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EKG Mod 2 > All Decks > Flashcards

All Decks Flashcards

1
Q

Delay or prevent the conduction of depolarization

What three types?

A

Heart Blocks

SA Node
AV node
Bundle Branches

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2
Q

This, when unhealthy, may temporarily fail to pace for at least 1 cycle

May resume pacing in step with the previous rhythm

Long pause may induce an escape contraction from an automaticity focus

A

Sinus Block

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3
Q

A group of arrhythmias caused by SA node dysfunction associated with unresponsive supraventricular automaticity foci

No escape contractions

A

Sick Sinus Syndrome

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4
Q

This can develop from sick sinus syndrome

—> intermittent episodes of SVT and sinus bradycardia

A

Bradycardia-Tachycardia Syndrome

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5
Q

Prevent or eliminate conduction from the atria to the ventricles

Three types that get worse

A

AV Block

First, Second and Third Degree

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6
Q

Prolonged PR interval >0.20 seconds (>5 small boxes)

PR remains consistently lengthened from cycle-to-cycle

P-QRS-T sequence is normal in every cycle

Constant, normal P-wave

A

First Degree AV Block

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7
Q

Two types of second degree AV blocks

A

2nd degree Wenckebach (type I) AV Block

2nd degree Mobitz (type II) AV blocks

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8
Q

PR Interval gradually lengthens in each successive cycle until the P wave fails to conduct to the ventricles (no QRS complex after P wave)

P-P wave intervals are regular and constant

A

2nd Degree Wenckebach AV Block

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9
Q

Totally blocks a number of paced atrial depolarizations (P waves) before conduction to the ventricles is successful

2:1, 3:1, or higher ratios of P waves: QRS complexes

PR intervals prolonged with a SUDDEN dropped QRS complex

P-P wave intervals are regular

A

2nd Degree Mobitz AV block

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10
Q

Total block of conduction to the ventricles. Atrial depolarizations are not conducted to the ventricles

Atria and ventricles are completely INDEPENDENTLY pacing

An automaticity focus below the complete block escapes to pace the ventricles at its inherent rate

P-P intervals remain regular and constant

A

Complete 3rd Degree AV Block

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11
Q

Block of conduction in the right or left bundle branches

The blocked bundle branch delays depolarization to the ventricle it supplies

A

Bundle Branch Block

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12
Q

Ventricles do not depolarize simultaneously -> produces the “widened QRS” appearance on the ECG

Each QRS complex is of normal duration but they superimpose on each other causing a widened QRS with two peaks

What does R’ represent?

A

Bundle Branch Block

R’ represents delayed depolarization of the blocked ventricle

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13
Q

Widened QRS

A

Greater than or equal to 3 small boxes (0.12 seconds or more)

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14
Q

Right Bundle Branch Block

V1 and V2 (look for bunny years)

A

Left ventricle depolarizes punctually, so the R represents left ventricular depolarization and the R’ represents delayed right ventricular depolarization

Widened QRS

R: left Ventricle
R’: Right Ventricle

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15
Q

Left Bundle Branch Blocks

V5 and V6 (look for bunny ears)

A

The left ventricular depolarization is delayed, so the right ventricle depolarizes punctually R, and the R’ represents delayed left ventricular depolarization

Widened QRS

R: Right Ventricle
R’: Left Ventricle

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16
Q

Direction of depolarization as it passes through the heart

Direction and magnitude of a depolarization

A

Axis

Vector

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17
Q

This conducts depolarizations from the endocardium (inside lining of the heart) to the epicardium (outside)

A

Ventricular Depolariation

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18
Q

These are special connections in the ventricle which transmit depolarizations from the endocardium to they myocardial cells

A

Purkinje Fibers

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19
Q

Sum of all small vectors of ventricular depolarization

A

Mean QRS Vector

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20
Q

Since the left ventricle is larger than the right ventricle, where does the mean QRS vector point normally?

A

DOWN and to the LEFT

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21
Q

How does ventricular hypertrophy effect the mean QRS vector?

A

More tissue that will change the magnitude and direction of the vector

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22
Q

Which direction will the vector point during hypertrophy?

A

The vector will point towards the hypertrophied side

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23
Q

How does myocardial infarction effect mean QRS vector?

A

Blockage of one or more of the coronary arteries results in necrosis of tissue due to lack of oxygen and blood.

