Acute resuscitation OSCE's Flashcards

1
Q

General approach to Adult Cardiac Arrest?

A

Initial
- Declare cardiac arrest and call code blue
- Attach defibrillator and monitoring
- Start CPR at ratio 30:2
- Fast: CPR 100-120 compressions per minute
- Hard: Lower half of the chest to 1/3rd depth of the chest (or 5cm)
- Use BVM

VF/VT
- Immediate shock
- Adrenaline after 2nd shock
- Amiodarone after 3rd sjock
- Adrenaline every 3-5mins (every 2nd shock) thereafter
- Consider Lidocaine

PEA/Asystole
- Immediate Adrenaline
- Adrenaline every 3-5mins (every 2nd cycle) thereafter
- Seek and treat reversible causes

Airway
- If hypoxic cause of arrest or issues with ventilation (ie bronchospasm or angioedema) then ETT is indicated
- If above not present then BV< is considered adequate as ETT placement in arrest has high rates of misplacement and interrupts CPR
- SGA’s have theoretical benefits but are unproven, not helpful in severe bronchospasm
- Once advanced airway in place change ventilation to 6-10/min and do continuous CPR

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2
Q

General approach to Paediatric cardiac arrest?

A

Initial
- Declare cardiac arrest and call paediatric code blue
- Attach defibrillator and monitoring
- Give 2 rescue breaths
- Start CPR at ratio 15:2
- Fast: CPR 100-120 compressions per minute
- Hard: Lower half of the chest to 1/3rd depth of the chest (or 5cm)
- Use BVM to deliver 100% 02, be careful not to overinflat
- Consider use of Ayers T-Piece if available to guide positive pressure delivered

Differences to Adults
- Most likely causes of arrest are hypoxia and hypovolaemia
- More likely to be non-shockable
- Higher priority to secure the airway for improved oxygenation (LMA or ETT)
- 6Hs and 5T’s

VF/VT
- Immediate shock 4J/kg
- Adrenaline 10mcg/kg after 2nd shock
- Amiodarone 5mg/kg after 3rd shock
- Adrenaline every 3-5mins (every 2nd shock) thereafter
- Consider Lidocaine 1-1.5mg/kg

PEA/Asystole
- Immediate Adrenaline 10mcg/kg
- Adrenaline every 3-5mins (every 2nd cycle) thereafter
- Seek and treat reversible causes

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3
Q

General approach to Newborn cardiac arrest?

A

Indications
- NeoResus only indicated in the peri-birth period, unclear exact cut off but >4-6hrs should change to paeds ALS

Equipment (see pic)
- Resuscitaire and warmer
- Neopuff and/or neonatal BVM at 20 IPAP and 8 PEEP
- Consider higher IPAP for initial breaths of 30cmH20 or higher to overcome atelectasis and lung fluid
- ETT uncuffed 3.0, 3.5 and 4.0
- size 1 LMA (2 - 5kg baby)
- size 0 and 1 miller blades, V/L size 1/0 Mac blade
- introducer, mec aspirator
- 10mcg/kg Adrenaline (30-50mcg)
- N. Saline, 10% dextrose and PRBC
- Umbilical line, 24g cannula, pink intraosseous needle

Steps for compromised baby
- Warm, briskly stimulate and dry the baby
- If preterm place in polyethylene bag and dry head/stimulate through bag
- If inadequate respiration then place in neutral position, may also jaw thrust/chin lift and put towel under shoulders
- Only suction airway if obvious evidence of obstruction
- Trial CPAP (PEEP) at 5cm/H20
- If insufficient add PPV (IPAP) 30cm/H20 at a rate of 40-60 breaths per minute
- Use room air at 21% 02 initially, max 30% 02 if sats failing to respond
- If requiring CPR switch to 100% 02

Neonatal CPR
- Indicated if HR <60 despite good assisted ventilation for >30secs
- 2 thumbs adjacent to each other over the lower 1/3rd of the sternum, with fingers around the thorax to support the back
- Compress chest 1/3rd of the anterior-posterior diameter
- Ratio of 3 compressions to 1 breath, 1/2 second pause to deliver breath
- 3:1 ratio delivers 90 compressions per minute
- If intubated can consider 120 compressions per minute with no break for ventilation (ie simultaneous) with 40 breaths per minute
- Still doing 2 minute cycles

Special points
- Any compromised baby should have their cord clamped ASAP, a low SVR placenta attached will render CPR ineffective
- If the neopuff flow rate is 10L/min (usual initial setting), then 30% Fi02 will be done with 9L/min air to 1L/min of 100% 02
- Once intubated the ETT will be placed at 8-9cm at the lips (Age/2 + 12 for older kids, not applicable here)

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4
Q

Approach to Angioedema?

