ADHD Flashcards

1
Q

Attention

A

Construct of attention is multidimensional
Vigilance: the ability to sustain attention/orientation to a task for the length of time to complete the task or persistence of effort
Distractibility: likelihood that a child responds to the occurrence of extraneous events unrelated to the task
Executive Functioning (e.g., planning & organization) & Working Memory
Can be apparent in both free-play settings, but most commonly with situations requiring child to sustain attention to dull, boring, & repetitive tasks
Difficulties with attention sustained throughout developmental years

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2
Q

Inattention

A

Children with inattention tend to have:

Poor persistence of responding

Impaired resistance to distraction

Deficient task re-engagement following disruption

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3
Q

Impulsivity

A

Deficiency in inhibiting behavior especially ones that obtain immediate reinforcement

Impaired motor inhibition

Poor sustained inhibition

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4
Q

Hyperactivity

A

Excessive and often task irrelevant motor and verbal behavior
Restlessness decreases with age, becoming more internal, subjective by adulthood.
Developmentally inappropriate levels of activity, restlessness, and fidgetiness

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5
Q

Course: Infancy to Age 6

A

Infancy
difficult temperament, negative mood
High activity level
Irregular sleeping & eating habits

Age 3
Start to show behavioral problems
Over activity & impulsivity
May have difficulties with toilet training
Complaints of restlessness, inattention, & oppositionality

Age 6
Continued problem behaviors including aggression
Social skills lacking
Struggle with consequential thinking
Experience strong emotionality
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6
Q

ADHD Adolescent

Common and Associated Problems

A
Common Characteristics::
Hyperactivity may decrease
Disruptive classroom behaviors
Failure to be compliant (homework)
Poor organization
Poor social skills
Clumsy
Inattention becomes more disabling 
Academic underachievement
School disciplinary action
May be perceived as lazy or unmotivated
Occupational difficulties
Associated Problems::
Conduct Disorder 
Mood Disorders
Anxiety Disorders
Substance Abuse
Risk for automobile accidents & traffic violations
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7
Q

ADHD in Adulthood
Common
Associated

A

Common Characteristics
More likely to drop out of college, complete less education
Riskier driving
Internal restlessness
Greater risk of having another mental disorder
Poor sense of time management
Difficulty organizing, planning and completing tasks
Lower socio-economic status
Frequent changes in employment
More likely to be a small business owner than a professional
Higher rates of divorce and multiple marriages
Higher self-report of interpersonal problems

Associated Problems
Anxiety
Substance Abuse
Antisocial personality disorder

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8
Q

Comorbidity and Associated Problems, Continued

A

Associated Problems
Medical issues (sleep)
Academic underachievement
Motor coordination & adaptive functioning
Family functioning- parent stress and psychopathology
Behavioral difficulties (i.e. noncompliance, aggression)
Peer relations or acceptance especially with children who display aggression

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9
Q

Etiology of ADHD

or Cause of ADHD

A

Disorder arises from multiple causes (true of all developmental disabilities)
All currently recognized causes fall in the realm of biology (neurology, genetics)
There is NO EVIDENCE that ADHD is causes by environmental causes (e.g. diet, watching violent TV, etc).
Little-no support for Feingold method (dietary sugar ingestion that cause ADHD)
Some correlation studies between maternal smoking & low birth weight
Social factors moderate types & degrees of impairment

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10
Q

Etiology of ADHD

A

Final common pathway for disorder appears to be the fronto-striatal-cerebellar circuits in the brain.
Some areas implicated: right frontal lobe: Orbital frontal area is smaller in children with ADHD, striatal basal ganglia, right side of the cerebellum
These structures are 10 – 15 % less active

Neurochemical Deficits
Dopamine dysregulation:  Drugs (stimulants) that help ADHD are dopamine regulators
Norepinephrine dysregulation (strattera affects this)
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11
Q

Neurobiology

A

Smaller, less active, less developed brain regions
Oribital-Prefrontal Cortex (primarily right side)
Basal Ganglia (Mainly striatum and globus pallidus)
Cerebellum (central vermis area, more on right side)
Size of network is correlated with degree of ADHD symptoms, particularly inhibition
No gender differences
Differences largely persist with age
Results are not due to taking stimulant medications.

