advanced cardiac life support Flashcards

1
Q

ACLS is an attempt at restoration of
spontaneous circulation using…..

A

basic CPR + advanced management of airway , Endotreacheal ET tube defibrillation , cardioversion , IV medication,

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2
Q

Causes of cardiac arrest…..*9( dieases related to heart…. , which part .–> 2 , family history , OD? / eating ? , e-? , imbalance of sth. ??» arrhythmia

A
  • Coronary artery disease
  • Left ventricular dysfunction
  • Dissecting, ruptured aortic or ventricular aneurysm
  • Congenital heart disease
  • Cardiac drug toxicity
  • Choking
  • Electrical shock
  • Electrolyte imbalance
  • Acid-base imbalance………………leads to arrhythmia
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3
Q

Clinical manifestations of Cardiac Arrest ( LOC? breath? pulse? HR? Pupils ? physical movement ? )

A
  • Loss of consciousness
  • Absence of breathing
  • Absence of pulses and audible heart sounds
  • Dilation of the pupils of the eyes
  • Convulsions 抽搐
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4
Q
A
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5
Q

Medical management of Cardiac Arrest

A
  1. Cardio–> correct dysrhythmia ; concious patiant with AF/
    supraventricular tachycardiaversion; restablish stable rhythm from SA node.
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5
Q

Transcutaneous pacing ( for what? astysole<? how to perform it (2 ways)

A

;) Noninvasive,
For hemodynamically unstable bradycardia e.g. low
BP
* Asystole with a short time (<10 mins)
* Instituted rapidly by delivering electricity from an
external power source through large electrodes
attached to the patient’s chest
* The electric current pass through the thorax
anteriorly to posteriorly (or sternum-apex) causing
depolarization of the myocardium, resulting
contraction

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5
Q

how to management pulseless VT and VF ? think about the procedure …

A

Defibrillation–> cardiac arrest–> 360 J ,no anesthetic drug, must not on button of syn

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5
Q

how to perform cardioversion ( how many J need for AF & Arterial flutter and SVT ? phrasic energy? any anesthesia ? synchorized button on/off ? any contradiction on ECG?

A

Low electrical energy: 50J
* for AF: monophasic—200J; biphasic– 120-200J (Warfarin)
* Atrial flutter and SVT: monophasic or biphasic– 50-100J
* VT with pulse: monophasic or biphasic– 100J
* An anesthetic drug is given before the procedure
* Synchronized button of the defibrillator must be pressed on
during the procedure
* Inappropriate sensing of the QRS complex may result in
improper timing of the discharge of the current e.g. T wave

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6
Q

Outline the step of C-A-B

A

C-A-B
* Step 1: Check for responsiveness (tab and shout “Are you
alright?”)
* Step 2: Activate emergency response system and get an AED
* Step 3: Look and scan the chest for movement
* Step 4: Check the carotid pulse for no more than 10 seconds
Do Step 3 and 4 simultaneously
* Step 5: With pulse: support the patient by rescue breath with
6 seconds per breathe
Without pulse: Start 30 compressions and 2 ventilation –> FOR complete chest recoil between compressions , < 10second interruptions , avoid ventilation…
* Step 6: Defibrillation either by AED or defibrillator

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6
Q

CPR technique

A

CPR technique, rate, depth, rotation

  • Simultaneous ventilation-compression CPR for
    protected airway (compression: 100-120 beats
    per minute; ventilation: 1 breath every 6
    seconds)
  • Compression rate: 100-120 compressions per
    minute
  • Compression depth of at least 2 inches (5cm),
    no more than 2.4 inches
  • 2 rescuers available: Rotate compressors every
    2 minutes
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6
Q

Primary CABD Survey

A

Focus: Basic CPR and defibrillation (AED)
* Location: street and home without equipment
1. Check responsiveness
2. Activate emergency response system
3. Call for defibrillator
4. Look and scan the chest for movement; Check the carotid
pulse for no more than 10 seconds
5. C– Circulation: give chest compressions
6. A– Airway: open the airway
7. B– Breathing: provide 2 breaths
8. D– Defibrillation: assess for and shock VF/ pulseless VT by
AED

