Addiction Flashcards

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1
Q

Cognitive explanation for gambling initiation

A

AO1-
Self medication (Gelkopf). People gamble to treat the psychological symptoms that they already suffer from.
They perceive it to make things better.

AO2-
Becona- major depressive disorder evident in majority of gamblers
There is no cause and effect relationship between depression and gambling
Li et al - Pathological gamblers who do it to self medicate were more likely to have other substance dependencies

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2
Q

Cognitive explanation for gambling maintenance

A

AO1-
Irrational beliefs about their ability, such as superstitious beliefs, a self confidence to ‘beat the system’, an attribution of successes to skill and failures to chance factors

AO2-
Delfrabbro- pathological gamblers were irration in some ways but just as accurate as none gamblers in estimating the odds of winning
Implications that there are more than one motivation leads to different treatments. CBT can correct these cognitive errors, therefore it gives the patient free will.

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3
Q

Cognitive explanation for gambling relapse

A

AO1-
Blanco et al- ‘recall bias’ is overestimating the wins and underestimating the losses, so that you remember the losses
Losses are not viewed as a disincentive, as they instead have the belief that more losses leads to the fact that they deserve to win (‘Just world’ hypothesis), motivating them to return

AO2-
CBT can correct these incorrect beliefs, thus giving the patient free will

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4
Q

Behavioural explanation for gambling initiation

A

AO1-
Psychological, physiological, social and financial rewards reinforce the behaviour
Rewards may be infrequent, but they focus more on the wins

AO2-
Adaptation means that we generally learn behaviours that work out on average

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5
Q

Behavioural explanation for gambling maintenance

A

AO1-
There is occasional reinforcement, and they become used to this.
More reinforcement comes from social approval. Lambos found that gamblers with peers and family’s approval were more likely to gamble more and less likely to intend to stop

AO2-
Social reinforcement- Anti-addiction programmes target beginner adolescents as they are most vulnerable to peer influences

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6
Q

Behavioural explanation for gambling relapse

A

AO1-

They relapse due to conditioned cues for gambling. The triggers increase arousal

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7
Q

Biological explanation for gambling initiation

A

AO1-
Gambling can be seen to run in families due to a genetic link. Black et al found that first degree relatives of pathological gamblers were more likely to suffer from pathological gambling than more distant relatives

AO2-
Explains why some out of two people with the same environment, one may gamble and be more resistant to treatment
However, this could also be due to social modelling
Diathesis stress model- some are more vulnerable to addiction, but only in situations of environmental stress.
Doesn’t explain why some forms of gambling are more addictive. Video gambling- takes a year to become addicted. Horse betting takes 3 and a half.

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8
Q

Biological explanation for gambling maintenance

A

AO1-
Gamblers have an underactive pituitary adrenal response, meaning they have a lower appreciation of risk. Paris et al- measured cortisol levels before and after participants watched a gambling video. Recreational gamblers had increased levels, pathological gamblers didn’t. Shows how they seek more arousal

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9
Q

Biological explanation for gambling relapse

A

AO1-
They are sensation seekers are need intense stimulation because they have a poor tolerance for boredom. Boredom leads to their relapse

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10
Q

Cognitive explanation for smoking initiation

A

AO1-
Smokers become addicted because of their expectancies of the costs and benefits
They report smoking when in negative mood states and expect it to alleviate their moods

AO2-
Research focusses on ‘problematic’ behaviour but does not explain the role of expectancy in ‘loss of control’

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11
Q

Cognitive explanation for smoking maintenance

A

AO1-
Unconscious expectancies involve automatic processing, which explains ‘loss of control’
Tate et al- told smokers they should expect no negative experiences during a period of abstinence, this lead to fewer somatic and psychological effects than a control group

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12
Q

Cognitive explanation for smoking breakdown

A

AO1-
The perceived pros and cons of smoking behaviour affected participants quitting behaviour. Those who perceive smoking to have many benefits and quitting to have few benefits will be more likely to relapse

AO2-
Moolchan- Nicotine patches improved cessation rates and reduced relapse rates, but only when taken with CBT to change the positive expectancies of smoking behaviour

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13
Q

Biological explanation for smoking initiation

A

AO1-
A US study by Boardman et al found that heritability for regular smoking is 42%, Vink et al found 44%

AO2-
Shows how genetics play a part, but that the external factors have an equal role

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14
Q

Biological explanation for smoking maintenance

A

AO1-
Nicotine releases dopamine which creates feelings of pleasure, which drops as nicotine levels drop, leading to a cycle of smoking which is hard to break away from
Vink et al found that nicotine dependency was influenced by 75% genetic factors

AO2-
Initiation is due to external factors more so, while maintenance is due to biology more
Leads to pharmacological treatments
Thorgurrson- found a gene variant that influenced the amount of cigarettes smoked a day and the risk of smoking related disorders. It is possible you could be genetically screened to find those with the higher genetic risk, to then change their behaviour. If successful, this would reduce the burden on public health care, however it is unlikely to be successful due to the small associations between specific genes and smoking addiction

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15
Q

Biological explanation for smoking relapse

A

AO1-

Xian et al- 54% of the risk for quit failure was attributed to genetics

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16
Q

Behavioural explanation for smoking initiation

A

AO1-
Begin smoking due to social modelling, ‘experimental smoking’.

