Airway & Airway Management Flashcards

1
Q

What are the causes of upper airway obstruction?

A
  1. Congenital/Genetic - large tonsils, macroglossia, micrognathia, neck masses, large adenoids
  2. Infectious: tonsillitis, peritonsillar abscess, pretracheal abscess, epiglottitis, laryngitis/RSV, Ludwig’s angina, retropharyngeal abscess
  3. Medical:
    –> Immunological: anaphylaxis, angio-oedema
    –> Cystic fibrosis
    –> Laryngospasm
  4. Trauma/Tumour:
    –> Laryngeal trauma
    –> Haematoma/masses
    –> Burns - thermal injury
    –> Smoke inhalation
    –> Foreign body/ haemorrhage
  5. Anatomical: tracheomalacia, subglottic stenosis
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2
Q

What are the predictors of airway difficulty?

A
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3
Q

Indications for Intubation

A

Failure to maintain own airway
* GCS < 8, aspiration risk, requiring manoeuvres and tolerating OPA to maintain airway

Hypoxic respiratory failure
Hypercapnoeic respiratory failure

Anticipated clinical course - airway burns, trauma, overdoses
Failure to oxygenate, ventilate or protect the airway

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4
Q

Examination findings for Intubating a patient

A
  1. Absent gag reflex
  2. Overt bleeding - anywhere, upper GI, local, neck
  3. Facial burns +/- soot in upper airway
  4. Neck swelling / subcut emphysema of neck
  5. Stridor, abnormal voice, drooling, foreign body
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5
Q

Relative contraindications for Intubation

A
  1. High risk of aspiration
  2. Difficult airway: known/suspected
  3. Inability to perform surgical airway
  4. Upper airway obstruction - foreign body, cancer, transection of trachea
  5. Haemodynamically unstable patient
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6
Q

Complications of Intubation

A
  1. Procedural complications: failed, surgical airway, trauma to teeth/vocal cords/trachea, pneumothorax, hypoxia
  2. Induction complications: hypotension, bradycardia, awareness
  3. Aspiration
  4. VALI - ventilator associated lung injury
  5. Auto-positive end expiratory pressure (auto-PEEP)
  6. Raised ICP
  7. VAP - ventilator associated pneumonia
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7
Q

Causes of hypotention post intubation

A

Pneumothorax
Induction agents
Hyperinflation
Acute myocardial infarction
Anaphylaxis

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8
Q

Causes of hypoxia post intubation

A

(DOPESM)
Dislodged ETT/ Right main bronchus or Oesophageal intubation
* Low PIP, ↓breath sounds, gastric breath sounds
* Prevented with waveform capnography
* Needs reintubation

Obstruction - kinking of ETT
* High PIP, ↑ secretions, high resistance to BMV
* Prevented + managed with suctioning, bite block, and ensuring tube not kinked

Patient factors: Pneumothorax, pulmonary contusions, APO, PE
* High PIP, high resistance to BMV, wheeze/crackles/subcut emphysema
* Management depends on aetiology

Equipment failure - Ventilator, ICC
* Patient improves when disconnected from ventilator
* ETT cuff leak - low PIP and low pilor balloon pressures
* Needs ETT exchange and prevented by checking ETT prior to intubation

Stacking, Spasm (bronchospasm)
Mucous plugging

Evaluate Airway pressures:
* If PIP and Pplat both high = decreased compliance from PTx, abdo distension, dyssynchrony
* If only PIP high = obstruction, bronchospasm

PIP - peak inspiratory pressure

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9
Q

7 Ps of RSI

A

(T = Intubation)
Preparation: T-10 mins
Preoxygenation: T - 5 mins
Physiologic optimisation: T-3 mins
Paralysis with induction: T
Positioning with protection: T + 30 s
Placement with proof: T + 45 s
Post-intubation management: T + 60s

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10
Q

Preparation for tracheal intubation includes:

