Arrythmias Flashcards
Sinus bradycardia- causes
Meds: beta blockers, CCB, amiodarone, Li, dig
Increased vagal tone: athletes, sleep, IMI
Metabolic: hypoxia, sepsis, mexedema, hypothermia, hypoglycemia
OSA
Increased ICP
Sinus brady tx
if no sx: atropine, b1 agonists (short term), pacing
Sinus brady- MOST COMMON CAUSE
Dr. Flitter: The most common cause for a pause is a blocked PAC
Sick Sinus Syndrome- causes
periods of unprovoked SB
SA arrest
tachy-brady syndrom
chronotropic incompetence with endo trach tube
Sick sinus syndrome- tx
often need combo
BB, CCB, dig for tachy
PPM for brady
AV block- Type I
prolonged PR (>200ms); 1:1 conduction
AV block Type II Mobitz I (Wenckebach)
progressively prolonged PR
worsens with carotid sinus massage, improves with atropine
AV T II Mobitz I (Wenckebach)- causes
abnml AV node- IMI, inflammation, myocarditis, high vagal tone, drugs
AV T II Mobitz I (Wenckebach)- tx
often paroxysmal, nocturnal, asx
Septal MI
V1-V2 +/- avR; proximal LAD
Anterior MI
V3-V4; LAD
Apical MI
V5-V6; Distal LAD, LCx, RCA
Lateral MI
I, avL; LCx
Inferior MI
II, III, avF; RCA (85%), LCx (15%)
RV MI
V1-V2 & V4R (most sensitive); proximal RCA
Posterior MI
ST depression V1-V3, ST elevation in V7-V9 (posterior leads); RCA, LCx
SVT- atrial
Sinus tach (pain, fever, hypovolemia, hypoxia, PE, anemia) Atrial tach (catchols, EtOH, dig, CAD, COPD) Multifocal atrial tach (increased automaticity at multiple sites; seen with underlying pulm dz) Atrial flutter (macroreentry- usually in RA) A fib (irregular AVN bombardment, often from pulm veins)
AV Jxn
AVNRT (reentrant circuit)
AVRT: (reentrant circuit and accessory path; may show pre-excitation (WPW))
NPJT: increased jxnal automaticity
Unstable SVT tx
cardioversion per ACLS
ST tx
treat underlying stressor
AT tx
BB, CCB, adenosine
Amiodarone?
Radiofrequency ablation, class IC/II antiarrhythmics
AVNRT or AVRT tx
Vagal maneuvers
Adenosine
CCB or BB, DCCV
Ablation
NPJT
CCB, BB, amiodarone
AF
BB, CCB, dig, AAD
AFL
BB, CCB, dig, AAD
MAT
CCB and BB