Anatomy Review: UMN Flashcards
UMN
Made of larger pyramidal cells
Neurotransmitter:
Detected by:
Glutamate
Glutamatergic receptors
UMN lesion aka __
occurs in the neural pathway above the __
pyramidal insufficiency
anterior horn
Weakness, ↓motor control (fine movements), ↑reaction of spinal reflexes (spasticity, clonus, and excessive extensor plantar) have Babinski’s response
UMN lesion or injury
A major function of the Corticospinal tract pathway
Fine voluntary motor control of limbs (voluntary body posture adjs)
Cortico-spinal tract (CST) controls
primary motor activity for the somatic motor
system from the Neck to the Feet
Injuries to the lateral corticospinal tract results in
same side paralysis (can’t move)
paresis (↓motor strength)
hypertonia (↑tone)
Ultimately, Corticospinal neurons synapse directly onto ___ for direct muscle control
Alpha Motor Neurons
Name the two pyramidal tracts
cortico-bulbar (neck ↑)
cortico-spinal (neck ↓)
Pyramidal tract (Ant. and Lat. CST) connections to SomatoSensory cortex suggest responsibility for modulating
sensory info from the body
Pyramidal tract lesions lead to paralysis on the __ side of the body.
corresponding (opposite)
After an injury to CST, pt can relearn
some crude touch
basic motions
BUT NO fine movement
The largest part of CST
lateral 90⁒ (transverse cut, oval area)
Lat. CST axons weave out of tract and back into __ of the spinal cord.
Ant. Horns
Lateral cortico-spinal tract controls
fine movement ipsilateral limbs
Anterior cortico-spinal tract controls
central axial and girdle muscles (Trunk)