Addiction Flashcards

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0
Q

(Smoking) Biological Initiation: Vink et al.

A

Studied 1572 Dutch twins

For both sexes, smoking initiation was explained by genetics (44%) and environmental influences (56%)

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1
Q

(Smoking) Biological Initiation: Family and Twin studies

A

Estimate the heritability of tobacco smoking to be between 39% and 80%

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2
Q

(Smoking) Biological Initiation: Boardman et al.

A

Conducted a US study of 348 MZ and 321 same-sex DZ twins

Estimated heritability for regular smoking to be 42%

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3
Q

(Smoking) Biological Maintenance: Buka

A

Mothers smoking during pregnancy doesn’t effect whether the child will later start smoking or not but does double the chances of addiction if they do

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4
Q

(Smoking) Biological Maintenance: Vink et al.

A

Affects brain chemistry by activating receptors on the brain leading to the release of dopamine
Creates short-term feelings of pleasure
Then this drops to impairment of mood and concentration due to nicotine level reductions
This is alleviated by smoking another cigarette

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5
Q

(Smoking) Biological Relpase: Khaled

A

Long term smoking has an adverse effect on mood because it permanently alters brain chemistry and smoking again alleviates this in the short term

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6
Q

(Smoking) Biological relapse: Uhl et al.

A

Research has tried to identify genes associated with quit success (not yet found)

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7
Q

(Smoking) Cognitive Initiation: Mermelstein et al.

A

Mermelstein et al also suggests that the expectation of states such as relaxation and increased self-confidence are reasons for adolescents beginning smoking.

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8
Q

(Smoking) Cognitive Initiation: Brandon et al.

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Expectancy theory is used to explain how a behaviour escalates into an addiction. Brandon et al suggest this is because of the individual’s expectations of the costs and benefits of an activity. For example adolescents may smoke when they are experiencing a negative mood as their expectation is that the cigarette will improve their mood.

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9
Q

(Smoking) Cognitive Maintenance: Automatic Processing

A

Automatic processing suggests that as an addiction develops, it is less about conscious expectancies and more about unconscious expectancies. This tries to explain the loss of control that many addicts experience.

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10
Q

(Smoking) Cognitive maintenance/relapse: tate et al.

A

Supportive research. Tate et al. told smokers to expect no negative experiences during a period of abstinence. This led to fewer reported somatic effects e.g. The ‘shakes’ and psychological effects e.g. Mood disturbance than the control group who were not told. Those told to expect somatic and not psychological problems experiences more severe somatic complaints than the control group who was not told to expect this.

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11
Q

(Smoking) Cognitive Relapse: DeVries and Backbier

A

Also DeVries and Backbier also suggest that expectations of the pros and cons of smoking and quitting affect their quitting behaviour. Smokers who expect that smoking has more benefits than quitting are more likely to relapse.

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12
Q

(Smoking) Cognitive Relapse: Moolchan et al.

A

Moolchan et al. demonstrated that nicotine patches only increased cessation rates and reduce relapse rates when accompanied by CBT to change the positive expectancies of smoking

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13
Q

(Smoking) Learning Theory Initiation: Karcher and Fisher

A

Youth with smoker parents were 1.88 times more likely to take up smoking
With smoker siblings, 2.64 times more likely to take up smoking
With close friends who smoke, 8 times more likely to take up smoking

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14
Q

(Smoking) Learning Theory Initiation: DiBlasio and Benda

A

Found that peer group influences were the primary influence for adolescents who smoke or take drugs

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15
Q

(Smoking) Learning Theory: Maintenance - Thewissen et al.

A

Cues were important in predicting smoking behaviour
In one room, smokers were presented with a cue associated with smoking and in the other, smokers were presented with a cue associated with not being able to smoke
Results showed the cue associated with smoking led to a greater urge to smoke

16
Q

(Smoking) Learning theory: Relapse - Drummond et al.

A

Developed a treatment based on this approach called ‘cue exposure’
Involves a person experiencing cues without the opportunity to smoke or engage in addictive behaviour
This breaks the association and reduces cravings
This was found to have good outcomes when tested on 35 alcoholics in 1994

17
Q

(Smoking) Learning Theory - Gender Bias

A

Gender bias - Nerin de Puerta and Jane (inherent gender bias in smoking research - onset follows different patterns for men and women) - theory fails to address this
Gender bias link - Lopez et al. found women start smoking later than men (gender Differences in initiation)

18
Q

(Gambling) Initiation: Biological - Shah et al

A

Conducted a twin study and found evidence of genetic transmission of gambling in men - 64% down to genes

19
Q

(Gambling) initiation biological - black

A

First degree relatives of pathological gamblers more likely to develop gambling addictions than more distant relatives

20
Q

(Gambling) biological initiation - family studies

A

Pathological gambling has been shown to run in families, suggesting genetic link

21
Q

Gambling biological maintenance - zuckerman

A

Claimed there are differences in the need for stimulation
High sensation seekers have a lower appreciation of risk and think of arousal as more positive than low-sensation seekers and so are more likely to gamble

22
Q

Gambling biological maintenance - cortisol

A

Cortisol is produced in the response to chronic stress
Under active cortisol levels were association with gambling (gamblers don’t find losing money stressful due to under active cortisol levels)

23
Q

Gambling biological maintenance - Paris et al

A

Recreational gamblers had increased cortisol levels after watching a video of their preferred method of gambling whereas pathological gamblers did not

24
Q

Gambling biological relapse - blaszcynski

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Poor tolerance for boredom may contribute to repetitive gambling behaviour
Gamblers may need the stimulation and excitement of gambling^

25
Q

Gambling cognitive initiation - Gelkopf et al

A

Gelkopf et al. suggests that addicts intentionally use pathological behaviours to treat psychological symptoms they already have. This results in a vicious cycle as addictions undoubtedly make the original problem worse in the long-run

26
Q

Gambling cognitive initiation - li et al

A

Li et al. found that pathological gamblers who gambled to escape the painful reality of life were significantly more likely to have other substance dependencies than pathological gamblers who gambled for pleasure

27
Q

Gambling cognitive initiation - Becona et al

A

Becona et al. suggested that major depressive disorder is evident in the majority of pathological gamblers

28
Q

Gambling cognitive maintenance - Oei and Gordon

A

Oei and Gordon argue that despite the odds of failure, gamblers frequently have irrational perceptions of their ability to influence the win. They often have exaggerated self-confidence in their ability to ‘beat the system’ and influence chance. They attribute success to skill or ability and failure to bad luck

29
Q

Cognitive gambling maintenance - Defabbro

A

Defabbro et al. found that pathological gamblers were just as accurate as non-gamblers in estimating the odds of winning

30
Q

Gambling cognitive maintenance - griffiths

A

Griffiths compared 30 gamblers to 30 non-gamblers in their verbalisations at fruit machines. Gamblers were more likely to be irrational. “It doesn’t like me”, “My luck’s in today” or “I wasn’t concentrating so I lost” are examples of irrational speech.

31
Q

Gambling cognitive relapse - blanco et al

A

Blanco et al. suggested that pathological gamblers suffer from a recall bias meaning they have a tendency to remember and overestimate wins while forgetting about losses. They believe in a just world where they will be rewarded for their efforts and believe they deserve to win because they have lost so often.