No vectors are derived from dead tissue so the mean QRS vector veers AWAY from an area of necrotic tissue

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24
Q

P wave represents the depolarization and contraction of both atria, we examine the P wave for evidence of what?

Enlargement includes both hypertrophy and dilation

A

Atrial Enlargement

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25
Q

Which lead do you look at for atrial enlargement?

A

Lead V1 is directly over the atria, so the P wave in V1 is the best source for atrial enlargement

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26
Q

With atrial enlargement, the P wave is usually….?

A

Diphasic (both positive and negative)

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27
Q

The chest electrode that records lead V1 is….

A

Positive

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28
Q

Right Atrial Enlargement criteria

A

A diphasic P wave in lead V1 with a large, often peaked initial component tells us that the right atrium is probably hypertrophied and dilated compared to the left atrium

If the height of the P wave in any LIMB lead (especially Lead II) exceeds 2.5mm (0.25mV) - amplitude - even if not diphasic, suspect right atrial enlargement.

(2.5 small boxes or more)

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29
Q

Point P waves are also called…

They are tall (>2.5mm) = Right Atrial Enlargement

A

P Pulmonale

30
Q

Left Atrial Enlargement criteria

A

Terminal portion of a diphasic P wave in V1 is large and wide

Terminal component of a diphasic P wave in lead V1 is usually negative

P wave duration is greater than or equal to 0.12 seconds (3 small boxes or larger) in LEAD II

This is also called P-Mitrale

31
Q

Bi-Atrial Enlargement criteria

A

Both left and right atrial enlargment

BLUF: tall p-wave in Lead II that is widened

32
Q

In right ventricular hypertrophy (RVH) there is a large…..in lead V1

What about the S wave?

A

R Wave

The S wave in lead V1 is SMALLER than the R wave in RVH

33
Q

When RVH is present, there is a large R wave in lead V1 that becomes……

A

Progressively smaller in the chest leads V2, V3 and V4

Enlarged right ventricle adds more vectors to the right side, so there is a right axis deviation

34
Q

How is the S wave in Lead V1 with left ventricular hypertrophy

A

DEEPER in lead V1 (left axis deviation is present)

Lead V1 is a right-sided leaded. LVH produces a large positive depolarization away from V1 hence creates large negative S wave

35
Q

How is R in LVH?

A

Tall R in lead V5

Lead V5 is over the left ventricle, so the increased depolarization is going toward the electrode of V5 when there is LVH

Result is more positive depolarization going toward the positive electrode of V5 which produces a tall R wave in that lead

36
Q

Sokolow-Lyon Criteria

Diagnosing LVH on an ECG

A

S wave V1 + R wave V5

If the depth (in mm) of the S wave in V1 + R wave (in mm) of the R wave in V5 >35mm, then LVH is present

37
Q

In LVH, which limb leads are ideal to check that may have a characteristic where the T wave has a gradual downslope and a steep return to the baseline, making it assymmetrical

A

Left chest leads V5 or V6

38
Q

Characterized by depression of the ST segment

What is the pathology?

A

Ventricular Strain

Increased resistance from a narrowed valve or from hypertension causes ventricle strain leading to hypertrophy (muscle working harder against resistance)

Depressed ST segment with an upward hump in the middle

39
Q

Which lead would indicate right ventricular strain and left ventricular strain?

A

V1 - right VS

V5 - left VS

40
Q

Overall guideline to check for cardiac hypertrophy

A
  1. Check lead V1 to see if the P waves are diphasic for atrial enlargement
  2. Check R wave in lead V1 or right ventricular hypertrophy
  3. Check S wave depth + R wave height in lead V5 for left ventricular hypertrophy
41
Q

Which coronary curves around the right ventricle?

A

Right Coronary Artery

42
Q

Which coronary curves around the left atrium?

A

Left circumflex

43
Q

Which coronary artery supplies blood to the left ventricle?

A

Left Anterior Descending

44
Q

What is the source of life-threatening ventricular arrythmias?

A

Hypoxic ventricular foci near the infarction

45
Q

What is cardiac ischemia, how is it characterized on the ECG, and what can it symptomatically cause?

A

Decreased blood supply from the coronary arteries

Characteristic sign of ischemia on an ECG is the INVERTED T-Wave

Cardiac ischemia alone can cause chest pain known as angina

46
Q

T wave changes are most pronounced in which leads?

T Wave inversion in which leads are pathological?

