ADULT BURNS Flashcards

0
Q

WHAT DO THE DEPTH OF INJURY MEAN? (1,2,3,4 DEGREE BURNS)

A

1-EPIDERMIS ONLY. REDNESS, PAIN, MINIMAL EDEMA, NO BLISTERS, IE SUNBURN.
2-AKA PARTIAL THICKNESS-SUPERFICIAL DERMIS AND DERMIS CAPILLARIES AND NERVE ENDINGS. BASEMENT MEMBRANE IS INTACT. PINK, WET, PAINFUL. FLASH FIRE.
3 AKA FULL THICKNESS- DEEP DERMIS REACHING LARGE VESSELS. LOOKS WHITE, DRY. PAINLESS. IMMERSION SCALD OR ELECTRICAL FLAME.
4-DEEP THERMAL INJURY INVOLVING BONY TENDON AND MUSCLE.

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1
Q

WHAT ARE THE 4 TYPES OF BURNS?

A

ELECTRICAL, CHEMICAL, THERMAL, RADIATION

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2
Q

WHAT IS THE RULE OF 9’S?

A

CLASSIFICATION OF BURN SIZE ON TOTAL BODY SURFACE AREA TBSA. ITS CUT UP INTO 9’S
HEAD 9, UPPER EXTREMITIES 9 EACH, ANTERIOR TRUNK 18, POSTERIOR TRUNK 18, LEGS 18 EACH.

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3
Q

WHAT BURNS QUALIFY FOR A HOSPITAL STAY?

A

PARTIAL THICKNESS GREATER OR= TO 25% TBSA
FULL THICKNESS GREATER OR= TO 10% TBSA
FULL THICKNESS OF FACE HANDS OR FEET
ELECTRICAL BURNS (POSS. INNER ORGAN PROB.)
SMOKE INHALATION INJURY (RESP. PROB)

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4
Q

WHAT ARE THE 4 CV EFFECTS THAT OCCUR WITHIN THE FIRST 24-48 HRS?

A

VASCULAR PERMEABILITY INCREASES
PLASMA VOL REDUCED
CARDIAC OUTPUT DECREASES
FLUID RESUSITATION

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5
Q

WHAT ARE THE CV EFFECTS AFTER 24-36 HRS?

A

HYPERDYNAMIC STATE
DECREASED RBC SURVIVAL TIME TO 40 DAYS
POTENTIAL FOR CARDIAC ARREST

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6
Q

VASCULAR PERMEABILITY INCREASES CAUSING A REGIONAL LOSS OF MICROVASCULAR INTEGRITY. A BURN OF WHAT % TBSA IS GENERALIZED LOSS EXPERIENCED?

A

30%

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7
Q

WHAT IS USED FOR FLUID RESCUSITATION IN THE FIRST 24-48HRS?

A

CRYSTALLOIDS. LR/NS. COLLOIDS WOULD JUST LEAK OUT AND BRING FLUID WITH IT…(IN THE RESUSUTATION PHASE)

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8
Q

WHAT IS THE KEY FACTOR IN DECREASING BURN DEATH AND WHO SHOULD IT BE GIVEN TO?

A

AGGRESSIVE FLUID RESUSCITATION. ADMINISTERED TO ALL BURN PT WITH GREATER THAN 15-20% TBSA BURN

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9
Q

HOW DO YOU CALCULATE FLUID VOLUME REPLACEMENT?

A

PARKLAND METHOD.
FOR FIRST 24HRS: 4ML/%BURN/KG. HALF IN THE FIRST 8HRS. THEN THE OTHER HALF IN THE NEXT 16 HRS. NO COLLOID.
2ND 24HRS: D5W MAINTENANCE. COLLOID: .5ML/%BURN/KG

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10
Q

WHEN DOES THE HYPERDYNAMIC STATE OCCUR? AND WHAT WILL YOU SEE?

A

AFTER 24-36 HRS. SEE FIGHT OR FLIGHT RXN. CARDIAC OUTPUT INCREASES, CATECHOLAMINE RELEASE. THIS CAN BE SUSTAINED FOR HOURS OR DAYS.

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11
Q

HOW IS CARBON MONOXIDE POISONING MEASURED IN PTS?

A

BY MEASURING CARBOXYHEMOGLOBIN LEVELS….NORMAL IS LESS THAN 1% NONSMOKER, LESS THAN EQUAL TO 10% IN SMOKER.

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12
Q

HOW DOES CARBON MONOXIDE CAUSE PROBLEMS?

A

IT HAS 200X THE AFFINITY FOR HGB OVER O2 SO OXYGEN ISNT DELIVERED TO TISSUES. SHIFT TO LEFT ON O2/HGB CURVE.

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13
Q

WHICH ORGANS ARE MOST SENSITIVE TO HYPOXIA?

A

HEART AND BRAIN

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14
Q

WHAT IS TREATMENT FOR CARBON MONOXIDE POISONING?

A

100% O2

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15
Q

WHAT ARE THE SYMPTOMS OF CO TOXICITY AS A FUNCTION OF BLOOD COHB LEVEL?

A

LESS THAN 15-20: HA, DIZZINESS, CONFUSION
20-40: N/V, DISORIENTATION, VISUAL DIST.
40-60: AGITATION COMBATIVENESS, HALLUCINATIONS, COMA, SHOCK
GREATER 60: DEATH

16
Q

WHAT IS THE MOST SENSITIVE INDICATOR OF VOLUME STATUS AND PERIPHERAL PERFUSION?

A

URINE OUTPUT

.5ML/KG/HR IS MINIMUM

17
Q

WHAT ARE YOU LIKELY TO SEE IN BURN PT AFTER GIVING THEM PROTEIN BOUND DRUGS LIKE BENZO’S OR DILANTIN?

A

A GREATER EFFECT BECAUSE MORE IS IN CIRCULATION AND NOT BOUND TO PROTEIN. LIVER TOOK A HIT AND IS PRODUCING LESS ALBUMIN.

18
Q

WHAT MUSCLE BLOCKER IS CONTRAINDICATED IN BURN PTS?

A

AVOID SUX 24 HRS TO 2 YEARS AFTER BURN INJURY. ITS OKAY IN THE FIRST 24HRS.

19
Q

HOW WILL BURN PT REACT TO NDMR?

A

THEY WILL CHEW THROUGH IT QUICKLY! BECAUSE THEY HAVE INCREASED RECEPTOR SITES, INCREASED BMR, INCREASE IN AAG, ALTERED RECEPTOR AFFINITY FOR MUSCLE RELAXORS.

20
Q

WHATS THE EBL FOR AN EXCISION AND GRAFTING?

A

EBL=200ML/1%BSA

PAST 24 HRS YOU CAN USE COLLOIDS AGAIN.