AO2-
Karcher and Finn- youth whose close friends smoked were 8 times more likely to smoke themselves

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17
Q

Behavioural explanation for smoking maintenance

A

AO1-
Sensory cues become conditioned stimuli with the effects of nicotine, so activate the same brain areas, making cessation difficult

AO2-
Thewissen- in one room smokers were presented with a cue predicting smoking in another, a cue predicted the unavailability of smoking. The first cue led to a greater desire to smoke

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18
Q

Behavioural explanation for smoking relapse

A

AO1-
Conditioned cues. Hogarth- craving increased significantly in response to conditioned stimulus

AO2-
Drummond et al- Cue Exposure. Presenting a cue without the opportunity to smoke may ‘unlearn’ the behaviour by extinguishing the association between the cue and smoking

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19
Q

Sulkunen (AO1 for Research into Film Representations of Addiction)

A

Looked at scenes from films that represented addictions. They presented drug use and the enjoyment of the effect, and contrasted it to a dull, ordinary life to show it as a way of alleviating problems

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20
Q

Gunasekera (AO1 for Research into Film Representations of Addiction)

A

Reviewed popular films for their portrayal of drug use. They portrayed drug use positively without showing the negative consequences of it

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21
Q

Sargant and Hanewinkel (AO2 for Film Representations of Addiction)

A

Tested whether adolescents seeing smoking in movies influenced their initiation of smoking. Surveyed 4000, and again a year later. Those who hadn’t smoked when fist surveyed were more likely to start smoking the following year if they had been exposed to it in films. -SHOWS LINK

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22
Q

Boyd (AO2 for Film Representations of Addiction)

A

Films do show the negative consequences. They show physical deterioration, sexual degradation, violence, crime and moral decline of drug use.

+ Also, film makers are provided with an incentive to show addiction in a negative way in the US!

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23
Q

Byrne on why films representations are important (AO2 for Film Representations of Addiction)

A

Popular films provide stereotypes for drug use and inform people’s opinions, so they have a responsibility to portray addictions accurately

24
Q

Bennett et al (AO1 for Role of the Media in Changing Addiction)

A

Evaluated ‘Psst…The Really Useful Guide to Alcohol’. Viewers were compared with matched controls. They showed an improvement in KNOWLEDGE, but no difference in attitudes

25
Q

Kramer (AO1 for Role of Media in Changing Addiction)

A

Assessed the effectiveness of ‘Drinking Less? Do it Yourself!’. Intervention group were MORE SUCCESSFUL than a control group in achieving low-risk drinking

26
Q

Do Anti-Drug Campaigns Work? (AO2 for Role of Media in Changing Addiction)

A
  • US Congress invested $1 billion on an Anti-Drug Campaign, including teaching resistance skills, self-efficacy and the teaching of negative consequences.
  • Hornik- failed to accomplish its goals. May have even lead to INCREASED marijuana use.
27
Q

Why don’t Anti-Drug Campaigns Work? (AO2 for Role of Media in Changing Addiction)

A

Hornik- not original, youths are exposed to so many similar ones

Contain the message that drug use is commonplace, that all of their peers are doing it, making them more likely to imitate it

28
Q

Kramer’s Analysis of ‘Drinking Less? Do it Yourself!’ Methodology (AO2 for Role of Media in Changing Addiction)

A

The study involved intervention and control group. Intervention group received weekly visits from researchers, the extra attention may have worked in their favour

29
Q

Risk factors - Stress - Sinha (AO1)

A

Used brain-imaging and found that the same part of the brain was activated during stress as during drug craving

30
Q

Risk factors - Stress- Driessen et al (AO1)

A

30% of drug addicts suffered from PTSD

31
Q

Risk factors - Stress- Cloniger (AO2)

A

Found 2 types of alcoholics. Type 1 drink to relieve tension (stress). Type 2 drink to relieve boredom. Type 1 are very vulnerable to stress influences.