A

STOP - MAID
S - suction
T - tools for intubation (DL/VL blades, handles, monitors, bougie…)
O - oxygen source for preoxygenation + ongoing ventilation
P - positioning

M - monitoring equipment: ECG, SpO2, BP, EtCO2
A - assistant, ambu bag + face mask, airway devices: ETT/stylet/LMA +
- airway assessment - LEMON, RODS, MOANS, SMART
I - IV access x 2
D - drugs - induction + nmba + fluids + pressors +/- fentanyl

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11
Q

3 basic preparation steps for RSI

A

Assess for anatomical / physiological difficulty
Develop airway management plan + backup strategy
Assemble all necessary personnel + equipment + medications

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12
Q

3 Strongest predictors of peri-intubation circulatory arrest

A
  1. Hypotension (SBP < 100)
  2. Elevated Shock Index (HR/SBP > 0.8)
  3. Hypoxaemia (SpO2 < 93%)
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13
Q

ETT sizes and lengths

A
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14
Q

Methods to confirm ETT placement

A
  • Conitnuous waveform capnography - preferred (or)
  • Digital / colorimetric EtCO2 (yellow = yes, needs 6 breaths)
  • Direct visualisation using bronchoscope or of tube going through cords
  • Ultrasound
  • Oesophageal detector devices
  • Palpation during intubation
  • Aspiration technique with cuff deflated
  • Clinical exam - bilateral rise and fall of chest wall
  • Misting of ETT
  • Auscultation bilaterally in each axilla
  • Chest Xray - only confirms depth

Impaired EtCO2 - in complete obstruction, asthma and cardiac arrest

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15
Q

Conditions associated with Hyperkalemia after Sux

A
  1. Burns > 10% - >5 days until healed
  2. Crush injury - >5 days
  3. Denervation (stroke, SCI) - >5 days until 6 mo post injury
  4. Neuromuscular diseases (ALS, MS, MD) - indefinitely
  5. Intra-abdominal sepsis - >5 days until resolution
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16
Q

Advantages of Non-invasive PPV

A
  1. Reduces WOB
  2. Improves pulmonary compliance
  3. Recruits atelectatic alveoli
  4. Less sedation
  5. Shorter hospital stay
  6. Decreases rate of ETT without its risks
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17
Q

Disadvantages of Non-invasive PPV

A
  1. Air trapping
    2.↑intrathoracic pressure –>↓VR + Afterload + CO –> Hypotension
  2. Pulmonary barotrauma –> Pneumothorax
  3. Respiratory alkalosis
  4. Abdominal compartment syndrome
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18
Q

Conditions associated with false capnographic/calorimetric CO2 readings

A

False Negative reading:
- Low pulmonary perfusion: cardiac arrest, inadequate chest compressions, massive PE, shock
- massive obesity
- tube obstruction (secretions/blood/fb)

False Positive reading:
- recent ingestion of carbonated beverage (<6 breaths)
- heated humidifier/nebuliser
- endotracheal adrenaline - gives transient false readings

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19
Q

Complications of Suxamethonium

A
  • Fasciculations
  • Transient ↑intragastric, IOP, ICP
  • Bradycardia
  • Masseter spasm
  • Malignant hyperthermia
  • Prolonged apnoea with pseudocholinesterase deficiency or MG
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20
Q

Regarding RSI, list 3 physiological effects of OBESITY on respiratory system and 2 physiological effects on other organ systems?

A

Respiratory effects:
- High incidence of resting hypoxaemia + hypercarbia
- ↑ O2 consumption + ↑ CO2 production + ↑ Airway resistance
- ↑ WOB especially supine
- ↓ TLC + ↓ VC + ↓ Expiratory reserve volume (due to collapse of small airways) + ↓ FRC (declines exponentially as BMI increases)

Non respiratory effects:
- ↑ intra-abdominal pressure + ↑ gastric volume –> ↑incidence of hiatus hernia + ↑GORD
- Higher Vd (Volume of distribution)

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21
Q

Special considerations for intubating PREGNANT woman?

