Atrial Fibrillation Flashcards

1
Q

how would the AF rhythm be described?

A
  • irregularly irregular
  • narrow QRS complexes
  • absent p waves
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2
Q

describe common causes of AF?

A
  • idiopathic (12%)
  • hypertension
  • coronary heart disease
  • valvular heart disease
  • cardiomyopathy
  • Respiratory disease (lung cancer, COPD, PE, pulmonary hypertension)
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3
Q

causes of acute AF?

A
  • alcohol
  • infection
  • surgery
  • pericarditis
  • MI/ PE
  • hyperthyroidism
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4
Q

what are the symptoms?

A
  • palpitations
  • syncope
  • breathlessness
  • fatigue
  • light headedness
  • stroke/ TIA
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5
Q

investigations?

A
  • ECG
  • CXR
    Bloods:
  • TFT’s
  • FBC
  • U&E (kidney function/ potassium)
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6
Q

when should an echo be done?

A

if the patient is <50 years old

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7
Q

How is paroxysmal AF managed?

A
  • Beta- blocker PRN (e.g.atenolol 50-100mg)
  • ” Pill in the pocket” (flecainide)
  • remove known precipitants ( caffeine /alcohol)
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8
Q

when would you not prescribe the pill in the pocket in paroxysmal AF?

A
  • systolic BP >100
  • resting HR of >70
  • LV dysfunction
  • valvular or ischaemic Heart disease
  • frequent paroxysms
  • pill in the pocket only useful if infrequent and no underlying causes as above
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9
Q

what is the definition of paroxysmal AF?

Persistent?

Permanent?

A
  • spontaneous termination within 7 days (most often within 48 hours)
  • non self-terminating, lasting longer than 7 days (even if terminated, still in persistent AF)
  • long standing (over a year)
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10
Q

explain the physiology behind pulmonary vein triggers?

A
  • pulmonary veins have excess muscle with same SA node embryological tissue therefore can synapse and cause arrythmias.
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11
Q

How do you broadly manage AF?

A
  • rate control
  • rhythm control
  • stroke/ bleeding risk
  • treat the causes
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12
Q

How would you rate control?

A

Rate control:
- beta-blocker (cardiospecific, NOT sotalol)

  • rate limiting calcium channel blocker (diltiazem or verapamil)
  • Digoxin (only in non-paroxysmal AF, only good for sedentary patients)
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13
Q

how is stroke/ bleeding assessed?

A
  • CHADS- VaSc score
    (in paroxysmal/ permanent/ persistent AF)
  • HAS-BLED to assess bleeding risk in those being offered anti-coagulation
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14
Q

who should be offered anti-coagulation? and what with?

A
  • those with CHADs-VASc score of 2 or above
  • (rivaroxaban/ apixaban)
    warfarin
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15
Q

when should you not offer rate control?

A
  • AF with reversible cause
  • heart failure caused by AF
  • new onset
  • atrial flutter with ablation
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16
Q

how do you rhythm control?

A
  • cardioversion
  • beta- blocker
  • dronedarone (2nd line)
17
Q

when should flecainide not be given?

A
  • people with known structural or ischaemic heart disease
18
Q

what is the benefit of catheter ablation?

A
  • electrical isolation of the pulmonary veins

- prevent triggers and drives

19
Q

what pulse would you like the hr at?

A

<110 bpm