32
Q

Risk factors - Age - Sumter (AO1)

A

We are more vulnerable to peer pressure when we are younger

33
Q

Risk factors - Peers - Karcher and Finn (AO2)

A

Youth whose close friends smoked were 8 times more likely to also smoke

34
Q

Risk factors - Peers - Shedler and Block (AO2)

A

18 year olds who hadn’t tried drugs were more likely to be socially isolated, potentially attributing their lack of drug use to less of an influence of peers, though this could also be due to personality factors

35
Q

Risk factors - Personality - Eysenck (AO1)

A

Devised 3 personality dimensions: psychoticism, extroversion and neuroticism.

36
Q

Risk factors - Personality - Francis (AO1)

A

Found that people with substance addictions scored highly on psychoticism and neuroticism scales

37
Q

Risk factors - Personality - Cloniger (AO1)

A

Thought that the 3 personality traits that predispose someone to an addiction are novelty seeking, harm avoidance and reward dependence

38
Q

Risk factors - Personality - Teeson (AO2)

A

Thought that it is difficult to entangle the effects of personality on addiction from addiction on personality (ie. cause or effect?)

39
Q

Biological Interventions - Strength of methadone

A

Allows for safer and controlled reduction and withdrawal under supervision

40
Q

Biological Interventions - Limitation of methadone

A

Many drug addicts who are on the methadone programme become just as reliant on methadone as they were on heroin. The UK Statistics authority showed that methadone was responsibly for over 300 deaths in the UK.

41
Q

Biological Interventions - Strength of Nicotine Replacement Therapy s

A

Eliminates the consumption of the harmful chemicals in cigarettes

42
Q

Biological Interventions - Limitation of Nicotine Replacement Therapy

A

Deliver nicotine more slowly, so many smokers perceive them as not being satisfying

60% relapse rate

Health issues- increased heart rate, constriction of coronary blood vessels, increased blood pressure, cardiovascular disease

43
Q

Biological Interventions - SSRI’s

A

There is evidence to support the fact that pathological gamblers have a serotonin dysfunction. SSRI’s increase levels or serotonin.

Hollander found that gamblers treated with SSRI’s showed improvements compared a control group.

44
Q

Biological Interventions - Naltrexone

A

Naltrexone is a dopamine receptor antagonist, which reduces the rewards and reinforcement of gambling

45
Q

Biological Interventions - Strength of Naltrexone

A

Kim and Grant found a significant decrease in gambling thoughts and behaviours after 6 weeks of naltrexone treatment

46
Q

Biological Interventions - Limitation of SSRI’s

A

Hollander’s study had a small sample size (only 10 participants). A larger and longer study by Blanco et al failed to demonstrate any superiority for SSRI’s over a placebo

47
Q

Psychological Interventions - Strength of CBT

A

Ladoucer et al allocated 66 pathological gamblers to a CBT or a ‘waiting list’ control group. After CBT, 86% of the group no longer fulfilled the DSM criteria for pathological gambling which was maintained even after a one year follow up

Freewill

48
Q

Psychological Interventions - Limitation of CBT

A

It is more effective if taken in conjunction with medication. Feeney et al found 14% abstention rates of a group of alcoholics in a CBT group, compared to 38% of a group having CBT in conjunction with medication

49
Q

Psychological Interventions - Strength of Aversion Therapy

A

Meyer and Chesser claimed a 50% success rate twelve months after with the treatment of alcoholics

50
Q

Psychological Interventions - Limitations of Aversion Therapy

A

Benefits are short term, it weakens over time. Relapse rates 70%, because patients have years of pleasant associations which over time override the more recent negative ones

Ignores the reason that lead to the addiction in the first place

Expensive therapy

Causes physical and psychological harm. Although, many actually prefer this rather than having to talk about their addiction and being challenged about it (CBT)

51
Q

Public Health Interventions - Limitations of Banning Smoking in Public Places

A

People may compensate by carrying out the behaviour even more at home

Encourages a sense of group solidarity amongst smokers, and a feeling of shared wickedness, which makes the habit seem more attractive

52
Q

Public Health Interventions - Restricting Advertising - Snyder

A

Studied young people aged 15-26. Found a strong correlation between the number of adverts for alcohol they ha seen and the amount of alcohol consumed

53
Q

Public Health Interventions - Strength of Restricting Advertising

A

Studies comparing cigarette consumption before and after the bans on advertising suggest there is a significant reduction

54
Q

Public Health Interventions - Limitation of Restricting Advertising

A

Not easy to conduct controlled studies of the effects of restriction, due to confounding variables

55
Q

Public Health Interventions - Strength of Increasing Costs

A

In cognitive terms, it could be a powerful factor when people weigh up the perceived costs of their behaviour against the perceived benefits

56
Q

Public Health Interventions - Limitation of Increasing Costs

A

For political reasons, increasing taxes is not as straight forward as it seems