A

AIRWAY
- Upper airway oedema + friability of tissues esp during labour and in pre-eclampsia pts –> poor Mallampati, cannot have repeated intubation attempts
- Hyperaemia + nasal polyps - ↑risk of bleeding with NPA + need smaller size ETT (<7)
- Breast tissue obstructing laryngoscopy blade - need short handle

RESPIRATORY
- ↑ Metabolic demand + ↓FRC –> shorter apnoea times, needs passive oxygenation with NP at 15L/min
- Harder BMV due to ↑intra-abdominal pressure so need HOB 30 deg

GASTROINTESTINAL
- Incompetence of Lower Oesophageal Sphincter with GORD
- Distorted gastric anatomy due to enlarged gravid uterus
- Delayed gastric emptying in labour
Therefore ↑Risk of Aspiration - so DO NOT bag patient during apnoeic phase + need cricoid pressure to compress oesophagus

POSITIONING
- Needs 15 deg left lateral tilt to prevent aorto-caval compression

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22
Q

Special considerations for intubating a GERIATRIC patient

A

4 principles of airway management in geriatric pts:
→ Increased likelihood of requiring intubation during acute illness
→ Increased difficulty with BVM + Intubation
→ Increased difficulty maintaining oxygenation + preventing complications due to poor cardiopulmonary reserve
→ Need for adjustment of drug selection and dosing during RSI

AIRWAY
→ Edentulous pts - poor mask seal → place lower rim of mask on the inside of pt’s lower lip to improve seal
→ Difficulty maintaining airway due to loss of upper airway tone
→ Reduced neck mobility - from C-spine arthritis +/- fusion - difficult laryngoscopy

RESPIRATORY
→ High rates of comorbid intrinsic lung diseases - difficult to preoxygenate and may require BiPAP
→ Impaired gas exchange from lung disease -> ↓PaO2 + ↓Apnoea time
→ More susceptible to permanent neuro/cardiac sequelae from brief apnoea periods - need to maintain SpO2 > 90%
→ ↓ Chest wall compliance - more difficult to ventilate through BMV + LMA
→ ↓ Lung elasticity + increased V/Q mismatch
→ ↓ Cough + mucociliary clearance -> ↑ risk of aspiration

**CARDIOVASCULAR **
→ ↓Cardiopulmonary reserve -> heightened sensitivity to negative inotropy + vasodilation from induction agents - need 30% dose reduction esp if showing signs of shock, + need fluid resus +/- inotropes

**DRUGS **
→ More likely to have comorbid CAD / Tachydysrhythmias - so avoid Ketamine as the catecholamine surge can ↑HR + ↑myocardial demand

ETHICAL
→ Should discuss with pts + their family to determine appropriate limits of care, ideally prior to any anticipated deterioration

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23
Q

How do you approach intubation in an Upper GI haemorrhage patient?

A
  • Ensure appropriate PPE - gowns, gloves, goggles, masks
  • Address hypotension: Blood transfusions/MTP +/- norad via RIC line
  • Head up positioning 45 deg
  • Pre-Tx: Consider large bore NGT + give 20mg IV Metoclopramide to ↓aspiration risk (but should not delay intubation)
  • Double suction setup with 2 assistants
  • Ketamine 1-2 mg/kg + Rocuronium 1.2 mg/kg
  • Avoid NIV / BVM for preoxygenation and apnoeic oxygenation due to risk of gastric insufflation [if BVM needed - gentle with <15 cm H2O PEEP]
  • VL with SALAD technique
  • If vomiting, release cricoid pressure and place in Trendelenberg position
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24
Q

What is the SALAD technique and how do you perform it?

