Atrial and Ventricular Dysrhythmias Flashcards

1
Q

Atrial Dysrhythmias: Types (2)

A

atrial flutter
atrial fibrillation

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2
Q

Atrial Flutter

A

-originates in the AV node (overrides the SA node and takes full control)
-reentry impulse that is REPETITIVE & CYCLIC
-regular atrial rhythm with an ATRIAL rate of >250 bpm
-ventricular rate is SLOWER
-P wave is classical “sawtooth” appearance
-QRS usually narrow
-May be 2:1, 3:1, or 4:1 (P waves : QRS)

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3
Q

Atrial Flutter: Causes (8)

A

Coronary heart disease
Cardiomyopathy
Heart valve disease
Congenital heart disease
Inflammation of the heart (such as myocarditis)
High BP
Other conditions such as lung disease or overactive thyroid
Electrolytes

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4
Q

Atrial Fibrillation (afib or AF)

A

multiple irritable spots in the atria
IRREGULARLY IRREGULAR (both atrial and ventricular)
HR: 100-175 bpm
NO identifiable P wave
“fibrillation” waves

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5
Q

afib: Clinical Manifestations (7)

A

may be asymptomatic
palpitations
heart racing
fatigue
dizziness
chest discomfort
SOB

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6
Q

afib: Causes (3)

A

electrolytes
hypoxia
cardiovascular disease (all)

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7
Q

afib: Complications (3)

A

decreased cardiac output
heart failure
EMBOLUS–> stroke

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8
Q

afib: Stats

A

An estimated 2.7 million people are living with it
Untreated afib DOUBLES your risk of heart-related death, and causes a 4 TO 5 TIMES GREATER RISK of stroke
About 15-20% of people who have strokes also have afib

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9
Q

afib: Treatment

A

the MOST common type of treated dysrhythmia
Pharmacological:
-rate control: beta
blockers, CCB, digitalis,
amiodarone
-stroke prevention:
anticoags, antiplatelets
Non-pharmacological:
-abalation, cardioversion

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10
Q

Ventricular Dysrhythmias: Types (3)

A

Premature Ventricular Contractions
Ventricular Tachycardia
Ventricular Fibrillation

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11
Q

Premature Ventricular Contractions (PVCs)

A

-contraction coming from an ectopic focus in the VENTRICLES
-it comes EARLIER than the QRS should come and doesn’t follow a normal rhythm or P wave
-wide and distorted in shape compared to normal QRS
-treat the cause

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12
Q

Premature Ventricular Contractions (PVCs): Causes (7)

A

stimulants
ELECTROLYTES
hypoxia
fever
exercise
emotional stress
CVD

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13
Q

Premature Ventricular Contractions (PVCs): Subtypes (3)

A

bigeminy
trigeminy
quadrigeminy

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14
Q

Ventricular Tachycardia (Vtach)

A

-Consists of 3 or more PVCs together
-Ectopic focus within ventricles takes control and fires repeatedly–> NO atrial contractions occurring
-SERIOUSLY decreases cardiac output
- considered DEADLY rhythm

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15
Q

Vtach: Characteristics

A

rate usually 150-200 bpm, usually regular
NO P wave evident, PR not measurable

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16
Q

Vtach: Associations (6)

A

MI
CAD
Significant electrolyte abnormalities
Heart failure
Drug toxicity
“Other bad things”

17
Q

Vtach: Treatment

A

ACLS–> depend on pulse, patient will be symptomatic very quickly unless converts back to other rhythm
-may need an anti-
dysrhythmic med like a
beta blocker or CCB
-electrolyte replacement
first question: if it’s going for more than 10-15 seconds, check for pulse or no pulse?–> if no pulse, start CPR

18
Q

Ventricular Fibrillation (Vfib)

A

DEADLY RHYTHM
irregular waveforms of varying shapes and sizes
the ventricles are just “quivering”
NO effective contractions: NO CARDIAC OUTPUT