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6
Q

Secondary CABD Survey

A

Secondary CABD Survey

  • Focus: advanced management
  • Location: A&E department or hospital
    1. Check responsiveness
    2. Look and scan the chest for movement; Check the carotid pulse for no
    more than 10 seconds
    3. Activate crash call system; Call for E-trolley
    4. C– Circulation: give intravenous access, give fluid, adrenergic agent,
    consider anti-arrhythmics, pacing, defibrillation, cardioversion
    5. A—Airway: inserted advanced airway device as soon as possible (ETT,
    laryngeal mask airway, Combitube)
    6. B– Breathing: confirm and secure tube placement, use a commercial
    tube holder to prevent dislodgement, confirm effective oxygenation by
    ventilator
    7. D– Differential diagnosis: search for and reversible causes
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7
Q

C- Circulation

A

Rhythm: VF and pulseless VT
* Useless quivering of heart no blood flow  cardiac 唔blood flow點都搓唔二
arrest
* Treatment: only one therapy works!
* DEFIBRILLATION
* Defibrillation success: chances drop every minute
* Reduce the time between compression and shock
delivery and the time between shock delivery and
resumption of compressions immediately after shock
delivery

C- Circulation

  • Approximately 50% survival after 5 minutes
  • Survival reduced by 7% to 10% per minute
  • Rapid defibrillation is KEY
    *
    XCPR
    CPR prolongs VF, slows deterioration
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8
Q

A- Airway ( how and any equiment to help?)

A

Opening the airway:
* Head tilt chin lift
* Jaw thrust]
*The oropharyngeal airway
* Nasopharyngeal airway

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8
Q

B- Breathing 1) Pocket mask device 2) Bag-mask ventilation* Given in approximately one second
any advantages and complication???

A

Advantages:
* Eliminate direct contact
* Enables positive pressure ventilation
* Easier to perform than bag-mask ventilation
* Best for small-handed rescuers

Advantages:
* Provides immediate ventilation and oxygenation
* Operator gets sense of compliance and airway resistance
* May provide excellent short-term support of ventilation
* High oxygen concentrations are possible
* Can be used to assist spontaneous respirations

Potential complications:
* Gastric inflation

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8
Q

B- Breathing
*Endo-tracheal tube
* Tracheal tube holder

A
9
Q

D- Defibrillation

A

Defibrillator; Automatic External Defibrillator; iphasic waveform allows lower energy

e.g. HeartStart FR3 ; Philips FR3; lofepak CR plus

9
Q

How to use AED?

A

The three basic steps:
1. Press On/Off button
(A) –> green on
2. Follow the prompts to
apply the pads (B)
Remove clothing from
the chest
* Open the package you
will find
– a pair of pads connected
to a single cable with a
connector at the end.
* Place the sticky side of
the pads as the figure.
(sternum and apex of heart )
Place the pads correctly and
press firmly.
* Plug the connector at
illustrated

  1. Press the shock
    button if prompted (C)
    * Don’t touch the patient when
    analysing rhythm
    * It takes up to 15 seconds
    * Stop CPR during the analysis
    * AED will give you voice and screen
    prompts if a shock is indicated
    * Say “Clear” and make sure no one is in
    contact with the patient
    * Press the Shock button
    * The shock will not be delivered unless
    you press the button
    Continue CPR as protocol if the AED gives a
    “No Shock Advised” message
    * Do not remove the pads during CPR
10
Q

Medications : Describe indications, contraindications and dosages for:
Epinephrine (Adrenaline)

A

Epinephrine (Adrenaline) –> Rapid effect no.1 choice unless allergic

  • Vasoconstrictor
  • Increase peripheral vasoconstriction, increase BP, increase
    contractility, increase coronary blood flow
  • Indication:
    –Cardiac arrest: VF, pulseless VT, asystole, PEA ( pulseless electrical activity
    –Bradycardia: after dopamine to treat bradycardia : , and transcutaneous pacing
    –Anaphylaxis reactions: with corticosteriods and anti-histamine
  • Dosage:

–1 mg, repeat every 3-5 minutes

11
Q

Vasopressin

A

(An adrenergic alternative to adrenaline)

slower action….

