all of it Flashcards
afterload
force against which ventricles push to eject blood - depends on arterial blood pressure and vascular tone
preload
degree of myocardial distention before shortening-increased stretch=increased force of contraction=increase cardiac output
gallop
S3 becomes intense and easy to hear, the resulting sequence of sounds simulates a gallop–called pre-diastolic gallop rhythm
click
semilunar valve stenosis will cause ejection clicks, mitral valve prolapse (less sharp of a sound) will be associated with mid-late systole
snap
stenosis of mitral valve will cause OPENING snap (usually opening of valves is silent) - loud snap=still mobile valve
S3
phase 2 of diastole, S3 is heard if an abnormal volume of blood is transferred into ventricle (sound is b/c ventricle dilates beyond the normal)
S4
late diastole, @ “atrial kick” sound is generated by pressure wave gradient and if ventricle is stiff and non compliant
grade 1-3 murmur
- faint, difficult to hear
- quiet but heard
- moderately loud, no thrill
grade 4-6 murmur
- loud with palpable thrill
- loud with palpable thrill, audible with stethoscope barely touching chest
- very loud with thrill, may be audible without stethoscope
pulsas alternans
alternating pulse with small amplitude and one with large amplitude–REGULAR rhythm (seen in L ventricular failure)
pulsas bigeminus
normal pulsation followed by premature contraction; the amplitude of premature contraction is less than regular (rhythm disorder)
pulsas bisiferans
best detected at carotid artery-characterized by 2 main peaks, 1st peak is “percussion wave” and 2nd peak is “tidal wave” (aortic stenosis + aortic insufficiency)
pulsas paradoxis
major decrease in amplitude of pulse during inspiration and increase in amplitude during expiration-rate is the same (asthma, emphysema, pericardial effusion)
pulse deficit
difference in rate auscultory and palpated–(a-fib)
homan sign
flex knee slightly and dorsiflex foot - calf pain is (+) sign and suggest thrombosis of superficial vein
vesicular breath sounds
soft, low pitched, heard over most fields long inspiration short expiration
bronchial
fast inspiration and slow expiration; high pitched, heard over manubrium
tracheal breath sounds
high pitched, harsh, heard over trachea, equal on inspiration and expiration
bronchovesicular breath sounds
medium pitched, heard mostly in first and second ICS anteriorly; equal inspiration and expiration
egophony
“eeee” to “aaaa” - intensity of spoken voice is intensified and has a nasal quality (upon auscultation) which would indicate consolidation
whispered pectoriloquy
whisper is clearly heard through stethoscope–increased intensity and pitch (consolidation in lungs, pneumonia, fibrosis)
bronchophony
increased clarity and loudness of spoken sounds (consolidation)