A

Suction Assisted Laryngoscopy Airway Decontamination - is a method of suctioning during intubation to prevent aspiration during intubation

  1. Suction of oral cavity with Yankauer sucker followed by laryngoscope blade insertion (avoid submerging optic module in vomit)
  2. Yankauer sucker as a tongue depressor to allow laryngoscope blade into correct position
  3. Suction of the hypopharynx and then insertion of Yankauer sucker into oesophagus for continuous drainage
  4. Resposition Yankauer sucker to left side of mouth - assistant holds it
  5. May need sligh leftward rotation of the L-blade by 30 deg if larynx not visible - otherwise intubate + inflate cuff + suction of tracheal tube prior to ventilation
25
Outline 5 strategies to improve safety of endotracheal intubations
1. Standardised pre-RSI checklist 2. Standardised difficult airway algorithm instituted 3. RSI performed only by adequately experienced operators 4. Standardised equipment availability including VL equipment 5. Mandated use of NP apnoeic oxygenation 6. Mandated use of bougie or stylet for all intubation attempts 7. Regular training on AIRWAY drills for staff with provision of cadaveric workshops
26
After successful intubation, list 3 interventions to decrease the risk of patient developing VAP
1. New circuit for a new patient 2. Semi-recumbent position 30-45 deg 3. Maintenance of Endotracheal tube cuff pressure of ~20 mmHg 4. Subglottic suctioning 5. Mouth cares 6. Avoid flushing of condensate into lower airway
27
ARC states CPR is likely to be futile > 20 mins when which 4 criteria are met:
1. No reversible causes found 2. Non shockable rhythm 3. No ROSC or Age > 80 yrs 4. Non witnessed arrest 5. Persistently low EtCO2 (<10 mmHg) during CPR in intubated pts after 20 mins 6. No cardiac activity on POCUS
28
State 5 clinical circumtances where prolonged resuscitation attempts may be warranted
1. Toxicological cause - full neurological recovery after 4 hours of CPR is possible 2. Post thrombolysis (2 hrs post) 3. Hypothermia (get core temp to at least 32C before ceasing) 4. Asthma - correct for hyperinflation 5. Pregnancy - prior to resuscitative C-section 6. Persistent VF in young people until reversible / therapeutic options exhausted
29
What are the physiologically difficult airways and why are they important?
Greatest predictor of CV collpase with RSI meds and transition to PPV * Hypotension (SBP < 100 and SI > 0.8) * Hypoxia (SpO2 < 93%) * Acidosis / Alkalosis * Underlying respiratory conditions - severe asthma, COPD, pulmonary HTN, RH failure * Medications/medical conditions - ↑ICP, AMI, tachydysrhythmias, obesity, pregnancy, advanced age * Sepsis
30
What are the criteria on waveform capnography to confirm tracheal intubation?
1. Wave rises during expiration and falls during inspiration 2. Peak amplitude is consistent and increasing over 7 breaths 3. Peak amplitude is > 7.5 mmHg *Gastric CO2 will give capnography for up to 5 breaths with EtCO2 < 7 and amplitude is inconsistent and not rising * *In cardiac arrest: EtCO2 normally still > 15 however may drop near or below 7.5 after prolonged CPR , will still have normal rise and fall *
31
What is the approach to intubation after a decision to intubate has been made?
Is this a crash airway? If NOT, is this a difficult airway? If NOT, then proceed to RSI
32
Describe crash airway and its algorithm
Crash airway in a patient who is agonal/near death/ in circulatory collapse and needs immediate intubation attempt without the use of drugs as their state of near arrest is predicted to be unresponsive to direct laryngoscopy. If first attempt fails, give a large dose of Sux to ensure airways are sufficiently relaxed. If unable to intubate despite 3 attempts or unable to maintain oxygenation, then it proceeds to a failed airway.
33
Describe a difficult airway and the algorithm
