anaesthetics Flashcards

1
Q

structure for dealing with anaesthetic related exam question?

A

A: airway - patent, or obstructed? do they need an ETT or can they sustain own airway - Mask

B: breathing: breathing on their own? maintained - - mask or LMA.
need mechanical ventilation: ETT

C: CVS - anaesthetic drugs - decrease CVS + resp - therefore monitor

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2
Q

LMA means?

A

laryngeal mask airway

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3
Q

ETT means?

A

endotracheal tube

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4
Q

3 steps prior to anaesthetic + op?

A

considerations

pre-op assessment

optimising

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5
Q

Considerations (pre-op)

A

Pt: risk - assess, high risk, minimise risk.
Info: informed consent - reduce anxiety etc

surgery: deal with the stress, blood loss, fluid shifts,

anaesthetic - ADRs, coma, CVS,CNS, resp problems

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6
Q

pre-op assessment

A

hx - known + unknown co-morbs - severe? control.
d+a
prev surg + prev anaesthetic problems

ex - ASA grading

exercise tolerance:
METs

investigations

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7
Q

exercise tolerance within pre op assessment?

A

MET - metabolic equivalents

2: walk around house
5: walk up hill , stairs
9: strenuous exercise

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8
Q

point of investiigations in pre-op assessment

A

establish baseline for post op monitoring.

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9
Q

aspects of optimisation

A
  • optimise: IHD, DM, COPD, HTN, asthma, epilepsy
  • do not stop
    anti epileptics, inhalers,
    anti-anginas
  • stop DM meds - pts done 1st
  • anticoagulants stopped if secondary
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10
Q

triad of anaesthesia

A

hypnosis (unconscious)

analgesia

muscle relaxants

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11
Q

if muscle relaxants used must have?

A

mechanical ventilation - ETT

and hypnosis - unconscious pt

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12
Q

Rapid onset IV induction agent?

A

propofol - readily distributes, fat soluble and crosses BBB

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13
Q

inhaled agent - for maintenance?

A

halogenated hydrocarbons - want a low MAC - minimum alveolar concentration - potent drug

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14
Q

common side effects of anaesthetic drugs - what should always be monitored

A
  • CVS: cause vasodilation (decreased TPR + BP) & venodilation (decreased venous return + CO)
  • resp: increased RR + decreased tidal volume/
    reduce hypoxic and hypercapnic drive
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15
Q

what anaesthetic drug doesn’t depress the CVS + resp systems?

A

ketamine

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16
Q

indications for muscle relaxants

A
  • neurosurgery (tight control of sats)
  • open cavity surgery - laparotomy
  • microscopic surgery
  • when ventilation + intubation are required
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17
Q

local anaesthetic - limiting factor

18
Q

aspects to help induction

A

quiet room /anaesthetic room

noise can trigger laryngospasm

19
Q

how do you monitor consciousness?

A
loss of verbal contact 
lack of movement 
resp pattern 
EEG 
stages of anaesthesia
20
Q

1st stage of anaesthesia?

A

stage 1: induction - between induction of agents + loss of consciousness
can still be communicating

21
Q

2nd stage of anaesthesia?

A

excited + delirious - resp + CVS become irreg.,

rapidly acting drugs are used to minimise the time spent in this stage

22
Q

3rd stage of anaesthesia?

A

surgical anaesthesia - skeletal m relaxes, resp depression,

23
Q

4th stage of anaesthesia?

A

overdose

severe brainstorm/medulla depression - resp ceases, potential cvs collapse

24
Q

simple airway management options?

A
  • face mask (triple airway manoeuvre)
  • oropharyngeal airway (feudal) must be unconscious
  • nasopharyngeal airway
25
Q

advanced airway management?

A
  • LMA: supraglottic - therefore does not protect the airway from aspiration or laryngospasm (where the vocal cords adduct)
26
Q

ETT allows for…

A

protection from gastric contents

muscle relaxants to be used

neurosurg - tight blood gas control

27
Q

airway complications?

A

aspiration

airway collapse - laryngospasm

28
Q

risks of unconsciousness?

A
hypothermia 
pressure sores 
hypoxia 
loss of protective reflexes 
VTE 
airway collapse
29
Q

monitor what?

A

Sp02, ECG,BP, Fi02, ETCO2, resp parameters, agent monitoring, temp, Urine output, NMJ, invasive venous / arterial monitoring, ventilator disconnect

30
Q

emergence substance?

A

sevoflurane

gas - commonly used in paeds + emergencies

31
Q

types of pain?

A

chronic (>3wks), + acute

cancer + non cancer

32
Q

nociceptive pain is?

A

obvious tissue injury or illness, normally well localised

33
Q

neuropathic pain description?

A

damaged or abnormal nervous system - described as burning, numbness, not well localised

34
Q

neuropathic pain tx?

A

does not respond to WHO pain ladder

35
Q

example of causes of neuropathic pain

A

nerve trauma
dm
fibromyalgia
chronic tension headache

36
Q

physiology of peripheral pain

A

tissue injury –> release of prostaglandin - stimulation of nociceptors - signal A delta + C fibres to spinal cord

37
Q

spinal cord pain physiology

A

dorsal horn (1st delay) fibres synapse - decuss at that level. ascend spinothalamic tract

38
Q

brain physiology of pain

A

thalamus –> cortex - pain perception

39
Q

physiology of pain modulation

A

descending pathway - gate theory: activation of inhibitor interneurone between nociceptor fibres and large distractive nerve fibre

40
Q

spinal cord pain tx

A

local anaesthetic
opioids
ketamine

41
Q

WHO pain ladder

A
  1. non opioid - aspirin, paracetamol
  2. weak opioid: codeine
  3. strong opioid: morphine,fentanyl