Adult Acquired Conditions Flashcards

1
Q

What is Parkinson’s Disease

A

PD is an age related degenerative condition caused by loss of nerve cells in the brain causing a dopamine reduction.

It is characterised by involuntary tremors, stiff/inflexible muscles, and slow movement

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2
Q

What is the role of SLT with Parkinson’s

A

Assessing communication and swallowing
Facilitating safe swallowing
Removing barriers to successful communication
Working in the MDT

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3
Q

How would a care plan for an individual with Parkinson’s look

A

Case history (?previous SLT input, severity of PD)
Initial Ax (speak to Pt and NOK/SO, gain understanding of baseline, what are Pts goals, assess swallow and comms, educate Pt RE swallow safety etc)
Therapeutic interventions based off initial Ax and Pts goals
Periodic reassessment
Onwards referral as required

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4
Q

How might you assess the speech and communication of someone with Parkinson’s

A

Informal Ax (what is Pts insight to their difficulties, speak to NOK/SO too if possible, how are their SLCN affecting their ADLs, what was their baseline, what are their goals, what communication environments do they particularly struggle in)

Formal Ax (maximum phonation time- pt say [a:] at a normal loudness and time for as long as possible and compare to norms, continuous articulation- [s]/[z] ratios, DDK rates- [p], [t], [k], Pt reading paragraph- to Ax breath support and voice quality,

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5
Q

How might you assess the swallow of someone with Parkinson’s

A

Informal Ax (has Pt/SO noticed any changes to their swallow, if so is it worse with certain foods/fluids/times of day, what environment do they eat in normally)

Formal Ax (oromotor Ax- Ax CN function, EAT 10- Ax qualitative impact of swallowing difficulties on the Pts QoL and ADLS, bedside Swallow Ax

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6
Q

What SLT interventions could support SLCN in someone with Parkinson’s

A

Singing/reading monologues to increase breath support

LSVT Loud (Lee Silverman Voice Training)
Voice excercises like SPEAK OUT program
Both help increase loudness, breath support and articulation

EMST

AAC

Memory strategies like chunking or association can support conversation

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7
Q

What SLT interventions could support swallowing in someone with Parkinson’s

A

EMST to strengthen muscles

Compensatory Strategies to increase swallow safety

Swallow coordination excercises

AND EATING!

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8
Q

What is a motor speech disorder

A

A motor speech disorder is characterised by difficulty moving the articulatory muscles needed for speech production due to weakness or reduced muscle coordination.

2 primary MSDs- Dysarthria and Apraxia

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9
Q

What is dysarthria

A

Dysarthria is when you have difficulty speaking because the articulatory muscles required for speech are weak. The term dysarthria refers to a group of speech disorders characterised by abnormal speed, strength, range, and accuracy of the movements required for phonation

Can be caused by conditions that affect the brain or nerves IE MS, cerebral palsy or Down’s syndrome

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10
Q

What are the 8 primary types of dysarthria

A

Flaccid
Ataxic
Spastic
Hypokinetic
Undetermined
Unilateral
Hyperkinetic
Mixed

(Fiona And Shrek Have Utterly Utterly Happy Marriage)

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11
Q

What are the 8 primary types of dysarthria and what features are associated with each type

A

Flaccid- breathiness, hypernasality, audible inspiration
Ataxic- distorted vowels, irregular articulatory breakdowns
Spastic- SLOW RATE OF SPEECH, strained/harsh voice quality
Hypokinetic- monopitch, monoloudness, palilalia (repetition of own speech)
Unilateral- reduced loudness, slow rate, imprecise articulation
Hyperkinetic- deterioration with increased rate, tremor, stoppages
Mixed- characteristics of 2 or more types
Undetermined- speech doesn’t fit into other categories

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12
Q

How does dysarthria differ from Aphasia or Apraxia of Speech

A

Characteristic AOS Dysarthria Aphasia
Muscle weakness No Yes No
Articulatory deficits Yes Yes No
Prosodic deficits Yes Yes No
Lang. processing deficits No No Yes
Consistent error patternsNo Yes No
Groping Yes No No

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13
Q

What are the primary indicators of dysarthric speech

A

1 UNCLEAR SPEECH

Difficulty moving articulatory (tongue, mouth, lips)
Slow/slurred speech
Difficulty controlling voice volume
Change in voice quality (IE becoming more nasal)
Hesitating a lot when speaking or using short sentences

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14
Q

How can the WHO ICF be used in assessing communication in adults

A

IMPAIRMENTS IN BODY STRUCTURE AND FUNCTION, including underlying strengths and weaknesses in speech production and verbal/nonverbal communication;

LIMITATIONS TO ACTIVITY AND PARTICIPATION, including functional status in communication, interpersonal interactions, self-care, and learning;

ENVIRONMENTAL AND PERSONAL FACTORS that serve as barriers to, or facilitators of, successful communication and life participation;

IMPACT OF IMPAIRMENT ON QoL and functional limitations relative to the individual’s premorbid social roles and abilities and the impact on their community.