Difficult airway is determined by the pre-intubation airway assessment → if yes, then RSI with double setup can be initiated ONLY * if the operator is forced to act (due to patient's current/expected deterioration) or * intubation is likely to be successful and oxygenation via BVM/SGA is possible Otherwise, proceed with awake intubation if time allows * awake technique done by direct/flexible/rigid VL If uanble to tolerate awake intubation or failed - attempt with flexible endoscopy, rigid bronchoscopy, intubating LMA, blind nasotracheal intubation, or cricothyrotomy *"Forced to act" scenario - where patient is rapidly deteriorating with impending respiratory arrest and clinician is compelled to take one best chance at securing the airway by giving RSI drugs - if this fails -> goes to failed airway algorithm *
34
What are the indications for an awake intubation?
* Significant risk of airway difficulty anticipated * Nasal or oral ETT is feasible * Compliant patient * Low risk of vomiting * Sufficient time for preparation
35
How do you perform an awake intubation using DL or VL?
1. Ideally performed with anaesthetics in OT 2. Pretreat with IV 0.2mg glycopyrrloate + IV 4mg Ondansetron 10-15 mins prior to if possible - for dry mouth and blunt gag reflex 3. Suction and pad dry mouth with gauze 4. Topicallise airway with nebulised 2% Lignocaine 8mls (or 4 mls of 4%) at 5 L/min followed by viscous Lignocaine gargle + use Mucosal Atomisation Device with 3mls of 4% Lignocaine or cophenylcaine spray to spray vocal cords and trachea under direct laryngoscopy 5. Lightly sedate with a dissociation dose of Ketamine 20mg (or 10mg propofol and 10mg ketamine) 6. Preoxygenate vai NRB mask or NIV, position and continue with 15L/min NP oxygenation 7. If patient coughs while passing bougie then: * Trans-cricoid injection of Lignocaine * Push another 50mg Ketamine as soon as bougie is passed through cords * Use mucosal atomisation device to spary 2-3 mls of 4% Lignocaine to cords and trachea
36
How do you perform awake fibreoptic intubuation (AFOI)?
1. 5 x sprays of cophenylcaine to each nostril while pt inspiring 2. Gargle Lignocaine viscous/spray oropharynx with cophenylcaine 3. Prime fibreoptic with size 7 ETT over the scope 4. Advance fiberoptic into nasopharynx until cords visualised 5. Spray cords and trachea with lignocaine 6. Cannulate trachea and advance ETT over scope
37
How do you perform a surgical cricothyroidotomy?
Scalpel-finger-bougie technique is preferred method for R hand dominant person: 1. Stand on patient's right side and immobilise larynx and palpate CTM with left hand 2. Make a 5 cm vertical incision midline over the CTM → using finger/forceps blunt dissect to the CTM 3. Make a horizontal incision through CTM → withdraw blade and immediately insert your finger 4. Dilate hole with your gloved finger (do not remove finger) and insert bougie through incision 5. Direct bougie down the trachea using volar pad of your finger - may feel tracheal clicks over bougie 6. Pass either cuff ETT size 4 or standard ETT size 6 over bougie until balloon passes through CTM 7. Inflate cuff + remove bougie + secure ETT with cloth tie 8. Confirm location with continuous waveform capnography + CXR
38
What are the CI and complications of surgical cricothyroidotomy?
Contraindications: * Child < 10 years old * Airway secured by less invasive means * Trauma with disrupted CTM or transection of trachea Complications: * Failure * Extratracheal placement with subcut emphysema * Damage to larynx/perforation of posterior trachea * Fistula formation, subglottic stenosis and voice changes are some of the long term complications
39
Outline the technique of needle cricothyroidotomy