Neurochemical imbalances: abnormalities in one or more of the monoaminergic systems (dopamine or norepinephrine) within the prefrontal limbic areas of the brain (prefrontal cortex–which is believed to be related to the inhibition of behavior)

Dopamine & dopamine receptor gene & transmitter gene- Stimulant medication temporarily increases the dopamine & norepinephrine in the synaptic cleft

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12
Q

Genetics

A

ADHD is one of the most highly heritable of all psychiatric disorders

  • Most studies reporting heritability estimates between 0.60 and 0.95, indicating that 60%-95% of the variance in the presentation of the disorder can be accounted for by genetic factors
  • Higher rate of ADHD symptoms in first-degree relative of children with ADHD
  • Evidence from twin studies .7-.97
  • More heritable than height
  • Important to keep in mind when working with parents of children with ADHD
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13
Q

Etiology: Acquired Cases: Prenatal

A

Drinking & smoking during pregnancy increases chance by 5
Maternal smoking in pregnancy (odds ratio 2.5)
Maternal alcohol drinking in pregnancy (odds 2.5)
Prematurity of birth, especially if brain is bleeding (45 %+)

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14
Q

Etiology: Acquired Post-natal

A

Head trauma, brain hypoxia, tumors or infection

Lead poisoning in preschool years

Survival from acute lymphoblastic leukemia. Treatments for ALL cause brain damage, affects frontal lobe

Streptococcal Bacteria infection triggers auto-immune antibody attack of basal ganglia (rare)

Post-natal elevated phenylalanine (dietary amino acid related to PKU)

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15
Q

Etiology: Psychosocial

A

Excessive TV/ Videogames (No evidence of causation- People with ADHD tend to watch more TV, read less, talk more on the phone
No evidence for “fast paced society”
Family Stressors (Linked to ODD/ CD/ MDD)
Poor Child Management (Parental ADHD/ ODD/ CDD)
Learning Disabilities (Comorbid but not causal)
No evidence for Intolerant teachers/ parents as causing ADHD
Diets: Not causal but some new research coming out that may indicate sensitivities in children with ADHD to certain foods (may exacerbate but not cause symptoms)

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16
Q

Situational Factors

A
Motivation: 
settings w/ high rates of immediate reinforcement
enjoyment
Instructional materials
Setting: 1-1
Time of day
Fatigue
Environment: demands
17
Q

Prognosis

A
Predictors of Outcome
Comorbidity
Low IQ
Poor peer acceptance
Emotional instability
Parent psychopathology
18
Q

What Does ADHD Look Like In The Classroom?

A
Overly active
Doesn’t sit still
Too talkative
Calls out without raising hand
Fails to start work
Work completed is sloppy
Class clown
Wants peer attention
Doesn’t accept consequences
Easily frustrated
Doesn’t adhere to rules
Wants immediate gratification
19
Q

What Does A Child With ADHD Look Like With Peers?

A
Physically intrusive
Touchy
Butts into activities
Bossy
Insists on own way
Misses social cues
Can’t see others’ viewpoints
Difficulty ignoring provocation
Wants to switch activities too often
Rejected by peers
20
Q

Social Difficulties

A

As a result of the behaviors noted on the previous slide, children with ADHD often have difficulty in social functioning as well as academic functioning
Children with ADHD were less socially preferred, had fewer dyadic friendships, and were given a “rejected” status by over half of their peers in one study
Difficulties:
Following social norms about conversational reciprocation
Joining into conversations without interruptions
Listening to others closely
Responding in a relevant manner
Joining into games without significant disruptions