ATYPICAL GENDER DEVELOPMENT Flashcards
What is gender dysphoria?
When a person experiences discomfort or distress because there is a mismatch between their sex assigned at birth and their gender identity. This is also the clinical diagnosis for someone who doesn’t feel comfortable with the sex they were assigned at birth.
How does DSM-4 (IV) classify atypical gender development?
classifies atypical gender development by the clinical label gender identity disorder (GID), a condition where individuals experience a mismatch between their biological sex and the sex they ‘feel’ they are
How does DSM-5 classify gender atypical gender development?
The more recent DSM-V now refers to GID as gender dysphoria in order to remove the damaging label of people with the condition as ‘disordered’. According to DSM-5, gender dysphoria is diagnosed when someone has experienced a psychological distress arising from this mismatch between sex and gender for atleast 6 months. It causes significant impairment in social, occupational and other important areas of functioning.
What is the difference between gender dysphoria and androgyny?
Dysphoria is not being happy with the gender you are and wanting to change - androgyny is having both masculine and feminine characteristics
DSM diagnosis of gender dysphoria
- Experience will affect ability to function in everyday life
- No biological disorder should occur at the same time
- Must experience ongoing identification with the opposite sex
- Feel a strong sense of discomfort with their own biological sex
What are the 2 explanations for gender dysphoria?
Biological explanations and Social-psychological explanations
What are the biological explanations for gender dysphoria?
Brain-sex theory
Genetic factors
What is brain sex theory?
This suggests that GD has a basis in brain structure - the bed nucleus of the stria terminalis (BST).
What does brain sex theory suggest?
Brain-sex theory suggests that dysphoria is caused by specific brain structures that are incompatible with a person’s biological sex.
Particular attention has been paid to those areas of the brain that are dimorphic (take a different form in males and females)
Ning Zhou (1995) studied the stria terminals which are assumed to be fully developed at age 5 and around 40% larger in males than females
Explain the bed nucleus of the stria terminalis (BST)
- this structure is involved in emotional responses and in male sexual behaviour in rats
- Kruijver found this area is larger in men than women and it was also found to be female sized in transgender females.
- Suggests people with GD have a BST which is the size of the gender they identify with rather than corresponding to their sex.
- This dimorphism in the BST (having 2 forms) fits with the reports from transgender people who say they feel they were born in the wrong sex. (ZHOU)
- Later research by Kruijver replicated this finding, this time by counting number of neurons. Again six male-to-female transgender individuals showed a ‘sex-reversed’ identity pattern with average neuron numbers in the female range.
What have post-mortems shown?
- In post-mortems of six male-to-female transgender individuals, the stria terminals were found to be a similar size to as heterosexual women
- This is supported by Kruijiver et al (2000) who focused on the number of neurons in the stria terminals rather than the volume.
- Again, the six transgender individuals showed a similar number of neurons in the stria terminals to those found in heterosexual women
- They concluded that the stria terminals provides evidence for a neurobiological basis of gender dysphoria.
What does research also show? (genetic factors)
Research shows GD may be genetic as there are high concordance rates between identical twins.
Who provides research support for genetic basis of GD?
Coolidge
Heylens
What did Coolidge do?
Coolidge (2002) assessed 157 twin pairs (96 MZ and 61 DZ) for evidence of gender identity disorder using the clinical diagnosis of criteria in DSM-4
What did Coolidge find?
62% of these cases said to be accounted for by genetic variance. This suggests there is a strong heritable component to GID