A

Angioedema with Urticaria DDx
- Anaphlayxis 90%
- Infection, autoimmune disease, idiopathic, pseudoallergic
- Treat with Adrenaline 10mcg/kg IM +/- adrenaline infusion (mainly for allergic causes)
- Hydrocortisone IV 4mg/kg or Dexamethasone 0.6mg/kg IV
- Consider emergent airway control +/- FONA

Angioedema NO Urticaria DDx
- Hereditary angioedema (most commonly C1-inhibtor deficiency)
- Acquired C1 deficiency (malignancy, autoimmune)
- Drug induced (ACE-I and NSAID induced Angioedema)
- Upper airway trauma or infection (ie recent ENT surgery or dental procedures/infection)
- Idiopathic angioedema
- Upper airway contact dermatitis
- Malignancy (ie Superior vena cava syndrome)

History
- Rash vs no rash
- GIT/NV symptoms
- Wheeze and pre-syncopal Sx
- Recent exposure to allergen
- PHX of swelling or allergy
- FHx of C1-inhibitor deficiency
- New medications
- Liver/Kidney/Heart (generalised oedema)

Tests
- Mast cell Tryptase
- C1 inhibitor concentration and function
- Inflammatory markers

Angioedema NO rash Mx
- ADRENALINE does NOT HELP
- ICATIBANT subcut 30mg (bradykinin receptor antagonist, usually patient will take prior to coming in)
- TXA 15mg/kg PO TDS, home medication
- C1 inhibitor concentrate (BERINERT, 25U/Kg IV, hospital medication)
- Fresh frozen plasma (2 units of 15ml/kg IV every 2-4hrs, contains C1 inhibitor)
- Ecallantide 30mg Sub cut (Kallikrein inhibitor)
- If in a rural area without access to above treatments (excluding FFP) consider early prophylactic intubation if airway involvement (awake oral or fibreoptic, mark neck for FONA)

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5
Q

Approach to the seriously unwell Pregnant patient

A

General Resus
- Aortocaval decompression (uterus to the left)
- CTG and consideration of emergent cesarean section
- Early Saline and Noradrenaline
- Consider peripartum cardiomyopathy
- Early discussion with NICU, anaesthetics and ICU
- HF nasal cannula vs BiPAP

Cardiac arrest causes
- Underlying cardiac disease
- PE
- Amniotic fluid embolism
- Haemorrhage
- Sepsis
- Hypertensive disorders
- Poisoning/self harm
- Any normal cause

Cardiac arrest management
- Standard ACLS management
- Aortocaval decompression (manually displace urterus to left, left lateral tilt 15-30 degrees)
- Prepare for resuscitative hysterotomy

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6
Q

General approach to starting ECMO?

A

General Indications
- Pulmonary +/- Cardiac failure
- Condition ir reversible
- OR transplantation is an option
- All other options have been exhausted or are likely to be futile

Neonatal indications
- Severe Mec aspiration
- Persistent foetal circulation
- Large congenital diaphragmatic hernia
- Severe pulmonary HTN
- Bronchiolitis
- Selected congenital HD’s

Specific Pathologies VV
- Status asthmaticus
- Large PE
- Traumatic pulmonary contusion
- Pneumonia/ARDS
- Post drowning

Specific pathologies VA
- Myocarditis
- Hypothermia
- Beta blocker/CCB overdose
- VT storm/intractable dysrhythmia
- E-CPR

Differences VA and VV
- VV only provides Gas exchange (+ dialysis and heat exchange)
- VA adds on cardiovascular support
- VA higher risk of limb ischaemia and thromboembolism

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7
Q

Approach to the Critical Asthma?

A
  • Escalation of therapies
  • Identification of complications (tension pneumothorax, salbutamol toxicity, arrhythmias etc)
  • Exclusion of alternate causes (APO, pneumonia, pneumothorax, PE etc)

Approach to intubation
- Try not to intubate!
- Recognise high likelihood to arrest on induction
- Adrenaline infusion and IV bolus 20ml/kg prior to induction
- Consider delayed sequence with NIV
- BVM 100% with NP15L/min underneath if/when apnoeic
- Induction with 1mg/kg IV Ketamine, 1.6mg/kg IV Rocuronium

