Arrhythmias Flashcards

1
Q

give some cardiac causes of arrhythmias

A
MI
coronary artery disease
mitral valve disease
cardiomyopathy
pericarditis
aberrant conduction pathways
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2
Q

give some non cardiac causes of arrhythmias

A
caffeine
smoking
alcohol
electrolyte imbalance (K, Mg, Ca)
hypoxia
thyrotoxicosis
drugs
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3
Q

how can arrhythmias present?

A
syncope/presyncope
palpitationa
chest pain
pulmonary oedema
hypotension
asymptomatic - incidental finding
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4
Q

what would you ask for in the history of arrhythmias?

A
precipitating factors
onset and offset
nature v- fast/slow, reg/irreg
duration
associated symptoms
drug history
PMH of cardiac disease
family history of cardiac disease
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5
Q

define bradycardia

A

HR < 60 bpm

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6
Q

under what circumstances should you treat bradycardia?

A

symptomatic (syncope, hypotension, HF)

or HR<40

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7
Q

how would you treat bradycardia?

A
treat any underlying cause e.g. hypothyroidism, medications, MI
atropine IV
isoprenaline infusion
temporary cardiac pacing
permanant pacemaker
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8
Q

what is isoprenaline and what must you be cautious of?

A

non-selective beta agonist
activates beta1 receptors in heart - positive chronotropy and inotropy

can cause beta2 vasodilation in muscle beds, causing hypotension

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9
Q

how does atropine work?

A

blocks vagus nerve so decreases parasympathetic effects on the heart via M2 receptors that are found in the SAN and AVN

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10
Q

give one contraindication to atropine

A

myocardial ischaemia

because atropine increases o2 demand or the AVN

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11
Q

what happens in first degree heart block

A

slow AVN conduction
PR interval prolonged >200ms
all atrial impulses are transmitted to venticles
benign

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12
Q

what happens in mobitz type 1 heart block

A

defect of AVN conduction
repeated increasing lengthening of PR until a QRS is dropped
usually asymptomatic and treatment not needed

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13
Q

what happens in mobitz type 2 heart block

A

conduction defect below AVN
constant PR with intermittent QRS dropped
may have 2:1 block
risks progression to complete heart block
may need pacing if symptomatic

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14
Q

why would atropine be ineffective in treating mobitz type 2 block?

A

the defect is below the AVN

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15
Q

what happens in complete heart block?

A

atrial impulses not conducted to ventricles - complete AV dissociation
both P waves and QRS escape complexes may be present, but occur independently.
pacemaker required

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16
Q

what is the most common cause of complete heart block?

A

myocardial fibrosis

17
Q

how would you investigate arrhythmias?

A

12 lead ecg
if paroxysmal, amulatory ecg monitoring
bloods: FBG, U&E, TFTs, glucose, calcium, magnesium, cardiac enzymes, toxicology if indicated e.g. digoxin

18
Q

define tachycardia

A

HR > 100bpm

19
Q

what are the two main types of tachycardia?

A

narrow complex, QRS<120ms

broad complec, QRS

20
Q

What is the CHADS2 score used for?

A

To determine long term stroke risk in AF

21
Q

What’s factors are included in the CHADS2 score?

A
Congestive heart failure (1 point)
Hypertension (1 point)
Age > 75 (1 point)
Diabetes (1 point)
Prior stroke or TIA (2 points)
22
Q

How would you decide whether or not a patient with AF needs anticoagulation therapy?

A

Score 0 - low risk. Aspirin
Score 1- moderate risk. Aspirin or warfarin
Score 2 or more - high risk. Warfarin to get INR between 2-3

23
Q

How would you determine if a patient with an arrhythmia is haemodynamically unstable?

A

Systolic BP < 80
Reduced consciousness
Severe pulmonary oedema

24
Q

How would you manage a patient with bradycardia who is haemodynamically unstable?

A

Atropine

0.6-1.2mg

25
How would you manage a patient with tachycardia who is haemodynamically unstable?
``` DC cardioversion Chemical cardioversion (adenosine 6-24 mg) ```