Arrhythmias Flashcards

1
Q

The Three main types of Arrhythmias are:

A

1)Atrial Fibrillation
2)Ventricular Arrhythmias (or ventricular fibrillation) - MOST dangerous and requires resuscitation
3)Paroxysmal supra-ventricular arrhythmias

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2
Q

Difference between Af and ectopic beats?
( but if ectopic did need treatment? What could we give))

A
  • Ectopic beats: spontaneous and go away alone - no treatment
    • If treatment needed, use a beta blocker
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3
Q

ATRIAL FIBRILLATION

A

more serious, Abnormal disorganised signals-fired casing the atria to quiver or fibrillate = rapid and irregular heart beat

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4
Q

Symptoms

A
  • Heart Palpitations = pounding/fluttering
    • SOB and dizziness,tiredness
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5
Q

Complications

A

Stroke and heart failure

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6
Q

Types of AF(paroxysmal, persistent, permanent)

A
  • Paroxysmal AF: episodes stop within 48 hours without treatment
    • Persistent AF :Episodes last more than 7 days
    • Permanent AF: present all the time
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7
Q

AF could ….

A
  • Can lead to stroke, values are not emptying as they should - blood doesn’t fully eject = clots
    • Patients should be assess for strokes
    • Manage through ventricular rate or rhythm control
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8
Q

There are two treatment pathways for Atrial fibrillation:

A

1) Rate control - controls ventricular rate
Rhythm - control restores and maintains sinus rhythm

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9
Q

Cardio version

A
  • 1)electrical - using direct current
    2)Pharmacological - Using an anti - arrhythmic
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10
Q

Can’t give if symptoms

A

have lasted more than 48 hours- increased risk of stroke

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11
Q

Electrical

A
  • Electrical is preferred if it has lasted more than 48 hours
    • Need to wait until the patient is fully coagulated for 3 weeks before cardio version can be used and continue this for 4 weeks after
    • If haemdynamically unstable, then emergency electrical cardio version - give a parenteral anticoagulant and rule out left atrial thrombus immediately before the procedure - this needs to be done ASAP to ensure that anti coagulation is not delayed
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12
Q

If life threatening haemodynamic instability what would we start?? What is there’s no life threatening harmony amid instability???( less than 48 hrs/more than 48hrs)

A
  • As discussed, if life threatening haemodynamic instability - electrical cardioversion
    • If no life threatening haemodynaic instability …..
      . Less than 48 hours = Rate or rhythm control(electrical or aminodarone/flecainide)
      .More than 48 hours = rate control (verapamil, beta blocker but not sotalol)
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13
Q

Maintenance Drug treatment:
1st line:
RATE!!!!

A
  • RATE control: Beta blocker (not sotalol), rate limiting calcium Chanel blocker( eg, verapamil, diltiazem), digoxin
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14
Q

2nd line: RHYTHM CONTROL

A
  • Beta Blocker or Oral Anti -arrhythmic drug
    (eg,sotalol,amiodarone,flecanide,propafenone,dronedarone)

Or ELECTRICAL

If over 48 hours we go for electrical cardio version but there is always a risk of clotting so patient must be sully anti-coagulated for at least 3 weeks
Or / and give oral anticoagulation - continued for at Lear 4 weeks after cardio version

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15
Q

Ventricular rhythm is controlled by … if symptoms persist… use a

A
  • Ventricular rhythm is controlled with standard beta blocker
    • If symptoms persist use SPAF
    • Patients with episodes of paroxysmal AF
    • Sinus rhythm can be restored using ‘pill in the pocket approach - flecainide/propafenone when required on symptoms
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16
Q

We have to keep treating clots; STROKE Prevention - CHADsVASc

A

Only Men with score of 0 and women with score of 1 do not require any thromboprophylaxis!!

Thromboprophylaxis Options: Warfarin or apixaban or rivaroxaban

New onset of AF: Parenteral anti- coagulant

17
Q

Atrial Flutters are treated suing what? But they usually react how? Tachycardia means? Bradycardia means?
What do patients need to be assessed for first??

A

Treated using rhythm or Rate but usually reacts less effectively with drug treatment.

1)Rate control is usually temporary and used until sinus rhythm is restored
Using beta blockers or rate limiting calcium Channel blockers

2) Rhythm control uses Cardioversion - direct current, Pharmacological cardioversion BUT catheter catheter ablation is more suitable

Patient needs to be assessed for stroke still and need to be put on an anticoagulant IV 3 weeks before and 4 weeks after

Tachycardia= Fast heart Rate
Bradycardia = Slower heart rate

18
Q

What is the most dangerous form of An arrhythmia??? - its features are what with regards to pulse??it requires what asa result (physically or IV) - UNSUSTAINED VENTRICULAR TACHYCARDIA REQUIRES WHAT? If it fails do what?? - stable vs unstable?? What is the maintenance required??

A

The most dangerous!!!!!!!!!!!!!!
= ventricular tachycardia
- Pulseless or fibrillation = immediate defibrillation and CPR (IV amiodarone is given refractory to defibrillation)
- Unstable sustained ventricular tachycardia = direct current cardioversion

If fails give IV amiodarone and repeat direct current
- Stable - Sustained ventricular tachycardia - IV anti arrhythmic drug(amiodarone is preferred)
- Non sustained ventricular tachycardia -Beta blocker

Maintenance treatment:
For patients at risk of a cardiac arrest;
- Most pts have a cardioverter defibrillator implant
- Some patients also require a drug:solatol, beta blocker alone or beta blocker and amiodarone

19
Q

Amiodarone is used to treat what type of arrhythmias?

A

This is used to treat Supra-ventricular tachycardia (heart beating faster than normal)

20
Q

Adenosine is used to treat what? What are the effects of adenosine enhanced and blocked by? Who’s should it be avoided in?? how is it ideally given to a patient? What are some of its adverse effects?

A

This is used to treat Supra-ventricular tachycardia (heart beating faster than normal)

The effects of adenosine are enhanced by dipyridamole(anti-platelet) and blocked by theophylline’s.Should be avoided with asthmatic patients, can cause bronchospasm.

Adenosine should be ideally given as an infusion via a large calibre cannula due to its short half life

Adverse effects -
- Chest pain
- Bronchospasm
- Transient flushing
- Can enhance conduction down accessory pathways resulting in increased ventricular rate - (WPW syndrome)- wolf Parkinson white syndrome

21
Q

Torsdae De Pointes (Prolonged QT interval) can be treated using?? Never use an anti arrhythmic when? What else might we consider???

A
  • Treatment: magnesium Sulphate (+beta blocker) (not sotalol) - consider atrial and ventricular pacing)- NEVER USE ANTI- ARRHTHMICS - they prolong QT interval further - symptoms are worse
    Causes:
    • Sotalol and other drugs that prolong QT interval , hypOKalaemia and bradycardia
    • This can be caused by drugs, especially ones that cause hypokalaemia and severe bradycardia - Amiodarone,amiodarone,sotalol,macroloids,haloperidol,SSRI’s,TCA’s and anti fungal