Cardiac arrest causes
- Terminal hypoxia from bronchospams and mucous plugging
- Arrhythmias from eletrolyte disturbance, hypoxia and adrenergic agents
- Dynamic hyperinflation (Auto-PEEP)
- Tension pneumothorax

Cardiac arrest alterations
- Follow standard resus guidelines considering the above
- Consider co-existing anaphylaxis
- Consider early endotracheal intubation
- If dynamic hyperinflation suspected then consider disconnecting tube with pressure ant-post and lateral chest wall followed by period apnoea (ie no breaths every 30 compressions)

Ventilator Settings
- PEEP is controversial, concerns about worsening iPEEP but also splinting open airways with ePEEP can help lower iPEEP
- Start Fi02 1.0, but can titrate down later to sats >90%
- Use expiratory hold to measure end expiratory pressure and subtract the extrinsic PEEP from this to get the iPEEP (aim iPEEP <10cmH20)
- TV 6ml/kg
- Permissive hypercapnoea aiming pH >7.2 and CO2 <80
- in line MDI for bronchodilators
- RR 8-12, closer to 8 if critical
- Combined TV and RR to a minute volume (MV) <115ml/kg/min
- short inspiratory/long expiratory time with I:E ratio 1:4 or greater
- High inspiratory pressure limit, pressure might be high but accept plateau pressure to 30 (measure with inspiratory hold maneuver)
- Higher inspiratory flow rates approx 60 - 80L/min
- Paralysis bolus/infusions
- Deep sedation
- Ongoing IV therapies

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8
Q

Approach to the Tracheostomy or Laryngectomy patient with respiratory compromise?

A

Laryngectomy (Lx) differences
- Lx patients are obligate stoma breathers
- Tx patients still have at least partially patent upper airway
- Lx stomas are typically larger
- Lx stomas will have a blind ending superior tracheal inlet
- Lx often have a heat moisture exchanger (HME) on the outside that can be removed
- Sometimes a flexible tube is attached to the HME (Larytube) that is used to maintain patency early post surgery
- Lx often have a tracheo-esopheal prosthesis (TEP) inserted inside to allow for speech (one way valve that should not be removed)
- Capnography should be placed over the stoma, if placed over the mouth it will be 0 (confirming a laryngectomy)

Laryngectomy complications
- Pharyngocutaneous fistulas, dehiscence and local haematoma in 7-10 days post surgery
- Obstruction by FB (parts of the stoma care or the TEP dislodging)
- Excess secretions (try 5mls of N. saline to break it up, suction)
Carotid Blowout Syndrome
- More common than tracheo innominate fistula in Lx (TIF rare in Lx patients due to lack of inner cannula)
- When the carotid or its branches erode into the Lx
- Heralded by new haematoma, bruise or bleeding
- Secure with ETT, direct pressure, early surgical/IR and MTP

Laryngectomy Emergency
- Call ENT/Anaesthetics
- Arrange OT
- NRB 15L to stoma +/- face if unclear of Lx vs Tx
- Can place LMA or paediatric face mask with BVM over stoma
- Ideally Mapleson C circuit with T-piece, but BVM will do
- Capnography to stoma
- Difficult airway trolley, ideally fibreoptic scope, size 6 or smaller ETT, trahcoestomy kit, soft suction catheters and bougie
- Remove outer covering, HME, any inner cannula and suction
- If not working then remove outer cannula as well (only stoma left)
- Never remove the TEP
- If still not working then ideally fibreoptic guide size 6 ETT into stoma

Tracheostomy Emergency
Tracheoinnominate fistula
- Overinflate the Tx balloon
- If fails then remove cannula and place finger into Tx and put direct pressure over bleeding site
- Ideally intubate from above to maintain airway in this situation

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9
Q

Approach to the Geriatric Airway and peri-intubation?

A

Issues
- Increased likelihood to need to be intubated due to lack of reserve
- Increased difficulty with BVM (missing teeth, less facial tissue)
- Reduced cardiopulmonary reserve leading to faster hypoxia and hypotension
- Need for adjustment of drug doses
- More friable airway tissue and may bleed easier (ie on anticoagulants)
- At greater risk of C-spine injury from laryngoscopy

Physiologic changes
- Impaired gas exchange
- Increase V/Q mismatch
- Decreased lung elasticity/decreased compliance leading to increased WOB
- Less chest wall muscle
- Reduced cough and mucocilliary clearance (aspiration risk)
- Reduced brain responsiveness to hypoxaemia and hypercarbia
- Heightened sensitivity to negative inotropy/vasodilation from induction agents
- Greater number of co-morbidities

Modifications
- Inquire about patient wishes and advanced care directives prior to intubation
- Adequate pre-oxygenation and apnoeic 02/BVM through apnoeic period
- Adequate cardiovascular resuscitation prior to induction, consider bolus/infusion vasopressors
- Normal dose of paralytics, but decrease induction dosing by 30-50%
- Use of 2-hand technique and NPA/OPA for BVM
- Use of VL and adjuncts to aid intubation
- Consider awake intubation

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10
Q

Approach to the Obese airway and peri-intubation?