* Use IV cannula size 14G in adults and 18G in children * Neck in extension (neutral if c-spine injury) + skin prep +/- LA * Attach saline filled syringe to cannula * Insert needle through CTM at 45 deg in caudal position * Draw back on syringe whilst advancing needle * Confirm endotracheal placement by aspiration of air (bubbles) * Advance cannula over needle until hub is at the skin * Secure cannula to skin and attach to jet insufflation circuit High flow Jet Ventilation * 1-3 mL/Kg * 50 mmHg (adults) * Passive expiratiorn important (NPA/OPA helps) * I:E 1:5 and RR 12
40
Describe a failed airway and its algorithm
1. SpO2 < 90% despite good two-person BVM ventilation 2. If in a CICO situation or 3 x failed attempts at laryngoscopy by experienced operator(s) 3. Skilled operator concludes intubation would be impossible even after a single attempt ==> FAILED AIRWAY If in CICO → proceed with surgical airway If the patient can be oxygenated (via BVM/SGD) but not able to be intubated → the alternative methods such as fiberoptic or intubating LMA may be used Otherwise, maintain oxygenation with SGD or proceed with surgical airway
41
What are the goals and methods behind preoxygenation?
1. Replace N2 with O2 in the gas-exchanging portion of lungs → to ↑safe apnoeic time 2. Needs at least 8 x TV breaths or ideally 3 mins before induction and continued duing the apnoeic oxygenation phase via NP at 15L/min of O2 3. Time to desat < 90% after proper oxygenation in: * Healthy 70kg male: 6-8 mins * Young children (10kg) <4 mins * Adults with chronic illness/obesity <3 mins * Near full term pregnancy: <3 mins If patient has inadequate spontaneous ventilation → use BVM with reservoir bag 15L O2 and gentl bagging synchromised with pt's breathing If adequate spontaneous ventilation in cooperative patient → HFNP or NIV with 100% O2 or flush flow O2 via NRB can be used as well If adequate spont ventilation in uncooperative pt → Sedate with Ketamine 0.5-1 mg/kg IV or 3-5mg/kg IM with aliquots of 0.5mg/kg titrated to effect with HFNP, NIV or NRB - this is delayed sequence intubation
42
Pre-treatment agents for RSI
1. Lignocaine 1.5mg/kg IV - mitigates bronchospasm in reactive airway disease and to prevent airway reflexes raising ICP in head injury 2. Fentanyl 3 mcg/kg IV - sympatholytic - mitigates the sympathetic discharge in cardiovascular disease and in head injury - give slowly to prevent chest wall rigidity 3. Nebulised Salbutamol 2.5-5mg in reactive airways 4. Atropine 20 mcg/kg IV in children < 5 years to prevent bradycardia Reactive airways - Ketamine 1mg/kg, Lignocaine 1.5 mg/kg Cardiovascular: Fentanyl 3-5 mcg/kg Elevated ICP: Lignocaine 1-1.5 mg/kg, Fentanyl 3-5 mcg/kg
43
ED algorithm to the Ventilated patient in Respiratory Distress
44
Post RSI Hypotension - common causes and interventions
Common causes: 1. High intrathoracic pressure 2. Induction agent effects 3. Significant prior or ongoing fluid loss / haemorrhage 4. Obstructive shock: PE, cardiac tamponade, cardiogenic shock 7. Distributive shock: Sepsis, anaphylaxis 8. Older pt with poor CV reserve
45
3 main principles of ECMO
1. If conventional resuscitation and critical care support is failing 2. Underlying condition is reversible 3. ECMO is a temporizing measure till he disease process has ran its course or definitive treatment can be arranged
46
What are som differences btw VV and VA ECMO
V-A ECMO: * Access major vein (IJ or Femoral) and major artery (Carotid) * Able to provide circulatory support and oxygenation * Used for pts with primary heart failure V-V ECMO: * Access to 2 x major veins (IJ or Femoral) or use a double lumen catheter to access 1 major vein * Lacks need to access and repair major artery * Provides oxygenation but lower than VA as oxygenated blood returns to venous side * Does not provide circulatory support * Well suited for reversible respiratory failure
47
What are some of the complications of ECMO?
* Thrombosis * Bleeding * Haemolysis * Infection * Limb ischaemia
48
List 3 important principles of post cardiac arrest care
Establish aetiology of arrest Prevent further ischaemic and reperfusion injuries Assess severity of injury for prognostication
