anaesthetics Flashcards

1
Q

structure for dealing with anaesthetic related exam question?

A

A: airway - patent, or obstructed? do they need an ETT or can they sustain own airway - Mask

B: breathing: breathing on their own? maintained - - mask or LMA.
need mechanical ventilation: ETT

C: CVS - anaesthetic drugs - decrease CVS + resp - therefore monitor

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2
Q

LMA means?

A

laryngeal mask airway

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3
Q

ETT means?

A

endotracheal tube

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4
Q

3 steps prior to anaesthetic + op?

A

considerations

pre-op assessment

optimising

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5
Q

Considerations (pre-op)

A

Pt: risk - assess, high risk, minimise risk.
Info: informed consent - reduce anxiety etc

surgery: deal with the stress, blood loss, fluid shifts,

anaesthetic - ADRs, coma, CVS,CNS, resp problems

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6
Q

pre-op assessment

A

hx - known + unknown co-morbs - severe? control.
d+a
prev surg + prev anaesthetic problems

ex - ASA grading

exercise tolerance:
METs

investigations

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7
Q

exercise tolerance within pre op assessment?

A

MET - metabolic equivalents

2: walk around house
5: walk up hill , stairs
9: strenuous exercise

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8
Q

point of investiigations in pre-op assessment

A

establish baseline for post op monitoring.

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9
Q

aspects of optimisation

A
  • optimise: IHD, DM, COPD, HTN, asthma, epilepsy
  • do not stop
    anti epileptics, inhalers,
    anti-anginas
  • stop DM meds - pts done 1st
  • anticoagulants stopped if secondary
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10
Q

triad of anaesthesia

A

hypnosis (unconscious)

analgesia

muscle relaxants

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11
Q

if muscle relaxants used must have?

A

mechanical ventilation - ETT

and hypnosis - unconscious pt

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12
Q

Rapid onset IV induction agent?

A

propofol - readily distributes, fat soluble and crosses BBB

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13
Q

inhaled agent - for maintenance?

A

halogenated hydrocarbons - want a low MAC - minimum alveolar concentration - potent drug

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14
Q

common side effects of anaesthetic drugs - what should always be monitored

A
  • CVS: cause vasodilation (decreased TPR + BP) & venodilation (decreased venous return + CO)
  • resp: increased RR + decreased tidal volume/
    reduce hypoxic and hypercapnic drive
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15
Q

what anaesthetic drug doesn’t depress the CVS + resp systems?

A

ketamine

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16
Q

indications for muscle relaxants

A
  • neurosurgery (tight control of sats)
  • open cavity surgery - laparotomy
  • microscopic surgery
  • when ventilation + intubation are required
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17
Q

local anaesthetic - limiting factor

18
Q

aspects to help induction

A

quiet room /anaesthetic room

noise can trigger laryngospasm

19
Q

how do you monitor consciousness?

A
loss of verbal contact 
lack of movement 
resp pattern 
EEG 
stages of anaesthesia
20
Q

1st stage of anaesthesia?

A

stage 1: induction - between induction of agents + loss of consciousness
can still be communicating

21
Q

2nd stage of anaesthesia?

A

excited + delirious - resp + CVS become irreg.,

rapidly acting drugs are used to minimise the time spent in this stage

22
Q

3rd stage of anaesthesia?

A

surgical anaesthesia - skeletal m relaxes, resp depression,

23
Q

4th stage of anaesthesia?

A

overdose

severe brainstorm/medulla depression - resp ceases, potential cvs collapse

24
Q

simple airway management options?

A
  • face mask (triple airway manoeuvre)
  • oropharyngeal airway (feudal) must be unconscious
  • nasopharyngeal airway
25
advanced airway management?
- LMA: supraglottic - therefore does not protect the airway from aspiration or laryngospasm (where the vocal cords adduct)
26
ETT allows for...
protection from gastric contents muscle relaxants to be used neurosurg - tight blood gas control
27
airway complications?
aspiration | airway collapse - laryngospasm
28
risks of unconsciousness?
``` hypothermia pressure sores hypoxia loss of protective reflexes VTE airway collapse ```
29
monitor what?
Sp02, ECG,BP, Fi02, ETCO2, resp parameters, agent monitoring, temp, Urine output, NMJ, invasive venous / arterial monitoring, ventilator disconnect
30
emergence substance?
sevoflurane gas - commonly used in paeds + emergencies
31
types of pain?
chronic (>3wks), + acute cancer + non cancer
32
nociceptive pain is?
obvious tissue injury or illness, normally well localised
33
neuropathic pain description?
damaged or abnormal nervous system - described as burning, numbness, not well localised
34
neuropathic pain tx?
does not respond to WHO pain ladder
35
example of causes of neuropathic pain
nerve trauma dm fibromyalgia chronic tension headache
36
physiology of peripheral pain
tissue injury --> release of prostaglandin - stimulation of nociceptors - signal A delta + C fibres to spinal cord
37
spinal cord pain physiology
dorsal horn (1st delay) fibres synapse - decuss at that level. ascend spinothalamic tract
38
brain physiology of pain
thalamus --> cortex - pain perception
39
physiology of pain modulation
descending pathway - gate theory: activation of inhibitor interneurone between nociceptor fibres and large distractive nerve fibre
40
spinal cord pain tx
local anaesthetic opioids ketamine
41
WHO pain ladder
1. non opioid - aspirin, paracetamol 2. weak opioid: codeine 3. strong opioid: morphine,fentanyl