angina Flashcards

1
Q

investigations for typical agina

A

ECG - to see st level
bloods (anemia can cause chest pain too)
CT coronary angigraphy
stress echo

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2
Q

what 2 /3 features is associated with angina

A

Constriction like pain in chest/neck/arm/jaw
Brought on by physical activity
Alleviated by rest or glyceryl trinitrate within minutes
2/3 features indicate atypical angina pain

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3
Q

tx of typical angina

A

aspirin

GTN- tell patient to take another does if it doesn’t leave after 5 mins

you always give GTN with either a bb or calcium channel blocker because some patients cant tolerate a bb!

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4
Q

when would you suspect ACS IN TYPICAL ANGINA

A

if after 2 doses of GTN the pain hasnt subsisde

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5
Q

warn patients about side effects of nitrates

A

dizziness
flushing

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6
Q

second line

A

bb and longer acting DHP ca channel blocker

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7
Q

indications for CABG

A

if a person has triple vessel disease
still have symptoms despite on treatment

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8
Q

what makes up ACS

A
  1. UNSTABLE ANGINA
  2. STEMI
  3. NSTEMI
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9
Q

CRITERIA FOR UNSTABLE

A

Chest pain at rest or minimal exertion lasting >15 minutes

ECG changes (new ST-depression or T wave inversion)

NO rise in troponin: no myocardial necrosis

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10
Q

CRITERIA FOR NSTEMI

A

Chest pain at rest or minimal exertion lasting >15 minutes

ECG changes (new ST-depression of T wave inversion)

Rise in troponin: myocardial necrosis

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11
Q

SO WHATS THE DIFFERENCE BETWEEN NSTEMI AND UNSTABLE

A

the troponin levels

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12
Q

differnece between stemi and n stemi

A

nstemi - partially occluded

stemi - fully occluded

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13
Q

criteria for STEMI

A

STEMI = completely occluded coronary artery

Chest pain at rest or minimal exertion, lasting >15 minutes

ECG changes (new ST-elevation or left bundle branch block)

RISE IN TROPONIN

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14
Q

when to consider a PCI

A

if patint has come hospital and within 90 mins MAX 2 HOURS send them to the closes specialist centre

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15
Q

what does pci entail

A

a stent to bypass the obstruction

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16
Q

what if patient is not eligible for PCI

A

fibrolytic therapy - MUST CHECK FOR CI

17
Q

CI FOR FIBRINOLYTIC

A

AGAINST
Aortic Dissection
GI bleed
Allergic reaction
Iatrogenic: recent surgery
Neurological disease: recent stroke (within 3 months), malignancy
Severe HTN (>200/120)
Trauma, including recent CPR

18
Q

TX OF NSTEMI

A

you calculate whether high risk(revasularization) or low risk (usually more conservative)

19
Q

levels of troponin after MI

A

roponin levels peak about 24 hours after the infarction, and then gradually lower back to baseline over the next 1-2 weeks

20
Q

CAUSES OF RAISED TROPONIN

A

Arrhythmias
Coronary artery spasms
Aortic dissection
Severe hypertension
Chronic kidney disease
Pulmonary embolism
Sepsis

21
Q

4 changes in a NSTEMI ECG

A

T wave inversion, ST depression, flat T waves/pseudo-normalisation of previously inverted T waves; normal ECG

22
Q

What must be considered in terms of the time when a troponin sample is taken?

A

Troponin will not start to rise until 2 - 3 hours after the onset of ischaemia
Troponin levels generally peak at around 24 hours after myocardial injury
Troponin elevation may persist for 1 - 2 weeks following myocardial injury

23
Q

DEFIINTVE TX OF STEM

A

ercutaneous Coronary Intervention (PCI)

24
Q

What do Q-waves represent in the context of myocardial infarction?

A

hat do Q-waves represent in the context of myocardial infarction?

Full-thickness myocardial infarction, they develop once myocardial injury is complete and generally no longer reversible

25
Q

WE ALWAYS ADMINISTER MORPHINE WITH

A

ANTIEMETICS AND PERHAPS LAXITIVE

26
Q

What early signs of STEMI may be seen on an ECG?

A

In STEMI this is characterised by a ST-elevation on the ECG. There may be peaked T waves which are suggestive of a hyperacute STEMI.

27
Q

treatment of unstable angina

A

well because now a clot is actually there, this is a risk for furture MI, so we take aspirin as well as bacially other antihypertensive drugs too like ca2+ , nitrates etc