A

Issues
- Increased thoracic fat distorts airway position when on back
- Large breasts
- Reduced intraoral space
- Harder to palpate CTM
- Raised Intra-abdominal pressure increased the risk of aspiration
- Facial fat makes BVM harder
- Reduced FRC and atelectasis
- Higher metabolic demand leads to faster desaturation
- Difficult IV access
- less reliable NIBP readings

Modifications
- use ramping to get tragus in line with sternal notch
- Consider short handly laryngoscope for large breasts
- Vertical incision then dissect down to CTM before horizontal CTM cut
- 2-handed BVM technique with NPA and OPA

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11
Q

What is the predicted BVM difficulty mnemonic?

A

MOANS

Mask Seal
- Bushy beard, crusted blood, disruption to facial continuity in trauma

Obesity/Obstruction
- Obesity, pregnancy, angioedema, ludwigs angina, upper airway, abscess, epiglottis

Age
- >55

No Teeth
- Leave dentures in edentulous patients

Sleep Apnoea/Stiff lungs
- COPD, asthma, ARDS

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12
Q

What is the predicted intubation difficulty mnemonic?

A

LEMON

Look externally
- Lower facial disruption, bleeding, small mouth, agitated patient

Evaluate (3-3-2 rule)
- Mouth opening >3 fingers
- 3 finger breadths from tip of mandible to anterior neck
- 2 fingers from the base of the mandible to the thyroid neck

Mallampati Score
- I Complete visualisation of soft palate
- II Complete visualisation of the Uvula
- III base of the uvula only
- IV No soft palate seen

Obstruction/Obesity
- obesity
- stridor, muffled voice, not swallowing secretions, sniffing position/tripod

Neck Mobility
- Trauma C-spine precautions
- Arthritis, ank spondylitis

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13
Q

What is the mnemonic for difficult LMA insertion?

A

RODS

Restricted mouth opening

Obstruction

Disrupted/Distorted anatomy

Stiff lungs/cervical spine

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14
Q

What is the mnemonic for difficult front of neck access?

A

SHORT

Surgery
- ie previous laryngectomy etc

Haematoma
- also infection, abscess, any swelling

Obesity/Obstruction

Radiation
- To the neck, distorts tissue planes

Tumour
- Neck and mediastinal tumours

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15
Q

An approach to massive haemoptysis SCBD

A

Life Threats!
- Massive PE
- Eroded tumour (Lymphoma, mets, primary bronchial tumour)
- Severe pneumonia or abscess (staph, strep, legionella)
- AVM or aortobronchial fistula
- aortic aneurysm
- Tracheoinnominate fistula or Carotid blowout syndrome (laryngectomy and tracheostomy patients)
- Severe coagulopathy/DIC (including leukaemia)
- Severe congenital or acquired heart disease or pulmonary hypertension (RHD with MS as example)
- Trauma or iatrogenic ie post procedural

DDx
- Bronchitis or Bronchiectasis (chronic infection, cystic fibrosis)
- TB
- Foreign body
- Non-eroding tumour
- Vasculitis (Wegeners, Goodpastures, SLE)
- Cocaine
- Mimics (haematemesis, post tonsillectomy, epistaxis)

History
- Rate, amount, and appearance (more than 500mls highly concerning)
- Previous episodes
- Known PHx
- PE risk factors
- Recent trauma/surgery
- Bleeding diathesis
- Medications (ie anticoagulants)

Investigations
- FBE and film
- Group and hold + cross match
- VBG, BSL and electrolytes
- Coags, D-Dimer, troponin
- ECG
- Sputum culture
- CXR/CTPA
- Early bronchoscopy

Emergency Management
- Soiled airway algorithm, difficult airway trolley, low threshold for FONA
- ENT/Resp/ICU/Anaesthetics
- Massive transfusion protocol, TXA 15mg/kg bolus then infusion (+/- Vit K, prothrombinex)
- Bleeding lung down (if known)
- Large bore ETT (ideally 8.5 or 9)
- Consider specifically intubating the R) or L) mainstem with 90 degree rotation, then can consider bronchial blocker post
- Anaesthetics may consider a double lumen tube
- Ultimately needs IR and angioembolization +/- thoracic surgery

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16
Q

What strategies can be implemented to tackle the physiologic challenges of intubating a critically ill patient?