A

V1-V6

V2-V6

47
Q

Wellens Syndrome

A

Marked T wave inversions in leads V2 and V3

STENOSIS of the left anterior descending artery

48
Q

Cardiac Injury

A

Indicates an acute infarct

Injury is characterized by ST SEGMENT ELEVATIONS

49
Q

Brugada Syndrome

A

Hereditary condition that can cause sudden death in individuals without heart disease

Right Bundle Branch Block + ST segment elevation V1-V3

ST segment elevations have a peaked downsloping shape

50
Q

Acute Pericarditis

A

Diffuse ST segment elevations that are flat or slight concave (middle sags down)

Diffuse PR depressions

51
Q

ST Segment depressions (in leads where the QRS is upright) indicates compromised coronary blood flow until proven otherwise

This may indicate….

A

Subendocardial infarction

Positive stress test

52
Q

Cardiac necrosis: diagnosis of myocardial infarction is usually based on the presence of significant Q waves produced by an area of necrosis in the wall of the left ventricle

What is the criteria for significant Q waves?

A

Greater than or equal to 0.04 seconds (1 small box) in duration and/or Q wave is 1/3 (height and depth) of the entire QRS complex

53
Q

4 general ares of left ventricular infarctions

A

Anterior/septal

Inferior

Lateral

Posterior

54
Q

Lateral MI leads

A

Lead I, V5, V6

Occlusion of the left circumflex

55
Q

Inferior MI leads

A

II, III, aVF

Occlusion of the terminal branch of either right or left coronary artery

56
Q

Anterior/septal MI leads

A

V1, V2, V3, V4

Occlusion of the left anterior descending

57
Q

Always check V1 and V2

A

ST Elevation and Q waves (anterior infarct)

ST depression and large R waves (posterior infarct)

58
Q

The ECG diagnosis of infarction is generally NOT VALID in the presence of….

A

Left bundle branch block

Why? = unable to tell if there are ST segment elevations or concerning depressions

59
Q

NEW Left bundle branch block

A

A new left bundle branch block along with clinical correlation may be indicative of an acute infarctions

If area of infarction causes injury/necrosis of left bundle branch, may cause a left BBB

60
Q

Prolonged QT interval is a marker for…

A

Ventricular arrythmias such as torsades de pointes and also a risk factor for sudden cardiac death

QT interval is DEPENDENT on heart rate

The faster the heart rate, the shorter the QT interval

61
Q

Prolonged if QT interval is greater than 1/2 R-R distance

What is the QTc?

A

Normal is 450ms

Men greater than 450ms

Females greater than 470ms

62
Q

Low Voltage ECG

A

Defined as a QRS amplitude of:

<5mm (0.5mV) in limb leads (I, II, III, aVR, aVF)

<10mm (1.0mV) in chest leads V1-V6

63
Q

Etiologies for Low Voltage ECG

A

Amyloidosis, sarcoidosis, pericardial effusions, COPD, obesity, anasarca

64
Q

Early Repolarization

A

Greater than or equal to 0.1mV J-point elevation in 2 or more adjacent leads with either a slurred or notched morphology

ASYMPTOMATIC patient

More prevalent in young, athletic patients

65
Q

Electrical Alternans

A

Alternating amplitude of the QRS complexes in any or all leads (due to shifting fluid)

MCC: pericardial effusion

66
Q

Slow (“poor”) R wave progression

A

R wave height normally becomes progressively taller from leads V1 through V6

Remains smaller than the S wave height OR remains at low amplitude across the entire precordium

Etiologies: lead placement, MI, LVH, LBBB, WPW pattern, chronic pulmonary disease, normal variant

67
Q

S1Q3T3 Syndrome (Pulmonary Embolism)

A

S1: large S wave in lead I
Q3: Q wave in lead III
T3: Inverted T wave in lead III

Other possible ECG findings:

Sinus tachycardia (MC)
St depression in lead II
Transient right axis deviation
T wave inversion V1-V4

68
Q

Hyperkalemia

A

Peaked T waves

P wave widening and flattening with eventual disappearance

QRS widening

Cardiac arrythmias develop such as ventricular fibrillation

69
Q

Hypokalemia

A

T wave flattening and/or inverted

U wave appearance

Cardiac arrhythmias develop

70
Q

Hypercalcemia

A

Short QT interval

(Normal QT interval is <1/2 R-R distance)

QTc is less than 430

71
Q

Hypocalcemia

A

Prolonged QT interval

EKG Mod 2 (1 decks)

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