49
List 4 criteria for achieving high quality chest compressions
1. Location: middle lower third of chest 2. Rate: 100-120 beats per min 3. Depth: 5 cm with full recoil 4. Minimise interruptions to CPR 5. Switch providers q2 mins or earlier if tired
50
Compare different ventilator settings for various ED patients
Use SIMV-VC mode **ARDS/Acute Lung Injury ** * Goals: recruitment, shunt reduction, avoiding atelectatic trauma, achieve adequate oxygenation * TV 6 mL/Kg (lower end of normal) * RR 14 (standard) * I:E ratio = 2:1 (reverse normal to ensure adequate recruitment) * PEEP 10-15 cm H2O (much higher than normal) * May need to lower TV and accept higher PCO2 to ensure Pplat < 30 cm H2O **ASTHMA/COPD** * Goals: adequate exhalation and avoid breath stacking and volutrauma while maintaining adequate oxygenation * TV 5-8 mL/kg * RR 8-10 (lower than normal to ensure exhalation with permissive hypercapnoea) * I:E ratio = 1:4-5 (long exhalation time) * PEEP 0 (for asthma), 5 for COPD - just enought for tubing * May need to consider permissive hypercapnoea but maintain pH > 7.15 * Asthmatics may need to accept higher peak pressures but aim Pplat < 30 **HEAD INJURY ** * Goals: avoid reduction in venous return and maintain CO2 at lwoer limit of normal * TV 6-8 mL/kg * RR 16 (marginally elevated) * I:E ratio = 1:2 (normal) * PEEP 5 (normal) - avoid high PEEP if possible and aim PCO2 35-40 * Tape rather than tie ETT **METABOLIC ACIDOSIS** * Goals: ensure adequate RR to maintain / improve compensation for metabolic acidosis * TV 8-10 mL/kg (higher than normal) * RR 20-30 (mimic RR prior to intubation) * I:E ratio = 1:1 or 1:2 * PEEP 5 (normal) * Begin with high RR and titrate as guided by serial gases **SEVERE OBESITY ** * Goals: Avoid atelectasis and shunting due to obesity * TV 8-10 mL/kg (use Ideal body weight) * RR 14 (standard) * I:E ratio= 1:2 (normal) * PEEP 10-15 (higher than normal) to maximise recruitment
51
Who qualifies for extubation in ED?
Improvement from underlying pathology that required intubation (toxidrome or suspected head injury in intoxicated pt ith normal CTB) * Easy intubation (high POGO or Cormack-Lehane score 1/2) * Passed spontaneous breathing trial on minimal ventilator settings - Sats > 95%, PaO2 >60 on 40% FiO2 on PEEP <= 5 with RR btw 6-30) * No ongoing haemodynamic compromise (HR < 100, SBP > 100) * Passed awakening trial with patient rousable, able to follow commands and strong enough to lift head off pillow and raise arms in air for 15 seconds Also palliated patients can be extubated in ED without meeting the above criteria
52
List clinically available modes of PPV
1. Continuous mechanical ventilation (CMV) - Assist control (A/C) * Ventilator will detect breathing and give whatever pressure/volume set at * Gives 100% of a breath when it senses + back up rate * For pts in total respiratory failure * Bad if pt is extremely tachypnoeic (DKA/ASA) 2. Intermittent mandatory ventilation (IMV) -SIMV * Gives a breath with support every time pt triggers, but doesn't add if they take extra 3. Continuous spontaneous ventilation - pressure support, CPAP, BiPAP
53
Draw out a normal EtCO2 capnogram in a healthy patient
54
What is happening here?
Someone trying to take a breath DDx: - Hypoxia, Hypercarbia, Inadequate anaesthesia
55
What is Mallampati score?
56
What is happening here?
Obstructive pattern - prolonged phase 2-3 DDx: - Obstructive lung disease (asthma, copd) - bronchospasm - kinked ETT - leaks in system
57
What is Cormack & Lehane grading scheme of a laryngoscopic view?
Grade I - entire glottic aperture seen Grade II - only a portion of the glottis seen IIa - aryternoids + part of cords IIb - arytenoids alone Grade III - epiglottis only Grade IV - not even epiglottis seen
58
What's this?
Endotracheal cuff leak
59
Equipment needed to perform a surgical cricothyroidotomy