Approach to Suspected Dysrhythmia Flashcards
Most arrhythmias can be classified as one of the 4 following:
- Disorders of impulse formation or automaticity
- Abnormalities of impulse conduction
- Reentry
- Triggered activity
cardiac dysrhythmias are divided into 3 groups:
- Premature / Ectopic / Escape beats or rhythms
- Bradyarrhythmias
- Tachyarrhythmias
Susceptibility to arrhythmias may result from many causes such as:
- Genetic abnormalities
- Acquired structural HD
- Lyte abnormalities
- Hormonal imbalances (thyrotoxicosis, hypercatecholaminergic states)
- Hypoxia
- Drugs (QT interval prolongation; changes in automaticity, conduction, and refractoriness)
- Myocardial ischemia
When assessing a patient with palpitations, the goal is to determine whether the symptoms represent:
- an arrhythmia that is minor and transient
- significant CVD
- a cardiac manifestation of systemic dz, such as thyrotoxicosis
- a benign somatic s amplified by underlying psychosocial characteristics of pt
Factors that increase risk of serious cardiovascular cause of palpitations
- Hx
- FHx of significant arrhythmias
- Personal/Fhx syncope or resuscitated sudden death
- Hx MI (and likely scarred myocardium) - PE
- Structural HD -dilated or hypertrophic cardiomyopathies
- Valvular disease (stenotic or regurgitant) - ECG
- Prolonged QTC
- Bradycardia
- 2nd- or 3rd-degree HB
- Sustained ventricular arrhythmias
common descriptions of palpitations
- “Flip-flopping” (or “stop and start” or “skipped beats”) = premature contractions
- Rapid “fluttering in the chest” = fluttering regular = SVT, sinus tach, VT; irregular = afib
- “Pounding in neck” = afib, aflutter, PACs
associated sx with palpitations?
CP, SOB
Dizziness, near syncope or syncope
PMHx that could relate to palpitations
- Chronic conditions
- CHF, CAD
- Valvular disease
- Thyroid disease, Diabetes, CKD - Social history: alc, Illicit drug use
- Meds: Stimulants, OTC cold medicine, AADs
- FHx: Sudden death or unexplained death, HD
Main dx studies for palpitations
12-lead ekg and ambulatory monitoring
May not actually see a dysrhythmia on EKG, but can obtain clues to any possible cardiac disease
- Prolonged QTc
- Bradycardia, heart block (AV block and BBB)
- Signs of old MI
- Atrial or ventricular hypertrophy / enlargement
- WPW or other pre-excitation syndrome (short PR interval)
types of ambulatory monitoring devices
- Holter monitor
- Event monitors
- Real-time monitors
- Patch recorders
- Implantable loop recorders
Useful if ambulatory monitor records a worrisome arrhythmia or if serious arrhythmias are strongly suspected despite normal findings on the appropriate ambulatory monitor
Invasive electrophysiology testing
Can induce suspected arrhythmia or map source of arrhythmia for possible ablation
other diagnostic studies for palpitations
EKG + exercise test: if palpitations w/ physical exertion and with suspected CAD
Echo: if PE/ECG suggests structural abnormalities or decreased ventricular function
Labs: guides therapy or evaluate discovered dysrhythmias; Thyroid, lytes
what’s Cardioversion?
Electrical - Uses electrodes/paddles (50-360 J) and causes mass depolarization of tissue = prolonged refractoriness, “resetting” AP / impulse propagation source
Chemical - Ibutilide (Corvert) – rarely used
Preparation / Procedure
* Pads placed or conduction gel applied to paddles
* Everybody must be clear of touching pt
* Requires informed consent, except in unstable emergency / cardiac arrest
* Requires sedation, except in unconscious unstable patient
indications for cardioversion
- UNSTABLE Tachyarrhythmias – SVT, AF, VT, VF
- Afib / flutter