Ankle-->hip Flashcards
inflammatory response 1 (macrotrauma)
acute-physical damage to capillary wall–>plasma + RBC’s + hemoglobin leak into interstitial space
-swelling and edema-blood migrates to skin and often causes ecchymosis
haemotoma
accumulation of necrotic debris at the site of injury
- removed by phagocytosis and proteolytic enzymes before healing can occur
- cells around it need nutrients and O2-cell death can cause Secondary Hypoxic Tissue Necrosis
- leads to swelling, pain, spasm, necrotic debris, and reduced joint motion due to pain
inflammatory response 2 (chronic microtrauma)
- capillary is dilated by chemical mediators (histamine, bradykinin) and space b/t cells allows some plasma proteins to leak out/fat molecules to leak in (Transudate=RBC free exudate, which doesn’t cause discoloration)
- osmotic gradient upset leads to greater net filtration and then swelling and edema
- eg achilles tendonopathy (chronic tendonitis) from wearing high heels (short) all day then running (trauma on tendon)
- leads to pain, swelling, spasm, necrotic debris, and reduced motion
effects of edema on ligaments
- disruption of a ligament inside a capsule causes swelling and pushes ligament ends away from each other
- can form block of scar tissue–>bones further apart and greater joint laxity
- importance of reducing edema ASAP and as much as possible-joint should be locked w/ligaments in shorter position (functional) can reduce laxity by causing some adhesions to form
treatment for most injuries
Rest
I: decrease pain, spasticity, and facilitate local blood flow to depth of about 5mm (ie, good for bony areas)
C: reduces edema (if the pressure is hard enough w/out occluding blood flow). Never wear at night b/c hyperemia happens
E: assist lymphatic drainage
when/where do you see the ecchymosis?
in subacute injuries (48-72hours after) and below the site of injury b/c of gravity
-ie a bang on the hip could possibly cause pain closer to the knee if ecchymosis goes that far
predisposition and causes of shock
illness, extreme fatigue, extreme dehydration, high anxiety
causes: extreme blood loss, pain, and/or psychological trauma
symptoms and treatment of shock
Sx: pallor of skin, clammy skin, weak rapid pulse and weak shallow breathing
Tx: 911, lay athlete flat, maintain body temp and monitor vitals, O2 if available, elevate legs slightly (controversial). also stop bleeding
secondary survey
HOPES- history, observation, palpatation, evaluation of function, selective tissue tensions
crural joint
-tibiofibular joint
-talofibular joint
-talotibial joint
does sagital plane movements of dorsiflexion and plantarflexion
subtalar joint
talus and calcaneas articulations
supination/pronation
midtarsal joints
b/t other tarsal bones
contribute to pronation/supination + some abduction/adduction
ankle bony stabablizers
-malleoli hold dome of talus
ankle static stabalizers (lateral)
bone: lat maleoulus
1. anterior tibiofibular ligament-holds them together
2. anterior talofibular-stops anterior motion of bones
3. calcaneal fibular-stops calcaneous from rotating down
4. lateral talocalcaneal
dynamic: peroneus brevis and logus (weak)
ankle static stabalizers (medial and anterior)
anterior
bone: wide margin of talus
static: joint capsule
1. anterior tibiofibular lig- syndesmosis joint (proximal and distal)
dynamic ant. compartment muscle and peroneus tertius
medial ankle stabalizers
bone: medial malleolus
1. deltoid ligament-stops pronation
2. spring ligament and short plantar ligament maintain longitudinal arch
dynamic: medial flexor group, tibialis anterior
cavus foot
high arch, permanent supination-good jumpers and leapers
- usually have clawed toes due to natural tension on flexor digitorum and flexor hallucis longus tendons
- rigid bone structure, doesn’t loosen to absorb the force
- common anterior compartment syndrome, stress fractures in tibia
planus foot (functional)
flat foot, excessive pronation
-can cause knee, hip, back problems
-sustentaculum tali roates down w/ the calcaneas (it’s usually stacked on top)
-the talus is no longer supported and subtalar valgus occurs
-can see a bend in the achilles b/c of extreme pronation
-disapate forces easily but inefficient toe off b/c can’t supinate to make rigid bone structure
+ internal torsion of femur
foot biomechanics: from heel plant to toe off
heel plant: subtalar joint and foot slightly supinated
-shock goes up calcaneas
midstance: joints unlock as foot pronates to absorb shock
toe off: supination and subtalar/midtarsal locking to provide rigid lever for propulsion
hyperdorsiflexion
talus wedges b/t bones and pushes malleolus away (if rotation happens)
hyperplantarflexion
can pull ant talofibular and and deltoid ligaments
-overstretches anterior capsule–>laxity in joint line
hallux valgus
mech: valgus displacement of 1st MTP joint
cause: genetic, mm imbalance, shoe fit
tx: taping, muscular control
- predisposition to bunions
morton’s neuroma
mechanism: enlargement from compression of interdigital nerve body
cause: shoe fit too narrow
tx: bare feet, wide forefoot shoes, surgery
- nerve thickens myelin sheath when compressed
morton toe
mx: elongated 2nd metatarsal, can cause sesamoiditis
cause: genetic
tx: nothing, proper fit shoes
- 2nd MT accepts too much ground reaction forces–>can stress fracture 2nd metatarsal
- PIP joint can buckle
claw toes
mech: contracture of DI
cause: cavus foot, short shoes
tx: stretching toes to dorsiflexion, correct shoe fit