  • Vasoconstrictor, effects duplicate adrenaline
  • Less severe adverse effects
  • 10-20 minutes half life (adrenaline: 3-5
    minutes)
  • One time dose of 40 units
12
Q

Amiodarone (Cordarone)

A

Antidysrhythmic drug
usage
* Increase action potential duration, increase PR and
QT intervals, decrease SA node automaticity
* Indications:

–First line antiarrhythmic for VF/ VT, wide
complex tachycardia
* Dosage:

–First dose: 300mg IV bolus
–Second dose: 0.5-0.75mg/kg IV

13
Q

Lidocaine

A
  • Ventricular antiarryhthmic drug
  • Decrease ventricular automaticity, reduce the
    disparity in action potential duration between
    ischemic and normal zones
  • Indications:
    –Suppress stable VT and wide complex
    tachycardias associated with acute myocardial
    ischemia and infarction
  • Dosages:
    –First dose: 1-1.5mg/kg IV
    –Second dose: 0.5-0.75mg/kg IV
14
Q

Magnesium

A

Cofactor in numerous enzymatic reactions
* Indications:
–Cardiac arrest associated with suspected
hypomagnesemic state
–Life-threatening ventricular arrhythmia
due to digitalis toxicity, tricyclic overdose
* Dosage:
–1-2g diluted in 10 ml D5 IV

AF or SVT, AR –> use digoxin to –> lead to toxicity–> hyperkalemia–> bradycardia –> ventricular tachycardia -

Contradiction w/ K-depleting diurectics, Rifampin ( treat TB) due to decreased serum concentration, thyroid ….

15
Q
A
16
Q

Procainamide

A

Procainamide

  • Ventricular antiarrhythmic
  • Decrease automaticity of all pacemakers, decrease
    intraventricular conduction
  • Indications:
    –Recurrent VT not controlled by lidocaine
    –Stable wide-complex tachycardia of unknown
    origin, procainamide is recommended before
    lidocaine and adenosine ( contradicted to 2nd/co3rd AV- block) SA node disease; contradict to carbamazepine , caffeine which lower effect if adenosine
  • Dosage:

–30mg/ min IV infusion, maximum 17mg/Kg

17
Q

Sodium bicarbonate

A

Sodium bicarbonate

  • Indications:
    –Preexisting hyperkalemia
    –Acidosis, tricyclic antidepressant overdose
  • Dosage:
    –Use ABG to guide therapy
18
Q

Asystole/ Pulseless electrical
activity (PEA)

A

Cardiac arrest rhythm associated with no
discernable ( visible) electrical activity on the ECG
* Successful resuscitation is rare
* Only if the cause of asystole is identified and
treated in a timely fashion can survive

19
Q

Causes : 5H & 5T

A

5 “H”
* Hypoxia
* Hypokalemia/ hyperkalemia
* Hydrogen ion (acidosis)
* Hypothermia
* Hypovolemia
5 “T”
* Tamponade, cardiac
* Thrombosis (pulmonary)
* Thrombosis (coronary)
* Toxins (drug overdose)
* Tension, pneumothorax

x

20
Q

Asystole/ PEA what to do?

A

Active search for “do not attempt
resuscitation” orders/ status
* Explicit criteria for stopping by physician
* Family presence at resuscitation efforts
* Survivor support plans
* More formal death notification

21
Q

choking what to do ??

A

Abdominal maneuver

22
Q

how to use ECG ????

A