AEMCA - Additional Review Flashcards
What are the GCS parameters?
E - Spontaneous
- Verbal
- Pain
- Absent
V - Oriented
- Confused
- Inappropriate words
- Inappropriate sounds
- Absent
M - Obeys commands
- Localizes to pain
- Withdraws from pain
- Abnormal flexion
- Abnormal extension
- Absent
What are the APGAR Parameters?
Appearance:
Pink - 2
Pink body, blue extremities - 1
Blue body, blue extremities - 0
Pulse:
> 100 BPM - 2
< 100 BPM - 1
Absent - 0
Grimace:
Strong cry/active movement to stimulation - 2
Grimace/weak movement to stimulation - 1
Floppy - 0
Activity:
Active - 2
Flexed arms and legs - 1
Absent - 0
Respiration:
Vigorous cry - 2
Slow and irregular - 1
Absent - 0
What are the LAMS score parameters?
Facial Droop:
Absent - 0
Present - 1
Arm Drift:
Absent - 0
Drifts - 1
Falls Rapidly - 2
Grip Strength
Normal - 0
Weak - 1
No grip -2
Sequence of the Upper Respiratory Tract?
Nasal cavities/sinuses, pharynx, larynx, trachea
Sequence of the Lower Respiratory Tract?
Bronchi, bronchioles, alveolar ducts/sacs, alveoli
Difference between Internal vs External Respiration?
Internal Respiration: Gas exchange between blood and tissue cells
External Respiration: Gas exchanged between air in lungs and blood
What is the #1 breathing muscle and is controlled by the phrenic nerve?
Diaphragm. If phrenic nerve is severed, then diaphragm will not work.
If C1 and C2 (atlas and axis) to C4 is damaged, what will occur?
Apnea (cessation of breathing)
What are CNS depressants?
Class of drugs that slow down activity of CNS (sedatives/tranquilizers). Can lead to dependence and addiction if misused.
Examples:
- Benzodiazepines (Ativan, Xanax, Valium)
- Barbiturates (Phenobarbital, Pentobarbital)
- Alcohol (Ethanol)
How do CNS depressants impair breathing?
Areas of the brain responsible for regulating respiration are suppressed.
1) Reduces brain’s signaling to respiratory muscles. Results in decreased drive to breathe (shallow breathing)
2) Some CNS depressants (such as benzos and barbiturates) causes muscle relaxation in the body. This includes muscles involved in breathing (diaphragm and intercostal muscles). This results in decreased breathing efficiency and reduced lung ventilation.
What is the CNS comprised of? What does the CNS do?
CNS is comprised of brain and spinal cord. Together, they integrate and process information, coordinate bodily functions, and regulate responses to stimuli.
Cerebrum: Largest part of brain. Responsible for consciousness, perception, memory, reasoning, and voluntary movement.
Cerebellum: Located below cerebrum. Responsible for coordinating and fine-tuning voluntary muscle movements, balance, and posture.
Brainstem: Connects cerebrum and cerebellum to spinal cord. Controls breathing, HR, BP, and sleep cycles.
What protects the CNS?
Meninges, CSF, blood-brain barrier
What is a priapism indicative of in an MVC?
Spinal cord injury
What is ARDS (Adult Respiratory Distress Syndrome)?
Also known as non-cardiogenic Pulmonary Edema.
- Inflammation in lungs damages alveoli, reducing normal gas exchange
- Increased permeability = fluid shift to interstitial space
- Increased osmotic pressure = pulmonary edema
- Occurs with anaphylaxis, pneumonia, sepsis, etc
What are the different lung volumes?
Inspiratory Reserve - 3300 mL
Expiratory Reserve - 1000 mL
Tidal Volume - 500 mL
Residual Volume - 1200 mL
Total Lung Capacity - 6000 mL
Vital Capacity - 4800 mL
*Residual Volume – Remains in the lungs to keep the alveoli open, even after all air expired
What is Pleurisy?
Inflammation of pleura caused by friction or a rub
- Sharp sudden and intermittent chest pain with dyspnea
- May increase or decrease with respiration
- Pleural “friction rub” may be audible
S&S of life-threatening asthma attacks?
- Silent chest (airways so constricted, they produce no wheezing)
- Decreased air entry
- Altered LOA
- Bradycardia
- Confusion
- Cyanosis
Tx for life-threatening asthma attacks?
- Epi 1:1000
- Ventolin
*Ventolin affects B2 receptors (bronchodilation)
*Ventolin slightly affects B1 receptors (increases HR minimally)
Patho of an asthma attack?
- Exposure to antigen
- Antigen stimulates massive amounts of immunoglobulin E (IgE) which is an antibody
- IgE attaches to mast cells
- Antigen binds to IgE on mast cells
- Triggers a massive release of histamine and leukotrienes
- Histamine causes vasodilation and leaky vessels
- Triggers mucous production and further constriction
What is a Pulmonary Embolism?
Blockage in the pulmonary artery/blood vessels in the lungs. Caused by a blood clot or other foreign material.
- Sometimes blood clots can form in deep veins of the body, usually in the legs (DVT)
- If this clot breaks loose from its original site, it can travel through bloodstream and reach the lungs.
4 causes of a Pulmonary Embolism?
- Air embolism (ex: IV)
- Long bone fracture
- Prolonged bed rest
- Post-surgery
S&S of Pulmonary Embolism?
- SOB/Dyspnea
- Tachycardia
- Pale, cool, diaphoretic skin
- Chest pain (increases with inspiration)
How can uncontrolled a-fib lead to emboli in the lungs?
Intra-atrial blood stasis (standstill of blood in atria)
What are the organs that lie within the retroperitoneal cavity?
ADUKPI
A - Aorta
D - Duodenum
U - Ureters
K - Kidneys
P - Pancreas
I - Inferior Vena Cava
What is the Cribiform Plate?
AKA - Ethmoid bone
- Separates brain from nasal cavity
- Thinnest part of the skull
- In a pt with facial trauma, never insert NPA as it can enter brain cavity through #cribiform plate
What is the Pterion?
- Weakest part of the skull
- Temporal lobe region
Signs of cerebral herniation?
GCS <9 or decreasing LOC with asymmetric pupillary response OR asymmetric motor response)
- Hyperventilate at 20-24 BPM
- Maintain ETCO2 at 25-35 mmHg
Cerebral hemorrhage often results in an increased ICP. Which vitals can you see notable changes in due to increased ICP?
BP, HR, Respirations.
*Pupils are not vitals, but extremely important to look at
How does ICP influence BP?
- As ICP rises, so does resistance to arterial blood flow to cranial vault
- Brain MUST be perfused. Therefore, pressure will rise in order to meet and exceed the rising ICP physiological limit
This phenomenon is referred to as a “widening pulse pressure”, where SBP increases further and further away from an unchanging diastolic pressure
How does ICP influence HR?
- Increase in ICP results in pressure on CN X (Vagus Nerve)
- CN X innervates SA node and is responsible for slowing the rate of cardiac conduction (activates PNS)
How does ICP influence the pupils?
- ICP compresses on CN III (occulomotor nerve)
- CN III innervates the eye; responsible for papillary dilation and constriction
- Compression of CN III results in a fixed, dilated pupil on the affected side.