A

Pre-oxygenation
- NRB or NIV
- Sit up until last moment

Apnoeic 02
- Nasal prongs vs BVM

Avoid Acidosis
- Important if already very acidotic or acid sensitive ie aspirin/TCA OD
- NIV, BVM in apnoeic period for ventilation, sodi bic

Hypotension
- Adequate pressors/fluids
- Reduced dose induction agents

Hypertension
- Pre-induction ie 2mcg/kg Fentanyl
- Important for hypertensive crises and strokes/bleeds

Aspiration
- Suction, head up 30 degrees
- SALAD

Difficult airway
- Most senior operator, VL, hyperangulated blade, ETT adjuncts

Pulmonary oedema
- NIV, BVM with PEEP
- Suction, vasodilators
- Sit upright, avoid fluids

General Answer to Intubation Q
Equipment
- ETT, bougie/stylet, adjuncts, difficult airway trolley, suction and justify)
Drugs
- type, doses and justify
Peri-Induction/Intubation
- Positioning, pre-oxygenation, resuscitate before intubate
Ventilator Settings
- based on clinical scenario
Post Intubation
- capnography, CXR, equal chest rise, auscultation, fogging, sedation and paralysis, NGT and IDC
Disposition
- Theatre vs retrieval vs ICU

17
Q

Spinal cord injured patient

A

High risk C-spine fractures
Jeffersons fracture
- C1 burst fracture
- Diving
Hangman’s Fracture
- C2 fracture
Bilateral facet joint dislocation
- Buckling or hyperflexion
- Anterolisthesis of the cephalad vertebra compared to the caudad vertebra
Flexion tear drop
- Hypeflexion
- Diving injuries
Chance fracture (thoracolumbar)
- T12/L1 hyperflexion
- Back seat car crash

Spinal Cord Injury complications ED
- Neurogenic shock
- Respiratory failure
- Expanding haematoma in the neck
- Urinary retention
- Aspiration
- Hypothermia from reduced autonomic regulation
- Pressure areas (if delay to transfer)

Spinal shock and Neurogenic shock
- not a true form of shock, it refers to the flaccid areflexia that occurs after spinal cord injury, and may last hours to weeks
- It may be thought of as ‘concussion’ of the spinal cord and resolves as soft tissue swelling improves. - Priapism may be present with complete transection (complete loss of sympathetics)
- Bulbocavernosus reflex is present in incomplete lesions with spinal shock but absent in cord transection/complete lesions (sacral sparing in incomplete lesions)
- Spinal shock at a level above T6 can lead to neurogenic shock
- Neurogenic shock is hypotension, bradycardia and peripheral vasodilation

SPINAL CORD SYNDROMES
Anterior Cord
- Complete paralysis below lesion + loss of pain and temp sensation
- Intact posterior columns including vibration and proprioception
- Poor prognosis

Central cord Syndrome
- Upper > Lower limb quadriparesis and loss of pain/temp sensation
- Good prognosis

Brown-Sequard Syndrome
- Ipsilateral loss paresis and loss of proprioception/vibration
- Contralateral loss of pain and temperature, these tracts cross the midline

General Management
- Assess and treat ABC’s (ETT, 02, fluids, inotropes)
- Spinal immobilisation/precuations!!
- Analgesia
- Early discussion with Neurosurgical or trauma service, organise retrieval if not on site
- Consider cause for injuries (intoxication, pathological fall, NAI or domestic violence, suicide attempt etc)
- Assess and manage associated injuries
- Attend to predicted complications (IDC, NGT, keep warm, fast, IV fluids, pressure ulcers etc)

18
Q

General approach to severe burns?