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15
Q

What are the primary indicators of Apraxia of Speech (AoS)

A

Inconsistent speech errors
Difficulty imitating or saying sounds on your own
Groping with articulators
Slowed speech rate
Unaffected automatic speech IE “Hello! How are you”
No speech at all (severe cases)
Errors in tone, stress or rhythm

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16
Q

What is Apraxia of speech

A

Apraxia is an acquired motor speech disorder. It can be caused by any kind of brain damage (IE TBI, stroke). People with apraxia of speech do not have muscular weakness, but have impaired ability to produce words/sounds as a result of brain damage.

Sometimes people with AoS are unable to speak at all

17
Q

How is Apraxia different to Dysarthria and how could you identify which is which

A

Apraxia is a motor speech disorder caused by damage to the brain, whereas dysarthria is a motor speech disorder caused by muscle weakness.

As apraxia is caused by brain damage, errors are often inconsistent, whereas dysarthria has more consistent error patterns, so if a Pt is repeating the same word/phrase with varying errors, this would indicate apraxia

Additionally, apraxia does not present with muscle weakness unless there is a co-occurring dysarthria

18
Q

Outline an initial assessment for motor speech disorders

A

CASE HISTORY (medical hx, Pt + family informal Ax: person specific goals/needs, impact on QoL, barriers & facilitators to communication
NON SPEECH AX (CN Ax, check dentition, sustained vowel [a:] to check breath support), assess DDK to check articulator precision and coordination, observe posture and breath support)
SPEECH AX(standardised tests for dysarthria IE N-DAT, standardised tests for apraxia IE BDAE, informal Ax: observe phonation (what is voice quality like), articulation (stress-testing- 2-4 mins reading aloud to ax deterioration, or repetition of words/phrases to Ax error patterns, ax prosody and intelligibility through conversation
FURTHER AX as required to rule out other conditions (IE receptive/expressive language ax to rule out aphasia, or swallow ax, fluency ax etc as req)
DEVELOP PERSON CENTRED INTERVENTION PLAN INFORMED BY ICF AND PT GOALS
PERIODIC REASSESSMENT

19
Q

What are the two styles of dysarthria intervention

A

Restorative (Impairment based interventions to restore the function of speech)

Compensatory (Function based interventions to support successful communication)

20
Q

Examples of restorative dysarthria interventions

A

Postural Adjustment (to support respiration)
EMST (respiration and breath support)
LSVT/LSVT Loud (phonation support)
Speak out intervention (phonation support supporting intentional speech)
Exaggerated articulation (increase articulator precision AKA clear speech)
Pacing speech using hand tapping (decreases speech rate supports prosody)
Matching breath to natural speech pauses (supports prosody and breath support)

21
Q

Examples of compensatory dysarthria interventions

A

Communication partner strategies (IE active listening, feedback)
Speaker strategies (conversationsational repair strategies IE gestures to support communication)
Modifying communication environment (reducing background noise, reducing distance between communication partners
AAC to facilitate meaningful communication

22
Q

Examples of apraxia of speech interventions

A

Cued Speech- gestures near the mouth that cue a sound (can help in understanding the speech of others and in producing clear speech)

Melodic Intonation Therapy (singing/humming words/phrases that are hard to say. Helps with motor planning

Pacing speech-using hand tapping (decreases speech rate and supports intelligibility)

Verbal Motor Production Treatment (VMPT)- repeated practice of sounds/words/phrases to help in motor planning for speech

AAC- facilitates meaningful communication

Some people with AoS regain speech skill without SLT intervention (spontaneous recovery)

26
Q

What is aphasia

A

Aphasia is an acquired, multimodal language disorder resulting from neurological damage such as stroke.

