ATRIAL DYSRHYTMIAS Flashcards

1
Q
SUPRAVENTRICULAR TACHYCARDIA (SVT)
(EKG CHARACTERISTICS)
A

P wave/PRI- Unable to distinguish p waves from t waves/UTD because there are no distinguishable p waves.

QRS: Narrow (.06-.10 seconds)

RATE: Ventricular rate is 150 to 250 beats per minute; Unable to determine atrial rate because there is no distinguishable P wave

RHYTHM: Ventricular rhythm is REGULAR; Unable to determine atrial rhythm because there is no distinguishable P wave

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2
Q

SVT NURSING INTERVENTIONS

A
Vital Signs (including SpO2)
Assess for shock (cool clammy skin, ALOC)
Decrease HOB for hypotension
Oxygen if hypoxic 
Assess for chest pain
IV access
12 Lead EKG
VAGAL MANEUVERS
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3
Q

SVT PHARMACOLOGIC AND ELECTRIAL INTERVENTIONS

A

Pharmacologic:
Adenosine
6 mg rapid IVP, follow with NS flush
If SVT does not convert in 1-2 minutes, give 12 mg rapid IVP, follow with NS flush

Electrical:
Cardioversion
Narrow regular QRS (.06-.10 sec) 50-100 Joules

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4
Q

PREAMATURE ATRIAL COMPLEXES (PACs)

EKG Characteristics

A

Early (premature) P waves

Positive (upright) P waves (in lead II) that differ in shape from sinus P waves

The early P wave may or may not be followed by a QRS complex.

The PR interval of the PAC may be normal or prolonged, depending on the prematurity of the beat. The QRS complexes associated with a PAC are typically identical or similar in shape and duration to those of the underlying rhythm since the impulse is conducted normally through the AV junction, bundle branches, and ventricles.

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5
Q

PAC common causes (6)

A
Hypokalemia
Hypomagnesemia
Hypoxia
Caffeine 
ETOH
Medication
(sympathomimetics)[epinephrine]
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6
Q

PAC NURSING INTERVENTIONS (4)

TREATMENT AND RISKS

A
Nursing interventions:
   Palpate pulse
   Chronic or new onset?
   Assess for cause
   Trend

Treatment:
Treat possible cause

Risks:
Patient at higher risk for atrial fibrillation

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7
Q
Atrial Fibrillation (A-FIB)
(EKG Characteristics)

(fibrillatory waves)

A

P wave: No distinguishable P waves, only fibrillatory waves, which are not the same as P waves

PRI: No distinguishable P waves so no PRI.

QRS: Narrow (.06-.10)

RATE: Controlled A-fib has a ventricular rate <100; A-fib with a RVR (rapid ventricular rate) >100 (sometimes called “uncontrolled” A-fib.) There is no atrial rate because there are no distinguishable P waves

RHYTHM: Ventricular rhythm is irregularly irregular; There is no atrial rhythm because there are no distinguishable P waves

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8
Q

A-FIB NURSING INTERVENTIONS (7)

rate dependent

A
Nursing: *RATE DEPENDENT*
   Vital Signs (including SpO2)
   Assess for shock (cool clammy skin, ALOC)
   Decrease HOB for hypotension
   Oxygen if hypoxic 
   Assess for chest pain
   IV access
   12 Lead EKG (new onset)
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9
Q

A-FIB Pharmacologic and Medical Interventions

A

Pharmacologic:
Beta-blockers
CCBs

Medical Interventions:
Ablation

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10
Q

ATRIAL FLUTTER
(EKG CHARACTERISTICS)

(FLUTTER WAVES/SAWTOOTH WAVES/PICKET FENCE WAVES)

A

P waves/PRI: No distinguishable P waves, only flutter waves. Since there are no distinguishable P waves= No PRI

QRS: Narrow (.06-.10 seconds)

RATE: Atrial rate= NONE because there are no distinguishable P waves. Atrial flutter waves occur about 300 times per minute. 
Ventricular rate = Varies, depending on the 
 conduction block (flutter wave) and is usually not over 100 bpm. (1 flutter wave= 1 big box)

RHYTHM: Atrial rhythm = NONE because there are no distinguishable P waves.
Ventricular Rhythm= Regular or Irregular

Conduction Blocks: Atrial flutter waves to QRS complexes can be 1:1, 2:1. 3:1, 4:1, 5:1, or can have a variable block. = irregular ventricular rhythm. (can hide behind the QRS)

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11
Q

ATRIAL FLUTTER NURSING INTERVENTIONS

A
Nursing: *Rate dependent*
   Vital Signs (including SpO2)
   Assess for shock (cool clammy skin, ALOC)
   Decrease HOB for hypotension
   Oxygen if hypoxic 
   Assess for chest pain
   IV access
   12 Lead EKG (new onset)
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12
Q

Atrial Flutter Pharmacologic and Medical Interventions

A

Pharmacologic:
Beta-blockers & CCBs

Medical Interventions:
Ablation

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