A

Resuscitation
Initial
- Send for help, trauma call, consider anaesthetics and ENT, massive transfusion protocol
- Analgesia as needed (IN fent)

Airway
- Assess for airway burns, airway trauma and obstruction, intubate as required
- Consider awake fibreoptic intubation/laryngoscopy, ketamine with topicalisation

Breathing
- Assess for chest wall injuries
- Consider need for escharotomy for chest wall burns
- Lung protective ARDS strategy if intubated

Circulation
- IV Saline bolus +/- MTP for shock
- Modified Parklands (3 x kg x TBSA), 50% in 1st 8hrs and 50% next 16hrs
- Early urinary catheter for adequate fluid balance assessment (1ml/kg/hr)
- In children also give maintenance fluid (4:2:1 rule)
- If fluid given prior then take this off the full parklands formula amount
- Some centres may also adjust rate based on number of hours since burn and also halve the parklands formula calculated amount to account for increased vascular permeability

Disability
- Fentanyl and ketamine for analgesia
- Assess for concomitant head/spine injuries
- Seek and treat hypoglycaemia, add 5% dextrose to Hartmans/saline

Extra/Environment
- Burns first aid
- Temperatures management
- Clean, debride and assess burn % and depth (Lund and Browder chart)
- Transfer if not at tertiary centre

19
Q

Approach to the critical trauma patient and traumatic cardiac arrest?

A

Permissive hypotension
- Minimal actual data
- Works best for penetrating thoracoabdominal trauma
- Contraindicated in head injured patients
- Relative contraindication in spinal cord injuries, small children, pregnant patients, pre-load dependent patients and those with baseline significant hypertension
- Usually 1L of crystalloid then blood products as needed, aiming SBP 80-90mmHg or palpable radial pulse and GCS 15
- Rationale is to reduce clot disruption and not contribute to the lethal triad of trauma
- Saline is acidic, cold and dilute the blood (clotting factors)

Massive Transfusion Protocol
Definition
- >10units of RBC’s or whole blood in 24hrs
- Others include use >4units in 1hr, >40ml/kg RBCs, >1 blood volume replacement in 24hr or >50% blood volume in 4hrs
Indications
- Severe thoracic, abdominal, pelvic or long bone trauma
- Anticipated ongoing haemorrhage
- Major obstetric, surgical/GI bleed
- TASH (trauma associated severe haemorrhage score) can be used to predict who will need it
Physiological Aims
- Temp >35, pH >7.2
- Lactate <4, BE <-6
- Ionised Calcium >1.1
- PT/APTT <1.5x normal
- INR <1.5, Fibrinogen >1.0
- Platelets >50

20
Q

Hanging SCBD

A

History points
Prognostic indicators
- Down time, first aid/BLS, inital vital signs and GCS
- Cardiac arrest at scene
- Estimated height of fall if did fall
PAIDEM’s
- Co-morbidities
- Drug, alcohol and mental health issues
Other injuries/pathology
- Other trauma, co-ingestion, environmental exposure

Clinical Features
- External trauma to neck (ie ligature marks)
- Significant facial oedema and cyanosis
- Tardieu spots (petechiael haemorrhages in the conjunctiva and mucous membranes cephalad to ligature, associated with asphyxiation death
- Thyroid cartilage and hyoid bone fracture
- BCVI, particularly carotid injury
- Hypoxic ischaemic encephalopathy

Complications
- HIE
- Cardiac arrhythias/arrest from hypoxic ischaemia
- Negative pressure APO
- Neurogenic APO
- Hyoid, thyroid and C-spine fractures

Testing
- Head and neck CTA + CTB

21
Q

Approach to Bradycardia

A

Stable vs Unstable

Symptomatic vs Asymptomatic

Reversible causes

Anatomic location
- Wide QRS vs narrow WRS

22
Q

Approach to Ventricular Tachycardia

A

Classification
- Structurally normal vs structurally abnormal heart
- Monomorphic vs polymorphic
- Stable vs unstable vs pulseless

DDx for wide complex tachy
- Monomorphic VT
- Polymorphic VT
- SVT with aberrancy
- AF/flutter with aberrancy
- HyperK and TCA
- Pacemaker mediated tachycardia
-

Risk factors for VT
- Structural heart disease
- Channelopathies
- Acute ischaemia
- Ischaemic cardiomyopathy
- Electrolyte disturbance
- Hypothermia
- QT prolongation
- Age >35

Brugada Criteria
- See pic
- 98% sensitive and 96% specific
- Cannot reliably distinguish VT from Antidromic AVRT, no well validated algorithm can

Basel Criteria
- 93% sensitive, 90% specific, takes substantially shorter time to apply the criteria
- VT present if 2 or more of the 3 criteria are present
- 1: High risk clinical feature (History MI, history of ICD, history of HFrEF <35%)
- 2: Lead II time to first peak (ie when QRS changes direction) of >40ms
- 3: Lead AVR time to first peak >40ms

Antidromic AVRT vs VT
- Rule not validated but sometimes used, realistically need EP lab
- Rule about 70% sensitive, but 100% specific
- If yes to any then treat as VT
1- Predominantly -ve QRS in V4-6?
2- Presence of qR complex in any of V2-6?
3- AV dissociation?