It can affect both written and spoken expressive and receptive language

27
Q

What are the types of aphasia and what are the symptoms of each

A

Expressive/nonfluent/Brocas Aphasia: lesion in Broca’s area, anomia (word finding difficulties), a graphic (difficulty with written expression), only able to use single words or short phrases

Receptive/fluent/wernickes Aphasia: lesion in wernickes area, impaired spoken language comprehension, impaired written language comprehension, alexia (impaired reading comprehension)

Anomic Aphasia: mildest form of aphasia, characterised by anomia and semantic paraphasia (substituting words with similar words IE bird for chicken)

Global Aphasia: Most severe form of aphasia, characterised by written and spoken receptive and expressive language difficulties

Primary Progresssive Aphasia (PPA): this is a frontotemporal dementia, characterised by deterioration of written and spoken language abilities over time

28
29
What should be considered when implementing AAC systems with adults
RCSLT guidance is that AAC is often for life regardless of the age of the user, and can take time to learn so this should be considered when implementing- IE is high tech AAC appropriate for EOL Pts Communication partner training alongside AAC can enhance user engagement Anticipate CHANGES TO NEEDS, IE in degenerative diseases, start using AAC prior to when it is actually required
30
What is the role of an SLT in MCA Ax
Advance care planning Advocating for client Informed decision making IE EDAR Educating professionals carers and families RE supporting people with reduced capacity RE communication partner training
31
Why are outcome measures important?
Allows SLTs to determine intervention effectiveness Supports decision making and evidence-based practice Supports person centred care-interventions can be tailored to individual needs Treatment planning- outcome measures allow SLTs to monitor progress and change interventions if required Inform service delivery and improvement- Allows SLTs to identify issues or weaknesses in service delivery and build on these
32
What are some examples of outcome measures
Using standardised Ax prior and after a course of intervention to gauge progress Using QoL questionnaires such as the ADL Questionnaire to see whether Pt feels intervention has had a positive impact Observations- has functional communication been improved, are swallows safer at mealtimes Clinical info- IE swallowing: has aspiration rate/chest Ix frequency improved? RCSLT have an outcome measure calculator
33
What is dyspraxia and verbal dyspraxia
It is a developmental coordination disorder affecting an individuals ability to plan and coordinate movements Verbal dyspraxia is a speech disorder caused by difficulty in planning and controlling voluntary articulator muscle movements
34
What are the differences between aphasia, dysarthria and apraxia
Aphasia: lang disorder affecting receptive and expressive speech Whereas apraxia and dysarthria are both related to expressive speech only Apraxia: motor speech disorder caused by damage to neural pathways related to planning and coordination of speech movements Whereas Dysarthria: motor speech disorder caused by muscle weakness and/or lack of coordination
35
How would you initially assess someone with aphasia
CASE HISTORY: medical hx (lesion location, date of stroke, SLT input following stroke, previous medical Hx, medications, relevant comorbidities IE dementia), Pt and family member/carer discussion, their perspectives of difficulties, baseline communication levels, communication environments, goals for SLT ASSESSMENT: *CN Ax looking at muscle strength to rule out Apraxia of Speech or dysarthria*, FAST is very good for initial assessment, if already diagnosed look at BDAE, BNT, CAT, WAB-R, pyramids and palm trees. Also good Ax for impact on ADLs is the Aphasia Impact Questionnare of the CAT as this can also inform goal setting and outcome measures Then plan interventions based off areas of difficulty
36
Name aphasia assessments and describe the,
BDAE (Boston diagnostic aphasia evaluation): COMPLETE aphasia Ax for all modalities FAST (Frenchay aphasia screening test) quick aphasia screen good for initial Ax BNT (Boston naming test) shorter version of BDAE based on picture naming WAB-R (western aphasia battery) good for DIFFERENTIAL diagnosis of aphasia as is a complete Ax of all language CAT (comprehensive aphasia test) lang battery and assesses impact on QoL Pyramids and Palm trees assesses word finding and picture/word comprehension
37
What are some potential interventions for a Pt with Aphasia
Melodic Intonation Therapy: uses prosodic features to improve expressive language Phonological Component Analysis (PCA): pt answers 5 questions about a picture prior to naming it. Supports word finding Word retrieval cueing strategies: Phonological - cueing first sound in a word, Semantic- providing contextual cues about the word Conversation Partner training- teaches nonverbal and verbal communication strategies to communication partners, supports functional communication. Also consider telehealth such as apps as these are very effective in aphasia pts