autism/ADHD Flashcards

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1
Q

hyperactivity

A

a higher than normal level of activity, excessive fidgetiness & not remain seated

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2
Q

impulsivity

A

the act of reacting to a situation without considering the consequences

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3
Q

DMS-5 criteria for ADHD (both types)

A

an ongoing pattern of inattention and/or hyperactivity & impulsivity that interferes with normal functioning or development

  • symptoms present before age 12
  • symptoms present in at least 2 settings
  • symptoms reduce the quality of educational, social or occupational ability
  • symptoms do not occur during another psychotic disorder and are not better explained by another mental disorder
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4
Q

DSM criteria for inattention ADHD

A

at least 6 symptoms for at least 6 months
- not paying attention or making careless mistakes
- ignores instructions
- difficulty maintaining attention
- difficulty organising
- forgetful
- doesn’t listen when spoken to directly

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5
Q

DSM criteria for hyperactivity/impulsivity ADHD

A

at least 6 symptoms for at least 6 months
- high level of fidgeting
- unable to engage in activities quietly
- excessive talking
- blurts out an answer before the question is finished
- has difficulty awaiting their turn
- interrupts or intrudes on others frequently

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6
Q

prevalence of ADHD

A

5% of school-age children & 2.5 % of adults
more common in boys
symptoms are not sex specific – but girls mostly have inattention & boys hyperactivity

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7
Q

consequences of ADHD

A
  • prone to temper outbursts, frustration, bossiness, stubbornness, changeable moods, and poor self-esteem.
  • academic achievement is impaired = conflict with teachers and family.
  • associated with peer rejection and accidental injury.
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8
Q

comorbidity of ADHD

A
  • oppositional defiant disorder (ODD) or conduct disorder
  • learning disabilities – e.g., dyslexia
  • depression & anxiety
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9
Q

bio factors of ADHD

A
  • bio causes more important than psychological
  • one of the most heritable psychiatric disorders

brains of children with ADHD are smaller and develop more slowly.
- main areas affected = 4 lobes
- deficits in executive functioning & difficulty inhibiting responses - normally controlled by frontal lobes.

problems with dopamine transmission
- struggle with rewards, especially long-term rewards – related to inhibiting behavior

some symptoms can become less visible with adulthood bcuz the brain develops – mostly hyperactivity & impulsivity, inattention stays

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10
Q

psychological factors of ADHD

A

appears to run in families = beyond the fact that there is a genetic component to the disorder

interactions between parent and child – if parent has ADHD they will have trouble dealing with a child who also has it
- parent wouldn’t have patience = makes child’s symptoms worse
- parents accidentally reinforce behavior – child learns that this behavior gets attention from parents (even if its negative) = do it more often

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11
Q

prenatal factors for ADHD

A

pregnant mothers smoking greater than 10 cigarettes per day = more likely to give birth to children with ADHD
- nicotine affects dopamine functioning

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12
Q

psychostimulants as treatment for ADHD

A

= medications that increase CNS activity.
paradoxical effect = psychostimulants lead to restless, even frenetic, behavior when abused – is a MYTH

most commonly prescribed psychostimulants:
Ritalin, Dexedrine, Cylert, and Adderall.

psychostimulant alternatives:
- strattera: NE reuptake inhibitor - the only non stimulant medications approved – severe side effects
- clonidine: can lead to decreases in aggressive behavior,
- antidepressants- may affect ADHD symptoms directly for unknown reasons

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13
Q

short-term & long-term effects of psychostimulants on ADHD

A

Short-term
Hyper/impulsive: dramatic improvements - less active and more focused - fewer social problems
inattention: more work completed, but no change in grades or standardized tests scores

Long-term
no demonstrated benefit for either

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14
Q

article - most appropriate intervention for adhd

A
  • preschoolers –> parent training
  • school-age w moderate impairments –> group parent training programmes & classroom behavioural interventions
  • school-age with severe impairments –> combined with stimulant medication
  • middle school/adolescent –> multimodal interventions + social skills training
  • adults –> stimulant medication, CBT
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15
Q

autism spectrum disorder

A

a spectrum of developmental impairments and delays that include:
- social & emotional disturbances
- intellectual disabilities
- language and communication deficits
- stereotype or self-injurious behavior patterns

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16
Q

triad of impairments (autism)

A
  1. lack of reciprocal social interaction
  2. impairments in communication
  3. impairments in imagination & flexibility of thought
17
Q

triad of impairments - lack of reciprocal social interaction

A
  • trouble initiating the interaction
  • non-verbal communication
18
Q

triad of impairments - impairments in communication

A
  • trouble maintaining eye contact
  • echolalia = the immediate imitation of words or sounds that have just been heard
  • pronoun reversal = when an individual refers to themself as ‘he’, ‘she’ or ‘you’,
19
Q

triad of impairments - impairments in imagination & flexibility of thought

A
  • need for sameness
  • restricted, repetitive and stereotyped patterns of behaviour and interests
  • specific and detailed interests
  • stereotyped (self-stimulatory) body movements
20
Q

prevalence of ASD

A
  • 1-2% of population
  • 80% males
  • increased diagnosis in recent years – due to extension of criteria
21
Q

empathizing-systemizing theory

A

a theory of the social and communication difficulties experienced by individuals w autism

  • superior skills in systematizing & finding patters – how they navigate the world
22
Q

consequences of ASD

A
  • trouble socializing & social interaction issues
  • difficulty inhibiting complex behaviors
  • trouble with planning & organization
23
Q

genetic factors for ASD

A
  • heritable mostly form the fathers gene
  • multiple genes
  • phenylketonuria (PKU)
  • fragile X syndrome
24
Q

prenatal factors for ASD

A

teratogens - drugs (thalidomide and valproate)
bleeding
immune functioning

25
Q

brain abnormalities for ASD (causes)

A
  • abnormalities in dopamine & serotonin NTs
  • brain is generally bigger than average (18m – 5y), except limbic system is smaller
  • abnormalities in pre-frontal cortex and amygdala = problems w ToM & exec function
26
Q

treatments for ASD

A

antipsychotics - reduce:
- repetitive and stereotyped behaviours
- levels of social withdrawal
- aggression and challenging behaviour

parent-implemented early intervention = using parents as effective trainers to teach children with intellectual disabilities basic self-help and communication skills

27
Q

comorbidity of ASD

A

savant syndrome = extraordinary proficiency in one isolated skill in individuals with multiple cognitive disabilities
closely linked to ASD and Asperger’s syndrome