Chemical Cardioversion
- Amiodarone 5mg/kg bolus followed by infusion
- Procainamide 15mg/kg IV
- Lignocaoine 1-1.5mg/kg bolus followed by infusion

Electrical cardioversion
Pulseless
- Unsynchronised 200J DC biphasic or 360J monophasic
-
Pulse
- 100-200J (1, 2, 4j/kg) synchronised

Electrical storm
Definition
- VT recurring within 5mins of cardioversion
- 3 or more episodes in 24hrs
- more PVC’s than sinus beats in 24hr
- Includes VT, pVT and VF
Triggers
- Drug toxicity or electrolytes
- Acute ischaemia
- Thyrotoxicosis
- QT prolongation
- New or worsened heart failure
Treatment
- Cardioversion
- Coronary revascularisation if ischaemia
- IV Amiodarone infusion
- IV/oral beta blocker (propranolol, sotalol or esmolol)
- Stellate ganglion block or thoracic epidural anaesthesia
- Intubation and ongoing sedation (reduces sympathetic tone)

Refractory VT
- As for electrical storm
- Overdrive pacing (above VT rate) followed by decremental pacing to slower rate (often done by AICD, but can be done with TV or TC pacing)

Polymorphic VT
- Congenital vs acquired (ischaemic vs non-ischaemic)
- Distinction based on differing treatment algorithms
- Long QT vs no long QT
Congenital
- Long QT, Catecholaminergic PVT and rarely Brugada syndrome
- General treatment is to slow down the rate with IV betablockers
Non-Ischaemic
- Almost invariably from long QT
- Antiemetics, psychotropics, antbiotics and antiarrhytmics
- Low K/Mg/Ca
- Hypothermia, RICP
- General treat with Mg+ and overdrive pacing, can consider Lignocaine
- Replace specific electroltyes and give specific antidotes if tox
- Amiodarone contraindicated
Ischaemic
- Causes both TdP from long QTc and PVT without long Qtc from myocardial irritability
- Thrombolysis/PCI, reduce ischaemia
- Overdrive pacing may worsen ischaemia and worsen situation
- Amiodarone contraindicated if QTc long, but not if PVT from with normal QTc

23
Q

General approach to undifferentiated shock?

A

General
- Consider the 5 causes of shock
Cardiogenic
- Arrhyhtmia, heart failure, ischaemia, myocardial contusion
Distributive
- Sepsis, anaphylaxis, neurogenic
Hypovolaemic
- Haemorrhage, sepsis, dehydration
Obstructive
- PE, tamponade, pneumothorax
Dissociative
- Severe anaemia
- Aspirin, CO, cyanide

Tip
- List relevant differentials as above, demonstrates thinking broadly
- Say as part of HEI how you would rule in or rule out the above

RUSH Protocol
- See image
- Add on DVT and ectopic pregnancy screens as indicated
- Assess for cardiac causes, PE, tamponade, dissection, AAA, haemopneumothorax and intrabdominal bleeding

24
Q

Stridor approach and intubation?

A

Critical DDx
- Epiglottis
- Anaphylaxis/Angioedema
- Trauma with expanding haematoma in the neck
- Ludwigs angina
- Retropharyngeal abscess
- Peritonsillar abscess
- Bacterial tracheitis/Laryngo tracheo bronchitis (more common in children, still possible in adults)
- Aspirated foreign body
- Caustic substance ingestion/inhalation
- Diptheria

Emergent DDx
- Trauma with non-expanding neck haematoma
- Tumour with mass lesion effect in the neck
- Laryngomalacia
- Subglottic stenosis
- Vocal cord dysfunction

Intubation approach
- Ideal is AFOI
- next is gas induction in theatre
- Next is laryngoscopic ketamine and lignocaine assisted awake intubation
- Next is double set up with RSI and FONA
- Alternatiely elective cricthyroidotomy or tracheostomy
- Dexamethasone 0.6mg/kg and nebulised adrenaline 5mg (5x 1:1000)

25
Q

Intubation with C-spine injury

A

MILS
- remove collar and have an assistant keep the head in the neutral position
- Place hands on angles of mandible or on side of head

Different options
- See pic

Risk
- Very low, only small retrospective studies
- If unable to intubate during laryngoscopy, remove MILS (airway over c-spine)

26
Q

Head injury intubation

A

Oxygenation
- PREVENT HYPOXIA
- Adequate pre-ox
- Apnoeic 02 or bag during the apnoeic period

