.

35
Q

Case Study: 78 yr old Female, Obese, C/O CP. HR 45, SBP 85, poor wave form on sats.

What are your concerns ? How would you approach ?

A
  • Pt is very unstable (HR and BP and CP and ? O2)
  • Will need significant resuscitation prion to intubation
  • Both HR and BP are low - - - so want a pressor that works on both alpha and beta - - - EPI is a good choice here. (Epi more B effect than NE , but each has both A and B) - so in this case EPI better than NE (until HR is at least about 60)
  • Fluids, but be careful…..frequent reassess q 250 cc, so you don’t overload, we’re unsure about the heart’s ability…..otherwise fluid will help with Preload and Stretch Response
  • If that not working - - - this is really a Symptomatic Bradycardia call…..you just have more tools now.
  • It’s not mentioned in case study, but assuming no AV block, can try Atropine as well (maybe before EPI)
  • At the end of the day, if none of this stabilizes, we’ll have to PACE
  • be careful with Procedural Sedation, don’t want to blunt SNS
  • Try another site for the O2 sat - see if that fixes it
  • If that isn’t the problem, and you need to increase O2, start with Non Invasive, but get ready to Tube as well, as she is quite sick
36
Q

CASE STUDY
23 yr old, Status Epilepticus, refractory to benzo, still seizing even after dose of dilantin, when you arrive.
HR 110, BP 134, RR 28 but ineffective, Sats 95% on Venturi Mask.

What are your concerns ? What is your approach ?

A

She’s been seizing for a while…..need to RSI asap! (this is the main point)

  • Paralytic will terminate seizure and help with intubation (stops convulsions but not seizure in brain)
  • Stopping convulsions will decrease metabolic demand
  • ABG ? Potential Metabolic Acidosis (lactic acid), but pCO2 will likely also be high (poor ventilation)
  • Could benefit with Amp of BiCarb
  • When paralyzed, the (brain) seizure might stop due to the induction agent (Ketamine, Propofol, Midaz) - but we wont’ really know - - can —-take a look at HR and see if it decreases as a bit of a clue.
  • Once intubated and ventilated then also manage the pCO2 / ETCO2 to help pH
  • Consider reversible cause of seizure (sugar? overdose?)
  • Usually Status Seizure is due to Neuro issue (disorder, trauma, swelling)
37
Q

What is the induction dose for Ketamine (beyond semester 1 as of Jan 2022, but fits in this sections.

How is it supplied ?

A

Pre-intubation dose: Induction Dose: 1-2 mg/kg IV over 30 seconds (or titration dose of 10-30 mg IV q 60 s, to goal of 2mg/kg within 5 min)

Supplied 500mg/50mL (10mg/mL), 100mg/2mL (50mg/mL), so say 70 kg, 2 mg/Kg - - - 140mg (14 mL of 500/50) or, just under 3 mL of the 50mg/mL ((((make sure you know which concentration you are working with)))

38
Q

What is the RSI dose of ROC ?

How is it supplied?

A

1.2 mg/kg IV direct induction dose, then 0.3 mg/kg q 20 min PRN

Supplied as 50mg/5mL

Say 70 kg, so 1.2 x 70 = 84mg. so, 84mg / 50mg/5mL = (84 *5) /50 = 8.4 mL for initial induction.

39
Q

What is the difference between: Non Depolarizing Neuro Muscular Blocker .,…and a Depolarizing Neuro Muscular Blocker ? Give examples of each. What reverses them ?

A

depolarizing muscle relaxants act as ACh (Succinylcholine) receptor agonists and cause depolarization, BUT are not affected by AntiCholinesterase…and remain bound, so no further action potential can be triggered (no Antidote, but lasts only 4-8 min if you don’t add more)

whereas nondepolarizing (ex: Rocuronium) muscle relaxants function as competitive antagonists of ACh receptors, they can be outcompeted by more Ach which can be achieved by using an AntiCholinesterase. (ex: Neostigmine ad Edrophonium) - Duration 22-67 minutes. Thankfully we have an antidote.

40
Q

How do you reverse a NON-depolarizing NMB ?

A

An anticholinesterase. Blocks ACh-ase, therefore Ach increases , and outcompetes the NON-depolarizing NMB (competitive agonist for Ach Receptor at NeuroMuscular Junction), example: Neostygmine

41
Q

How do you reverse a DEPolarizing NMB ?

A

You wait, no antidote. Duration 4-8 min.

42
Q

What’s the nmeumonic to assess for Surgical Airway ? (CRIC)

A
SHORT
 Surgery
 Hematoma
 Obesity
 Radiation
 Tumor
43
Q

Explain CRASH intubation vs RSI ?

A

CRASH is when patient is crashing, and you haven’t had time to prepare (resuscitation (fluids, pressors) or pre-ox)

Should never really be doing CRASH……throw in an SGA and stabilize

Shock Index > 1 (HR/SBP) increased risk of crashing