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Pharmacology > ANS > Flashcards

ANS Flashcards

1
Q

Output of SNS

A

T1-L2

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2
Q

Effects of PSNS

A

Miosis, increased secretion, decreased HR, constricts bronchi, increased peristalsis, contract bladder

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3
Q

Effect of SNS

A

Mydriasis, decrease secretion except saliva and sweating, increase HR, dilates bronchi, inhibit peristalsis, inhibit bladder, eyelid open, NA and A secretion

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4
Q

Effect of horners syndrome (neck symp chain damaged on one side)

A

Miosis, anhidrosis, ptosis

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5
Q

Innervation of blood vessels

A

Sympathetic

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6
Q

Innervation of ciliary eye muscles

A

Only parasympathetic

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7
Q

Where does PSNS dominate

A

GIT, bladder, salivary glands

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8
Q

Common features of SNS and PSNS

A

Cell bodies in lateral horn, 2 axons, Ach at ganglionic synapses nAChR.

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9
Q

Features of PSNS transmission

A

Long preganglionic axon, short post, 1:3 pre post ratio.

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10
Q

Features of SNS transmission

A

Short preganglionic axon, long post, 1:10 ratio

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11
Q

PSNS post ganglionic neurotransmitter and receptor

A

Ach and mAChR

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12
Q

SNS post ganglionic neurotransmitter and receptor

A

NA and adrenoceptor

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13
Q

neurotransmitter and receptor at adrenal medulla

A

Ach and nAChR

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14
Q

What type of receptor is nAChR

A

Ionotropic, pentameric, alpha 3

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15
Q

What activates nAChR

A

Nicotine, ACh

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16
Q

What type of receptor is mAChR

A

Metabotropic, monomeric, GPCR, m1-m5

17
Q

What activates and deactivates mAChR

A

Muscarine and Ach

Atropine blocks

18
Q

What type of receptors are adrenoceptors

A

Metabotropic, alpha 1,2 beta 1,2,3

19
Q

What activates adrenoceptors

A

NA, A, isoprenaline

20
Q

Subunits of nAChR

A

Alpha, beta, delta, epsilon, gamma

21
Q

Action of nAChR

A

2ACh bind. Na in, K out -> depolarisation

22
Q

NAChR subtypes where

A

Ganglia alpha 3. AM and NMJ alpha 1

23
Q

Mechanism of metabotropic/GPCR

A

A binds to R, GP binds to RA, GDP displaced by GTP, beta gamma and alpha split. Activate of inhibit E. GTP hydrolysed to GDP, su’s rejoin

24
Q

Activation of PLC

A

Gq - PLC forms DAG, stims PKC, phosphorylates. Also forms IP3 stimulates R on ER, efflux of Ca

25
Q

M2 cardiac effect and mech INDIRECT

A

Inhibit AC, inhibit ATP to cAMP so less active PKA, less phosphorylation so more k out but less ca in - inhibit contraction

26
Q

M2 cardiac effect and mech DIRECT

A

Activate GIRK which acts on ion channels, more K out, less Ca in

27
Q

What do autoreceptors do?

A

Inhibit ACh release

28
Q

Output of PSNS

A

Cranial and sacral

Pharmacology (4 decks)

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