CO2
- Prevent hypo/hypercarbia!
- Attach capnography to BVM
- Aim CO2 35-40 with BVM and post when ventilated

Blood pressure
- Avoid HYPER and HYPOtension
- If hypertensive them give 3-4mcg/kg of IV fentanyl
- Propofol for induction (low dose), ketamine not contraindicated and preferred if patient hypotensive
- Consider short acting hypotensive agents (ie esmolol)
- Reduce laryngeal stimulation and reflex hypertension, gentle laryngoscopy (video, optimise positioning, 1st pass, most senior operator)
- Consider lignocaine 1.5mg/kg IV
- Have short acting hypertensive ie metaraminol if BP drops

ICP
- Consider Mannitol 0.5gm/kg or 3ml/kg of 3% saline prior to intubation
- Head in neutral position, 30-45 degrees up, remove collar, dont use ETT tie

Paralysis
- Rocuronium preferred
- Sux may increase ICP
- Remain paralysed and well sedated post (prevent coughing, straining etc)

Post Intubation
- Aim CO2 35-40
- Sats 95% aim (avoid hypo and hyperoxia)
- Minimum PEEP necessary to avoid hypoxia

27
Q

Drowning approach?

A

Assessment
- Immediate life threats
- Trauma including head/neck
- pulse present?
- Aspiration/lung injury
- Prognostic indicators

Exam
- Maintaining airway, signs of aspiration, adequacy of breathing (sats, RR)
- Signs of perfusion (Hr, BP)
- Temperature, at risk hypothermia
- Signs of head/neck trauma, neurological deficits, consider empiric spinal precuations

Good Prognostic factors
- Submersion time <5mins
- Time to effective BLS <10mins
- No CPR in progress on hospital arrival
- GCS >5
- Rectal temp >30C
- Arterial pH >7.1 on arrival
- 1st spont breath within 30mins
- Immersion in water <10 degrees C

Management
- Respiratory distress in semi conscious patient can escalate from NRB to NIV to intubation
- Decompress stomach with NG (often very distended)
- Support BP with judicious fluids and vasopressors
- No specific indication for antibiotics unless contaiminated water

28
Q

Post intubation hypoxia and hypotension

A

Hypoxia
DOPES Mnemonic
- Displacement of ETT
- Obstruction of ETT
- Patient: pneumothorax, PE, aPO etc
- Equipment: Ventilator issue, tubing kinked, 02 disconnected etc
- Stacked breathes: Bronchospasm, incorrect ventilator settings

Management
- Disconnect tube and attached BVM
- Give 100% Fi02 with PEEp valve
- Determine patient vs Equpiment
- Check EtCo2, consider laryngoscopy to confirm position
- If BVM easy but no chest movmeent then likely dislodged ETT
- If BVM hard with slight chest movement consider R) mainstem, bronchospasm, hyperinflation and pneumothorax, block/kinked ETT
- If BVM easy, chest moves and patient improves then consider circuit/machine issue

Post Induction Hypotension
AAH SHITE Mnemonic
A- Acidosis
A- Anaphylaxis
H- Heart (tamponade, pHTN, arrhythmia)
S- Stacked breaths
H- Hypovolaemia
I- Induction agent
T- Tension PTX
E- Electrolytes

Management
- Assess for obvious cause based on 5 causes of shock
- CXR, bedside echo, ECG, examine
- judicious fluid bolus
- Early vasopressors including IM adrenaline for anaphylaxis
- As per DOPES disconnect and hand ventilate
- Decompress chest if needed
- Early discussion with ICU, consider IABP or ECMO

29
Q

Heat stroke SCBD

A

DDx
- Sepsis (meningoencephalitis, Staph TSS etc)
- Toxidrome (SS, NMS, MH, anticholinergic)
- Intracranial bleed (SAH, thalamic bleed, head trauma)
- Metabolic (Hyperthyroid, hypoglycaemia, dehydration)
- Neurological (Post seizures, thalamic dysfunction)

Complications
-Rhabdomyolysis
- DIC
- Cerebral oedema
- AKI
- Multiorgan failure

Management
- Cooled IV fluids, usually hyperhydration due to concomitant rhabdo (1-2ml/kg/hr UO, IDC, early consideration of dialysis))
- Escalating cooling measures (remove clothes, tepid water, fans, ice packs, ECMO/DIalsysis, lavage)
- Intubation and paralysis (dont use Sux) for lung and neuroprotection but also prevent shivering
- Empiric broad spectrum antis if possibly sepsis
- Toxidrome antidotes if applicable